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Why Ketoacidosis Cause Coma

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Diabetic Coma Causes

Diabetics with prolonged blood-sugar extremes (either too high or too low blood-sugar level) may lead to a diabetic coma. Causes of Diabetes coma Many condition diabetes conditions lead to the cause of diabetes coma. They are: Diabetic ketoacidosis (DKA) - If the muscle cells become energy starved, and the body may respond by breaking down stored fats. Breaking down of fats produces a toxic acid known as ketones and this breakdown is called as ketoacidosis. If it left untreated, DKA can lead to a diabetic coma. DKA is most common among diabetes type 1, but can also affect the type 2 diabetes or gestational diabetes. Diabetic hyperosmolar syndrome - If the blood-sugar level rises to 600 milligrams per deciliter (mg/dL), or 33 millimoles per liter (mmol/L) it is called as diabetic hyperosmolar syndrome. Such a high level of glucose in blood makes the blood thicker like syrup. This excess sugar level is removed from the blood through the urine, which remove tremendous amounts of fluid from the body causing dehydration. If it left untreated diabetic hyperosomolar syndrome can lead to dangerous dehydration and coma. Hyperosmolar syndrome is more common among type 2 diabetes. Hypoglycemi Continue reading >>

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  1. Santosh Anand

    Insulin plays a key role in helping sugar (glucose) enter your cells, thus providing them energy. When your cells don't get the glucose they need for energy, your body begins to burn fat for energy, which produces ketones. Ketones are acidic and so when they build up in the blood, they make the blood more acidic, leading to the condition called diabetic ketoacidosis (DKA).
    Now, in type-1 diabetes, there is no insulin production whereas in type-2, there is impairment of insulin production. Thus why Type-2 diabetic people hardly get DKA.
    Note: Diabetic ketoacidosis is a serious condition that might lead to diabetic coma or even death.

  2. Lucas Verhelst

    In order for the cells in your body to access the glucose in your bloodstream so they can use it as energy they need insulin. Insulin acts like a key, opennin the cell door to allow the entry of glucose. Type 1 diabetics produce no insulin and need to inject it, thus the amount of insulin they have is strictly limited. Once they run out of insulin the glucose remains in the blood stream. If this occurs over a long period of time their blood glucose levels will rise due to the release of glucose from the liver. High blood sugar levels causes ketoacidosis which leads to coma and death.

  3. Keith Phillips

    Although type 2 diabetics suffer from insulin resistance, the condition rarely has an absolute negative effect on the bodies ability to convert glucose to usable energy. Type 1 diabetics have little or no ability to produce insulin. With the exception of neural cells, the rest of the body which without insulin is experiencing starvation, will consume its own tissues. (this is how people have endured periods of famine). This process however produces by products that eventually overwhelm the body's ability to process toxins.

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Diabetic Coma

Print Overview A diabetic coma is a life-threatening diabetes complication that causes unconsciousness. If you have diabetes, dangerously high blood sugar (hyperglycemia) or dangerously low blood sugar (hypoglycemia) can lead to a diabetic coma. If you lapse into a diabetic coma, you're alive — but you can't awaken or respond purposefully to sights, sounds or other types of stimulation. Left untreated, a diabetic coma can be fatal. The prospect of a diabetic coma is scary, but fortunately you can take steps to help prevent it. Start by following your diabetes treatment plan. Symptoms Before developing a diabetic coma, you'll usually experience signs and symptoms of high blood sugar or low blood sugar. High blood sugar (hyperglycemia) If your blood sugar level is too high, you may experience: Increased thirst Frequent urination Fatigue Nausea and vomiting Shortness of breath Stomach pain Fruity breath odor A very dry mouth A rapid heartbeat Low blood sugar (hypoglycemia) Signs and symptoms of a low blood sugar level may include: Shakiness or nervousness Anxiety Fatigue Weakness Sweating Hunger Nausea Dizziness or light-headedness Difficulty speaking Confusion Some people, especial Continue reading >>

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Popular Questions

  1. Santosh Anand

    Insulin plays a key role in helping sugar (glucose) enter your cells, thus providing them energy. When your cells don't get the glucose they need for energy, your body begins to burn fat for energy, which produces ketones. Ketones are acidic and so when they build up in the blood, they make the blood more acidic, leading to the condition called diabetic ketoacidosis (DKA).
    Now, in type-1 diabetes, there is no insulin production whereas in type-2, there is impairment of insulin production. Thus why Type-2 diabetic people hardly get DKA.
    Note: Diabetic ketoacidosis is a serious condition that might lead to diabetic coma or even death.

  2. Lucas Verhelst

    In order for the cells in your body to access the glucose in your bloodstream so they can use it as energy they need insulin. Insulin acts like a key, opennin the cell door to allow the entry of glucose. Type 1 diabetics produce no insulin and need to inject it, thus the amount of insulin they have is strictly limited. Once they run out of insulin the glucose remains in the blood stream. If this occurs over a long period of time their blood glucose levels will rise due to the release of glucose from the liver. High blood sugar levels causes ketoacidosis which leads to coma and death.

  3. Keith Phillips

    Although type 2 diabetics suffer from insulin resistance, the condition rarely has an absolute negative effect on the bodies ability to convert glucose to usable energy. Type 1 diabetics have little or no ability to produce insulin. With the exception of neural cells, the rest of the body which without insulin is experiencing starvation, will consume its own tissues. (this is how people have endured periods of famine). This process however produces by products that eventually overwhelm the body's ability to process toxins.

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What is DIABETIC KETOACIDOSIS? What does DIABETIC KETOACIDOSIS mean? DIABETIC KETOACIDOSIS meaning - DIABETIC KETOACIDOSIS definition - DIABETIC KETOACIDOSIS explanation. Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. SUBSCRIBE to our Google Earth flights channel - https://www.youtube.com/channel/UC6Uu... Diabetic ketoacidosis (DKA) is a potentially life-threatening complication of diabetes mellitus. Signs and symptoms may include vomiting, abdominal pain, deep gasping breathing, increased urination, weakness, confusion, and occasionally loss of consciousness. A person's breath may develop a specific smell. Onset of symptoms is usually rapid. In some cases people may not realize they previously had diabetes. DKA happens most often in those with type 1 diabetes, but can also occur in those with other types of diabetes under certain circumstances. Triggers may include infection, not taking insulin correctly, stroke, and certain medications such as steroids. DKA results from a shortage of insulin; in response the body switches to burning fatty acids which produces acidic ketone bodies. DKA is typically diagnosed when testing finds high blood sugar, low blood pH, and ketoacids in either the blood or urine. The primary treatment of DKA is with intravenous fluids and insulin. Depending on the severity, insulin may be given intravenously or by injection under the skin. Usually potassium is also needed to prevent the development of low blood potassium. Throughout treatment blood sugar and potassium levels should be regularly checked. Antibiotics may be required in those with an underlying infection. In those with severely low blood pH, sodium bicarbonate may be given; however, its use is of unclear benefit and typically not recommended. Rates of DKA vary around the world. About 4% of people with type 1 diabetes in United Kingdom develop DKA a year, while in Malaysia the condition affects about 25% a year. DKA was first described in 1886 and, until the introduction of insulin therapy in the 1920s, it was almost universally fatal. The risk of death with adequate and timely treatment is currently around 1–4%. Up to 1% of children with DKA develop a complication known as cerebral edema. The symptoms of an episode of diabetic ketoacidosis usually evolve over a period of about 24 hours. Predominant symptoms are nausea and vomiting, pronounced thirst, excessive urine production and abdominal pain that may be severe. Those who measure their glucose levels themselves may notice hyperglycemia (high blood sugar levels). In severe DKA, breathing becomes labored and of a deep, gasping character (a state referred to as "Kussmaul respiration"). The abdomen may be tender to the point that an acute abdomen may be suspected, such as acute pancreatitis, appendicitis or gastrointestinal perforation. Coffee ground vomiting (vomiting of altered blood) occurs in a minority of people; this tends to originate from erosion of the esophagus. In severe DKA, there may be confusion, lethargy, stupor or even coma (a marked decrease in the level of consciousness). On physical examination there is usually clinical evidence of dehydration, such as a dry mouth and decreased skin turgor. If the dehydration is profound enough to cause a decrease in the circulating blood volume, tachycardia (a fast heart rate) and low blood pressure may be observed. Often, a "ketotic" odor is present, which is often described as "fruity", often compared to the smell of pear drops whose scent is a ketone. If Kussmaul respiration is present, this is reflected in an increased respiratory rate.....

What Are Symptoms Of Diabetic Ketoacidosis?

Diabetic ketoacidosis is a serious complication typically faced by people with type-1 diabetes, which occurs when body starts running out of insulin. This condition can leave the patient in coma or even death if not treated immediately. Diabetic ketoacidosis occurs when body is lacking lacking insulin to allow sufficient glucose to enter cells. Therefore the body absorbs energy by burning fatty acids and in turn produce acidic ketone bodies. Blood containing high levels of ketone bodies can cause serious illness. Diabetic ketoacidosis is itself a symptom of undiagnosed type-1 diabetes. Other symptoms of diabetic ketoacidosis include: 1. Dehydration 2. Vomiting 3. Nausea 4. Blurred vision 5. Fatigue & sleepiness 6. Frequent urination 7. Rapid heartbeat 8. Unusual smell on the breath (similar to that of pear drops) 9. Confusion and disorientation 10. Hyperventilation or deep laboured breathing 11. Coma The symptoms of diabetic ketoacidosis usually develop over a 24-hour period if blood glucose levels are and remain very high (hyperglycemia). Continue reading >>

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  1. Steve Rapaport

    Correlation isn't causation, so the question's presupposition of high blood sugar 'resulting' in diabetic coma is wrong. They both result from common causes.
    Diabetic coma (advanced diabetic ketoacidosis or DKA) is not the result of high blood sugar, but of low insulin and water levels. Low insulin levels lead to high blood sugar AND to ketoacidosis. Hence there's no minimum blood sugar level to watch for (though there may be for a given individual).
    The best way to avoid DKA is to keep insulin levels steady in the bloodstream, keep well hydrated, and keep small amounts of food in the system at all times.
    DKA is a result of the body demanding sugar for fuel, and being denied it through lack of insulin. The body burns fat instead, which produces ketone bodies as a byproduct. The ketones build up in blood, making it acid and highly concentrated. Concentrated blood sucks water out of cells by osmosis. Dehydration makes this worse. The ketones signal the liver that glucose is desperately needed, so it dumps stored glucose to help out, but in the absence of insulin this just makes things worse -- now the blood is full of ketones AND glucose, and even more highly concentrated. Both of these conditions will get worse until fast-acting insulin and missing electrolytes are added in carefully controlled doses, including a drip-feed for hydration and frequent recheck and adjustment of all those values.

  2. Suhail Malhotra

    First we must know that there are 2 types of diabetes.
    IDDM(insulin dependent diabetes mellitus) aka Type 1
    NIDDM( non insulin dependent diabetes mellitus) aka Type 2.
    Type 1 is due to loss of insulin secretion by pancreas as in destruction of pancreas.
    Type 2 is due to insulin resistance that is insulin secretion is ok but body cells don't respond to it.
    Now the comas in these two types are different to the extent that they are named differently.
    The coma of type 1 is called the DKA(diabetic ketoacidosis) and that of type 2 is HONK( hyperosmotic non ketotic coma) now known as HHS(hyperglycemic hyperosmolar state).
    DKA occurs in type 1 diabetes or situations simulating type 1 mechanism like when a patient forgets to take his dose of insulin or in states when patient is regular with insulin but the body needs more than normal as in cases of surgery or illness or pregnancy.
    Blood glucose ranges in DKA from 250 to 600 mg/dl( 13 to 33 mmol/l) with increased ketones in blood which being acidic drive the blood ph to acidic levels ( <7.3). Symptoms include vomiting,increased urination, increased thirst, abdominal pain,increased rate of respiration(Kussumaul breathing) and in the end coma.
    HONK or HHS is caused by type 2 diabetes or situations similar to it like relative insulin deficiency combined with inadequate fluid intake and often precipitated in patients with type2 DM and a concurrent illness.
    Blood glucose ranges from 600 to 1200 mg/dl (33 to 66 mmol/l). The blood ph is normal (>=7.3) as ketones are absent. Patient is lethargic with increased thirst and increased urination leading to coma.
    Symptoms absent in HONK are nausea, vomiting, abdominal pain and increased rate of respiration which were very much a part of DKA.

  3. Jae Won Joh

    If you are asking what blood sugar levels are commonly seen in diabetic coma[1], there is a very wide range. Patients naive to the condition typically present with blood glucoses around the 300s, while those with chronic poorly-managed diabetes can present with blood glucoses over 1000.
    [1] As Steve Rapaport already pointed out in his answer, the high glucose level is not, in and of itself, the problem.

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