What Is The Opposite Of Acidosis?

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Respiratory acidosis #sign and symptoms of Respiratory acidosis Respiratory acidosis ABGs Analyse https://youtu.be/L5MWy1iHacI Plz share n subscribe my chanel is a condition that occurs when the lungs cant remove enough of the Suctioning https://youtu.be/hMJGkxvXTW0 carbon dioxide (CO2) produced by the body. Excess CO2 causes the pH of blood and other bodily fluids to decrease, making them too acidic. Normally, the body is able to balance the ions that control acidity. This balance is measured on a pH scale from 0 to 14. Acidosis occurs when the pH of the blood falls below 7.35 (normal blood pH is between 7.35 and 7.45).Rinku Chaudhary NSG officer AMU ALIGARH https://www.facebook.com/rinkutch/ Respiratory acidosis is typically caused by an underlying disease or condition. This is also called respiratory failure or ventilatory failure. Suctioning https://youtu.be/hMJGkxvXTW0 Normally, the lungs take in oxygen and exhale CO2. Oxygen passes from the lungs into the blood. CO2 passes from the blood into the lungs. However, sometimes the lungs cant remove enough CO2. This may be due to a decrease in respiratory rate or decrease in air movement due to an underlying condition such as: asthma COPD pneumonia sleep apnea TYPES Forms of respiratory acidosis There are two forms of respiratory acidosis: acute and chronic. Acute respiratory acidosis occurs quickly. Its a medical emergency. Left untreated, symptoms will get progressively worse. It can become life-threatening. Chronic respiratory acidosis develops over time. It doesnt cause symptoms. Instead, the body adapts to the increased acidity. For example, the kidneys produce more bicarbonate to help maintain balance. Chronic respiratory acidosis may not cause symptoms. Developing another illness may cause chronic respiratory acidosis to worsen and become acute respiratory acidosis. SYMPTOMS Symptoms of respiratory acidosis Initial signs of acute respiratory acidosis include: headache anxiety blurred vision restlessness confusion Without treatment, other symptoms may occur. These include: https://www.healthline.com/health/res... sleepiness or fatigue lethargy delirium or confusion shortness of breath coma The chronic form of respiratory acidosis doesnt typically cause any noticeable symptoms. Signs are subtle and nonspecific and may include: memory loss sleep disturbances personality changes CAUSES Common causes of respiratory acidosis The lungs and the kidneys are the major organs that help regulate your bloods pH. The lungs remove acid by exhaling CO2, and the kidneys excrete acids through the urine. The kidneys also regulate your bloods concentration of bicarbonate (a base). Respiratory acidosis is usually caused by a lung disease or condition that affects normal breathing or impairs the lungs ability to remove CO2. Some common causes of the chronic form are: asthma chronic obstructive pulmonary disease (COPD) acute pulmonary edema severe obesity (which can interfere with expansion of the lungs) neuromuscular disorders (such as multiple sclerosis or muscular dystrophy) scoliosis Some common causes of the acute form are: lung disorders (COPD, emphysema, asthma, pneumonia) conditions that affect the rate of breathing muscle weakness that affects breathing or taking a deep breath obstructed airways (due to choking or other causes) sedative overdose cardiac arrest DIAGNOSIS How is respiratory acidosis diagnosed? The goal of diagnostic tests for respiratory acidosis is to look for any pH imbalance, to determine the severity of the imbalance, and to determine the condition causing the imbalance. Several tools can help doctors diagnose respiratory acidosis. Blood gas measurement Blood gas is a series of tests used to measure oxygen and CO2 in the blood. A healthcare provider will take a sample of blood from your artery. High levels of CO2 can indicate acidosis.

Respiratory And What Is The Opposite Of Acidosis

Respiratory and what is the opposite of acidosis? Alkalosis, and vice versa. 2. Example: if you have metabolic acidosis (decrease bicarb) what do we have to do with the pCO2? We have to get rid of it. If we decrease the nominator, we have to decrease the dominator in order for the equation to stay the same. Therefore, we have to blow off the CO2(hyperventilation). 3. Ventilation is a CO2term! Hyperventilation = Increase in respiratory rate allows for the blowing off of CO2, therefore results in respiratory alkalosis. For the treatment of respiratory alkalosis is to give the pt a paper bag and ask to breath in it, b/c then they are re-breathing their own CO2. Hypoventilation = Decrease in respiratory rate allows for the retention of CO2, therefore results in respiratory acidosis. Full compensation does not exist; you never bring back the pH to the normal range. There is one exception: chronic respiratory alkalosis in high altitude; ie mountain sickness (ie peru). C. Respiratory conditions: acidosis and alkalosis 1. Things that deal with CO2: a) Respiratory center is in medulla oblongata, which controls the breathing rate b) Upper airways if obstructed, there will be a problem getti Continue reading >>

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  1. PPumpItUp

    I have been reading conflicting information on blogs and threads. Some people say that too much protein will kick you out of ketosis, others say that you need excess protein. I lift weights so I try to take 1g for each pound of lean body mass so that around 220g of protein per day. I think I should be safe with this no matter which theory you go by, correct?

  2. PPumpItUp

    I guess the protein is supposed to convert to glucose but even that has controversies around it. Some people say it does happen and others say it does not.

  3. MizTenaciousT

    I've heard different things about this too. I'm hoping some of the more experienced ketoers will chime in. From what I understand, as long as you're staying close to the traditional 5-20-75 CPF ratio, with carbs under 50g, you should be good.

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Abg Analysis Article

the"6 Easy Steps to ABG Analysis" were developed to provide nurses with what? an accurate and systematic method of easily interpreting arterial blood gases 5. Does the CO2 or the HCO3 go the opposite direction of the ph? 6. Are the pO2 and the O2 saturation normal? Normal blood pH is 7.4, plus or minus 0.05, forming the range 7.35 to 7.45. If blood pH falls below 7.35 it is acidic. If blood pH rises above 7.45, it is alkalotic. If it falls in the normal range, label what side of 7.4 it falls on. Lower than 7.4 is normal/acidotic, and higher than 7.4 is normal/alkalotic. Label it. Normal pCO2 levels are 35-45 mmHg. Below 35 is alkalotic and above 45 is acidic. Label it. A Normal HCO3 level is 22-26 mEq/L. If below 22 then pt is acidic. If the HCO3is above 26, the patient is alkalotic. Label it. Step 4: Match the CO2 or the HCO3 with the pH Next match either the pCO2 or the HCO3 with the pH to determine the acid-base disorder. For example, if the pH is acidotic, the CO2 is acidotic, then the acid-base disturbance is being caused by the respiratory system = respiratory acidosis. If the pH is alkalotic and the HCO3 is alkalotic the acid-base disturbance is being caused by metabolic (r Continue reading >>

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  1. kailey_elise

    I have a friend. I noticed while kissing him last night that, a few different times, I noticed a very distinctive acetone smell/taste on his breath.
    While most assuredly an alcoholic, he's never been one in a point of desperation to the point of, say, drinking IPA or something. He drinks beer, and only the same kind of beer (no mixing brands or anything).
    My (limited) research shows that acetone is a metabolic byproduct of drinking IPA (uh, meaning isopropyl alcohol, you know, rubbing alcohol - not a nice IPA beer, hahaha), but haven't really noticed anything about ethyl alcohol/ethanol & acetone production.
    Does anyone know anything about why this might be? He's not a huge guy, so it's good he sticks to beer, but can easily (& somewhat often does) get fucked up to the point of being incomprehensible.
    A potentially important point to note is that he is on some *serious* antibiotics. Like, some hardcore, there aren't things much stronger, antibiotics. He has some kind of bone infection or something that they're trying to keep under control with the meds for as long as they can. I don't know if something like that could be affecting the way he processes the alcohol?? Maybe?
    No known immune system issues (barring any that could come from the bone thingy & antibiotics). No other illicit drugs other than cannabis is being consumed, the only other drugs are the aforementioned ABs.
    I really like this guy, and it's worrisome to me to notice this (not to mention it kinda tastes weird, hahaha). I know alcohol isn't good for you, and as an alcoholic he probably has all kinds of liver damage (or will soon), but I'm just worried that there is *extra* damage being done, since he is burning off acetone when he's drinking.
    Thanks in advance for ANY help anyone can provide, even if it's just better search terms to try or other websites to read...ANYTHING would be helpful, please!!

  2. Down In Albion

    Interesting question, the thing that instantly sprung to mind when you mentioned the Acetone smell is Ketosis, which in case you didn't know is a process of the body when you starve it of carbs (like when you're on the atkins diet). It creates a sickly sweet solvent taste very similar to Acetone in my opinion.
    Alcohol contains sugar, and so carbs, but maybe if he has a low carb diet, combined with tolerance to huge amounts of sugar from daily binge drinking, that would possibly allow his body to be in a state of ketosis.
    It's a long shot but it's the only explanation I can think of other than it just being a direct effect of the antibiotics, many can do odd things like change the colour of your pee or sweat etc... so why not breath?
    I can't remember the name, but there's one antibiotic that makes your TEARS turn red lol. So I'd guess it's highly possible that one could alter a persons breath!
    Edit: I need to learn to read things twice when I'm spun. The part about Acetone being a bi-product of IPA totally went in and out lol. I'll have a look, see if I find anything.
    More Editing:
    Ketone bodies
    From Wikipedia, the free encyclopedia
    Ketone bodies are three water-soluble compounds that are produced as by-products when fatty acids are broken down for energy in the liver and kidney. They are used as a source of energy in the heart and brain. In the brain, they are a vital source of energy during fasting. Although termed "bodies", they are dissolved substances, not particles.
    The three endogenous ketone bodies are acetone, acetoacetic acid, and beta-hydroxybutyric acid, although beta-hydroxybutyric acid is not technically a ketone but a carboxylic acid. Other ketone bodies such as beta-ketopentanoate and beta-hydroxypentanoate may be created as a result of the metabolism of synthetic triglycerides such as triheptanoin.
    Uses in the heart and brain
    Ketone bodies can be used for energy. Ketone bodies are transported from the liver to other tissues, where acetoacetate and beta-hydroxybutyrate can be reconverted to acetyl-CoA to produce energy, via the citric acid cycle.
    The heart gets little energy from ketone bodies except under special circumstances; it uses mainly fatty acids.
    The brain gets its energy from ketone bodies when glucose is less available (e.g., during fasting, strenuous exercise, low carbohydrate, ketogenic diet and in neonates). In the event of low blood glucose, most other tissues have additional energy sources besides ketone bodies (such as fatty acids), but the brain does not. After the diet has been changed to lower blood glucose for 3 days, the brain gets 25% of its energy from ketone bodies. After about 4 days, this goes up to 70% (during the initial stages the brain does not burn ketones, since they are an important substrate for lipid synthesis in the brain).
    Ketone bodies are produced from acetyl-CoA (see ketogenesis) mainly in the mitochondrial matrix of hepatocytes when carbohydrates are so scarce that energy must be obtained from breaking down fatty acids. Because of the high level of acetyl CoA present in the cell, the pyruvate dehydrogenase complex is inhibited, whereas pyruvate carboxylase becomes activated. Thus, the oxaloacetate produced will enter gluconeogenesis rather than the citric acid cycle, as the latter is also inhibited by the elevated level of NADH resulting from ß-oxidation of fatty acids. The excess acetyl-CoA is therefore rerouted to ketogenesis. Such a state in humans is referred to as the fasted state.
    Acetone is produced by spontaneous decarboxylation of acetoacetate, yielding levels of acetone much lower than those of other ketone bodies. Acetone cannot be converted back to acetyl-CoA, so it is excreted in the urine, or (as a consequence of its high vapor pressure) exhaled. Acetone is responsible for the characteristic "fruity" odor of the breath of persons in ketoacidosis.
    Click to expand... Down In Albion added 13 Minutes and 11 Seconds later...
    I guess you don't have too much to worry about (other than the obvious issues of alcoholism), as it's a naturally occurring thing.

  3. beentheredonethatagain

    maybe it was speed breath, you know it could seem like acetone and really be another chemical, just a thought ,

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What is ALKALOSIS? What does ALKALOSIS mean? ALKALOSIS meaning - ALKALOSIS pronunciation - ALKALOSIS definition - ALKALOSIS explanation - How to pronounce ALKALOSIS? Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. SUBSCRIBE to our Google Earth flights channel - https://www.youtube.com/channel/UC6Uu... Alkalosis is the result of a process reducing hydrogen ion concentration of arterial blood plasma (alkalemia). In contrast to acidemia (serum pH 7.35 or lower), alkalemia occurs when the serum pH is higher than normal (7.45 or higher). Alkalosis is usually divided into the categories of respiratory alkalosis and metabolic alkalosis or a combined respiratory/metabolic alkalosis. Respiratory alkalosis is caused by hyperventilation, resulting in a loss of carbon dioxide. Compensatory mechanisms for this would include increased dissociation of the carbonic acid buffering intermediate into hydrogen ions, and the related excretion of bicarbonate, both of which lower blood pH. Hyperventilation-induced alkalosis can be seen in several deadly central nervous system diseases such as strokes or Rett syndrome. Metabolic alkalosis can be caused by repeated vomiting, resulting in a loss of hydrochloric acid within the stomach content. Severe dehydration, and the consumption of alkali are other causes. It can also be caused by administration of diuretics and endocrine disorders such as Cushing's syndrome. Compensatory mechanism for metabolic alkalosis involve slowed breathing by the lungs to increase serum carbon dioxide, a condition leaning toward respiratory acidosis. As respiratory acidosis often accompanies the compensation for metabolic alkalosis, and vice versa, a delicate balance is created between these two conditions. Metabolic alkalosis is usually accompanied by low blood potassium concentration, causing, e.g., muscular weakness, muscle pain, and muscle cramps (from disturbed function of the skeletal muscles), and muscle spasms (from disturbed function of smooth muscles). It may also cause low blood calcium concentration. As the blood pH increases, blood transport proteins, such as albumin, become more ionized into anions. This causes the free calcium present in blood to bind more strongly with albumin. If severe, it may cause tetany.


Also found in: Dictionary, Thesaurus, Encyclopedia, Wikipedia. Related to alkalosis: metabolic acidosis, metabolic alkalosis, respiratory alkalosis al·ka·lo·sis (al-kă-lō'sis), Do not confuse this word with ankylosis. A state characterized by a decrease in the hydrogen ion concentration of arterial blood below the normal level, 40 nmol/L, or pH over 7.4. The condition may be caused by an increase in the concentration of alkaline compounds, or by a decrease in the concentration of acidic compounds or carbon dioxide. alkalosis /al·ka·lo·sis/ (al″kah-lo´sis) a pathologic condition due to accumulation of base in, or loss of acid from, the body. Cf. acidosis. alkalot´ic altitude alkalosis increased alkalinity in blood and tissues due to exposure to high altitudes. compensated alkalosis a form in which compensatory mechanisms have returned the pH toward normal. hypochloremic alkalosis metabolic alkalosis marked by hypochloremia together with hyponatremia and hypokalemia, resulting from the loss of sodium chloride and hydrochloric acid due to prolonged vomiting. metabolic alkalosis a disturbance in which the acid-base status shifts toward the alkaline side because of retention Continue reading >>

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  1. chaiminda

    I understand that the theory is that if I am passing ketones, then my body is burning fat and protein for energy. My doctor says that might mean I'm not getting enough energy, period. Okay, but I am gaining weight and I have as much energy as most 8-months-pregnant women. Baby is a normal size as far as can be determined. Doctor said she doubted that anything I could do at this stage would be a problem for the baby, but said vaguely that it might cause "metabolic issues" or problems with breastfeeding supply for me.
    Are there any scientific studies that actually make it clear what the danger is here?
    (I'm not that interested in "well, my doctor 10 years ago said x" or "my equally vague understanding is..." type replies. I want science.)

  2. peacheater

    Doctors tend to be more scared of ketones than they should be because they're also produced as a result of ketoacidosis, which is a very bad sign in people affected by Type I Diabetes (i.e. not the kind you have, but the kind where insulin is unable to be produced). It is also produced as a result of ketosis, which is when your body is burning fat for fuel, such as on a low-carb diet. This fat could be from your diet or from your body - so I could see how seeing ketones in the urine would be a sign that a person was not getting enough calories if that person was on a regular carbohydrate-heavy diet. Others can weigh in on whether there are pregnancy-specific factors regarding ketones that should be considered, but there are good reasons to be skeptical of doctors who raise alarms regarding ketones, because they've had it drilled into them that ketones = bad, since they most often arise as a reason of ketoacidosis.

  3. instamatic

    PubMed has a lot of studies on this, but most seem vague or 10-20 years old. I found one mouse study that suggests that organ growth is the main concern (most specifically, size of he heart and brain). I read one note about excessive ketones causing similar symptoms to Fetal Alcohol Syndrome. And this:
    "Maternal ketonuria or acetonuria during pregnancy is a concern because it can result in neonatal or childhood neurocognitive dysfunction."
    Here is a more lay-friendly write up that has a bibliography for additional reading.
    However, the vast majority of abstracts in PubMed seem to echo your doctor-- concern abou maternal metabolic issues and breastfeeding. And none of it seems to be studied independent of general gestational diabetes risks. (I don't know enough about fetal organ development to know risks at 8 months pregnant, but hopefully someone with more pregnancy-specific medical knowledge can back up/interpret the studies.)

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