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Reversing Type 2 Diabetes Through Fasting

Reversing Type 2 Diabetes Through Fasting

Reversing Type 2 Diabetes Through Fasting

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By Dr. Edward Group
Guest writer for Wake Up World
Type 2 diabetes, sometimes referred to as adult-onset diabetes, doesn’t have to be permanent. Fasting and calorie restriction can help you get control of your blood sugar, lower your blood pressure, and even help reverse type 2 diabetes. But, before we get into how fasting can undo the damage of type 2 diabetes, we first need to understand how type 2 diabetes affects the body.
How Does Type 2 Diabetes Develop?
Diabetes develops when fat accumulates in areas of the body that shouldn’t accumulate fat. It all starts with an abundance of fat in your muscle tissue. Typically this is caused by a family history, poor diet, or sedentary lifestyle. This fat is called intramuscular fat. It’s like the marbling on a steak, only it’s inside your muscles, and it causes insulin resistance — the characteristic that distinguishes type 1 diabetes from type 2.[1, 2] Even worse, intramuscular fat causes muscles to produce toxic fat metabolites like ceramide and diacylglycerol (DAG). These toxins also contribute to insulin resistance.[3]
High Insulin Levels Lead to a Fatty Liver
When blood sugar is high, the pancreas produces insulin to lower blood sugar. However, insulin resistance causes the liver to stop responding to insulin.[4, 5] In fact, the liver keeps producing sugar despite a high level of sugar in the blood. Consuming food that’s high in sugar is like throwing gas on the fire, and the abundance of sugar is converted to fat and stored in the liver.[4]
When the liver accumulates fat, non-alcoho Continue reading

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Solera Health and the American Diabetes Association® Collaborate to Help Prevent and Delay Type 2 Diabetes for Millions of Americans

Solera Health and the American Diabetes Association® Collaborate to Help Prevent and Delay Type 2 Diabetes for Millions of Americans

Relationship Will Facilitate Access to CDC-Recognized Diabetes Prevention Programs for Eligible Consumers
PHOENIX, AZ – October 18, 2017 – Solera Health (Solera), a preventive care benefits manager, today announced that they have joined forces with the American Diabetes Association (Association) to connect at-risk Americans to a Diabetes Prevention Program (DPP) recognized by the Centers for Disease Control and Prevention (CDC). The initiative seeks to help millions of Americans prevent or delay the onset of type 2 diabetes by providing a streamlined process for adults at risk for type 2 diabetes to enroll in a community or digital DPP.
The National DPP is a partnership of public and private organizations working together to reduce the growing number of Americans with and at risk for prediabetes and type 2 diabetes. There are currently more than 1,500 DPPs, including brick-and-mortar locations such as community non-profits, health systems, retail pharmacies and faith-based organizations, as well as virtual DPPs that deliver content to mobile devices, computers and/or telephones. All programs are working to make it easier for people at risk for type 2 diabetes to participate in affordable and high-quality lifestyle change programs to reduce their risk and improve their overall health.
According to the CDC’s National Diabetes Statistics Report, 2017, more than 84 million American adults are estimated to have prediabetes and are at risk for developing type 2 diabetes. Nearly 90 percent of American adults who have prediabetes are not aware they have it. Through this colla Continue reading

17 Tips to Manage Diabetes in Hot Weather or on Vacation

17 Tips to Manage Diabetes in Hot Weather or on Vacation

Summer brings special challenges for people with diabetes. These tips can help you safely manage diabetes in hot weather and when you’re on vacation.
The effects of heat are more strongly felt by people with type 1 or type 2 diabetes for several reasons:
Complications that are common for people with diabetes include damage to nerves or blood vessels. This damage means sweat glands cannot effectively cool the body. When the humidity is also high, sweat doesn’t evaporate and the body cannot cool itself. It’s easier for people with diabetes to develop heat exhaustion or heat stroke, according to the Centers for Disease Control and Prevention (CDC). Consider this to be a medical emergency and get help quickly.
They more quickly lose water from their bodies (become dehydrated) when it’s hot, compared to people without diabetes. They need to drink more fluids to keep blood sugar levels from increasing. High blood sugar increases the need to pee, making dehydration worse. Also, “water pills” (diuretics) used to treat high blood pressure can worsen dehydration.
Insulin use can change because hot weather changes how the body uses insulin. The CDC recommends testing your blood sugar more often, adjusting the insulin dose, and adjusting what you eat and drink.
Top tips for summer if you have diabetes:
Drink more fluids, especially water, even if you’re not thirsty. Waiting until you’re thirsty means you’re already dehydrated. Avoid alcoholic beverages and those with caffeine, including coffee, tea, energy or sports drinks and cola drinks. They can make you lose more Continue reading

Diabetic by HbA1c, Normal by OGTT: A Frequent Finding in the Mexico City Diabetes Study

Diabetic by HbA1c, Normal by OGTT: A Frequent Finding in the Mexico City Diabetes Study

The agreement between glucose-based and hemoglobin A1c (HbA1c)–based American Diabetes Association criteria in the diagnosis of normal glucose tolerance, prediabetes, or diabetes is under scrutiny. A need to explore the issue among different populations exists.
All 854 participants without known diabetes had both oral glucose tolerance test (OGTT) and HbA1c measurements on the same day of the 2008 phase.
We found by OGTT normal glucose tolerance (NGT) in 512 (59.9%) participants, prediabetes [impaired fasting glucose (IFG) and/or impaired glucose tolerance (IGT)] in 261 (30.5%), and diabetes in 81 (9.4%). In total, 232 in the NGT group (45.3%) and 158 in the prediabetes group (60.5%) had HbA1c ≥6.5%. Body mass index, waist circumference, and blood pressure were significantly different among OGTT-defined diabetic status groups but not in the HbA1c-diagnosed group. We identified 404 participants in the NGT group with confirmed NGT throughout all phases of the Mexico City Diabetes Study. Of these, 184 (45.5%) had HbA1c ≥6.5%. In a vital/diabetes status follow-up performed subsequently, we found that, of these, 133 remained nondiabetic, 3 had prediabetes, 7 had diabetes, and 13 had died without diabetes; we were unable to ascertain the glycemic status in 5 and vital status in 23.
The patient was a 47-year-old Japanese man whose parents were first cousins. He lacked secondary sexual characteristics owing to normosmic CHH. Exon segments for the KISS1R gene in this patient were screened for mutations. Functional analyses were performed using HEK293 cells expressing KISS1R mu Continue reading

Sugar, Uric Acid, and the Etiology of Diabetes and Obesity

Sugar, Uric Acid, and the Etiology of Diabetes and Obesity

The intake of added sugars, such as from table sugar (sucrose) and high-fructose corn syrup has increased dramatically in the last hundred years and correlates closely with the rise in obesity, metabolic syndrome, and diabetes. Fructose is a major component of added sugars and is distinct from other sugars in its ability to cause intracellular ATP depletion, nucleotide turnover, and the generation of uric acid. In this article, we revisit the hypothesis that it is this unique aspect of fructose metabolism that accounts for why fructose intake increases the risk for metabolic syndrome. Recent studies show that fructose-induced uric acid generation causes mitochondrial oxidative stress that stimulates fat accumulation independent of excessive caloric intake. These studies challenge the long-standing dogma that “a calorie is just a calorie” and suggest that the metabolic effects of food may matter as much as its energy content. The discovery that fructose-mediated generation of uric acid may have a causal role in diabetes and obesity provides new insights into pathogenesis and therapies for this important disease.
Fructose-induced weight gain and metabolic syndrome
Experimental studies from the 1950s showed the peculiar ability of fructose to induce insulin resistance in laboratory rats. Today, fructose intake has been shown to induce all features of metabolic syndrome in rats, as well as oxidative stress, endothelial dysfunction, fatty liver, microalbuminuria and kidney disease (rev. in 1). Similar findings can be shown when animals are fed sucrose or high-fructose corn s Continue reading

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