Uncompensated Metabolic Acidosis

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Respiratory acidosis #sign and symptoms of Respiratory acidosis Respiratory acidosis ABGs Analyse https://youtu.be/L5MWy1iHacI Plz share n subscribe my chanel is a condition that occurs when the lungs cant remove enough of the Suctioning https://youtu.be/hMJGkxvXTW0 carbon dioxide (CO2) produced by the body. Excess CO2 causes the pH of blood and other bodily fluids to decrease, making them too acidic. Normally, the body is able to balance the ions that control acidity. This balance is measured on a pH scale from 0 to 14. Acidosis occurs when the pH of the blood falls below 7.35 (normal blood pH is between 7.35 and 7.45).Rinku Chaudhary NSG officer AMU ALIGARH https://www.facebook.com/rinkutch/ Respiratory acidosis is typically caused by an underlying disease or condition. This is also called respiratory failure or ventilatory failure. Suctioning https://youtu.be/hMJGkxvXTW0 Normally, the lungs take in oxygen and exhale CO2. Oxygen passes from the lungs into the blood. CO2 passes from the blood into the lungs. However, sometimes the lungs cant remove enough CO2. This may be due to a decrease in respiratory rate or decrease in air movement due to an underlying condition such as: asthma COPD pneumonia sleep apnea TYPES Forms of respiratory acidosis There are two forms of respiratory acidosis: acute and chronic. Acute respiratory acidosis occurs quickly. Its a medical emergency. Left untreated, symptoms will get progressively worse. It can become life-threatening. Chronic respiratory acidosis develops over time. It doesnt cause symptoms. Instead, the body adapts to the increased acidity. For example, the kidneys produce more bicarbonate to help maintain balance. Chronic respiratory acidosis may not cause symptoms. Developing another illness may cause chronic respiratory acidosis to worsen and become acute respiratory acidosis. SYMPTOMS Symptoms of respiratory acidosis Initial signs of acute respiratory acidosis include: headache anxiety blurred vision restlessness confusion Without treatment, other symptoms may occur. These include: https://www.healthline.com/health/res... sleepiness or fatigue lethargy delirium or confusion shortness of breath coma The chronic form of respiratory acidosis doesnt typically cause any noticeable symptoms. Signs are subtle and nonspecific and may include: memory loss sleep disturbances personality changes CAUSES Common causes of respiratory acidosis The lungs and the kidneys are the major organs that help regulate your bloods pH. The lungs remove acid by exhaling CO2, and the kidneys excrete acids through the urine. The kidneys also regulate your bloods concentration of bicarbonate (a base). Respiratory acidosis is usually caused by a lung disease or condition that affects normal breathing or impairs the lungs ability to remove CO2. Some common causes of the chronic form are: asthma chronic obstructive pulmonary disease (COPD) acute pulmonary edema severe obesity (which can interfere with expansion of the lungs) neuromuscular disorders (such as multiple sclerosis or muscular dystrophy) scoliosis Some common causes of the acute form are: lung disorders (COPD, emphysema, asthma, pneumonia) conditions that affect the rate of breathing muscle weakness that affects breathing or taking a deep breath obstructed airways (due to choking or other causes) sedative overdose cardiac arrest DIAGNOSIS How is respiratory acidosis diagnosed? The goal of diagnostic tests for respiratory acidosis is to look for any pH imbalance, to determine the severity of the imbalance, and to determine the condition causing the imbalance. Several tools can help doctors diagnose respiratory acidosis. Blood gas measurement Blood gas is a series of tests used to measure oxygen and CO2 in the blood. A healthcare provider will take a sample of blood from your artery. High levels of CO2 can indicate acidosis.

Respiratory Acidosis

Practice Essentials Respiratory acidosis is an acid-base balance disturbance due to alveolar hypoventilation. Production of carbon dioxide occurs rapidly and failure of ventilation promptly increases the partial pressure of arterial carbon dioxide (PaCO2). [1] The normal reference range for PaCO2 is 35-45 mm Hg. Alveolar hypoventilation leads to an increased PaCO2 (ie, hypercapnia). The increase in PaCO2, in turn, decreases the bicarbonate (HCO3–)/PaCO2 ratio, thereby decreasing the pH. Hypercapnia and respiratory acidosis ensue when impairment in ventilation occurs and the removal of carbon dioxide by the respiratory system is less than the production of carbon dioxide in the tissues. Lung diseases that cause abnormalities in alveolar gas exchange do not typically result in alveolar hypoventilation. Often these diseases stimulate ventilation and hypocapnia due to reflex receptors and hypoxia. Hypercapnia typically occurs late in the disease process with severe pulmonary disease or when respiratory muscles fatigue. (See also Pediatric Respiratory Acidosis, Metabolic Acidosis, and Pediatric Metabolic Acidosis.) Acute vs chronic respiratory acidosis Respiratory acidosis can be acut Continue reading >>

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    I have had great success with two rounds of Atkins in the past (each time losing over 50lb).
    But I have had two recent attempts to get back into the diet (on a quite rigorous induction regimen) and it is like the "mechanism" has broken. My body is not going into ketosis after 2 or even 3 weeks on the diet and (unsurprisingly) this is also reflected by lack of weight loss or measurements loss; and, indeed, on the latest attempt . . . I put ON three pounds. Whereas previously ketone strips were highly reactive (bright pink) they did not show ANY color at all after the induction periods. Obviously, absent the fat-burning, then a relatively fat-heavy intake is bound to result in weight gain. I also do not have that sense of increased energy that comes with being in fat-burning mode.
    It is almost as if my body has learned something about the diet and it will no longer cooperate with it. Either that, or it is something to do with being older (near 60).
    Has anyone else seen this phenomenon, and have they found any strategy to get around it.
    I always felt Atkins was a reliable way for me to be able to shed pounds, but without its "magic bullet" I feel rather lost as to what to try, as I have had very bad lucky with restrictive calorie (and other) diets.
    [ed. note: MICHAEL (2768058) last edited this post 8 months, 4 weeks ago.]

  2. Kathryn

    Ketostix are unreliable, throw them away and just follow the rules. If you do, then you have to be in ketosis because the body doesn't have carbs to burn for energy so it uses fat instead - if it didn't then you'd be dead!
    Putting on 3 pounds suggests that either you're eating too much, eating the wrong things or have some intolerance to something you're having. Without knowing what you eat on a typical day and how much you eat, then we can't really help. Post a list of what you're eating and we'll take a look.

  3. Ellen

    If you aren't getting into ketosis (and Kathryn is right about the ketostix) then somewhere you're taking in too many carbs and/or too much protein. If you are actually in ketosis but not losing...Or indeed gaining, then you are either sensitive to something you're eating...Like sweeteners for instance, or you are generally eating too much.
    Post a days menu with quantities and we'll take a look

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What is CLINICAL PATHWAY? What does CLINICAL PATHWAY mean? CLINICAL PATHWAY meaning - CLINICAL PATHWAY definition - CLINICAL PATHWAY explanation. Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. SUBSCRIBE to our Google Earth flights channel - https://www.youtube.com/channel/UC6Uu... A clinical pathway, also known as care pathway, integrated care pathway, critical pathway, or care map, is one of the main tools used to manage the quality in healthcare concerning the standardisation of care processes. It has been shown that their implementation reduces the variability in clinical practice and improves outcomes. Clinical pathways aim to promote organised and efficient patient care based on evidence-based medicine, and aim to optimise outcomes in settings such as acute care and home care. A single clinical pathway may refer to multiple clinical guidelines on several topics in a well specified context. A clinical pathway is a multidisciplinary management tool based on evidence-based practice for a specific group of patients with a predictable clinical course, in which the different tasks (interventions) by the professionals involved in the patient care are defined, optimized and sequenced either by hour (ED), day (acute care) or visit (homecare). Outcomes are tied to specific interventions. The concept of clinical pathways may have different meanings to different stakeholders. Managed care organizations often view clinical pathways in a similar way as they view care plans, in which the care provided to a patient is definitive and deliberate. Clinical pathways can range in scope from simple medication utilization to a comprehensive treatment plan. Clinical pathways aim for greater standardization of treatment regimens and sequencing as well as improved outcomes, from both a quality of life and a clinical outcomes perspective. Clinical pathways (integrated care pathways) can be seen as an application of process management thinking to the improvement of patient healthcare. An aim is to re-center the focus on the patient's overall journey, rather than the contribution of each specialty or caring function independently. Instead, all are emphasised to be working together, in the same way as a cross-functional team. More than just a guideline or a protocol, a care pathway is typically recorded in a single all-encompassing bedside document that will stand as an indicator of the care a patient is likely to be provided in the course of the pathway going forward; and ultimately as a single unified legal record of the care the patient has received, and the progress of their condition, as the pathway has been undertaken. The pathway design tries to capture the foreseeable actions which will most commonly represent best practice for most patients most of the time, and include prompts for them at the appropriate time in the pathway document to ascertain whether they have been carried out, and whether results have been as expected. In this way results are recorded, and important questions and actions are not overlooked. However, pathways are typically not prescriptive; the patient's journey is an individual one, and an important part of the purpose of the pathway documents is to capture information on "variances", where due to circumstances or clinical judgment different actions have been taken, or different results unfolded. The combined variances for a sufficiently large population of patients are then analysed to identify important or systematic features, which can be used to improve the next iteration of the pathway.

Metabolic Acidosis | Pathway Medicine

Metabolic Acidosis is a pathophysiological category of acidosis that refers to any cause of decreased ECF pH not due to a ventilatory defect (i.e. Respiratory Acidosis). Although the primary metabolic disturbance can cause a significant decrease in blood pH, respiratory compensatory mechanisms can largely correct the pH over several hours. The fundamental primary disturbance in a metabolic acidosis is a decrease in the levels of ECF bicarbonate concentration ([HCO3-]). Decreased bicarbonate results in an misalignment of the Henderson-Hasselbalch Equation for the bicarbonate buffer which largely determines the pH of the extracellular fluid. Mathematically, the reduced ECF pH results from an increase in the ratio between the partial pressure of arterial carbon dioxide (PaCO2) relative to the ECF concentration of bicarbonate ([HCO3-]). More colloquially, metabolic acidoses are caused by a pathologic consumption of the weak base form of the bicarbonate buffer, that is bicarbonate (HCO3-), resulting in a decrease in ECF pH. Metabolic Acidoses can be compensated by the actions of the lungs which serve to realign the bicarbonate buffer Henderson-Hasselbalch Equation over a period of hour Continue reading >>

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  1. Frentzke

    I'm curious to hear the experiences of others with distance running while being Keto. I'm in the second week of training for a half marathon in April. I've had no problem running 3 miles fasted for around 22 hours, but I'm interested in the possible need to fuel with fat on longer runs. Last October I completed a 10k after doing a fat fast for 5 days and then the morning of the race ate three eggs fried in a ton of butter. I actually beat my best training time by a decent amount with this plan, but 13.1 miles could require a different strategy. Thanks for any tips and Keto on!

  2. bodwest

    Check this podcast out - it has information that should be very helpful: https://blog.bulletproof.com/102-extreme-endurance-training-and-ketosis-with-ben-greenfield-podcast/

  3. Jacquie

    I think @trekkin1 is a keto long distance runner and @Emacfarland is a shorter keto distance runner. Hopefully, they will chime in soon with lots of good information.

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Anion gap usmle - anion gap metabolic acidosis normal anion gap metabolic acidosis

Metabolic Acidosis Treatment & Management

Approach Considerations Treatment of acute metabolic acidosis by alkali therapy is usually indicated to raise and maintain the plasma pH to greater than 7.20. In the following two circumstances this is particularly important. When the serum pH is below 7.20, a continued fall in the serum HCO3- level may result in a significant drop in pH. This is especially true when the PCO2 is close to the lower limit of compensation, which in an otherwise healthy young individual is approximately 15 mm Hg. With increasing age and other complicating illnesses, the limit of compensation is likely to be less. A further small drop in HCO3- at this point thus is not matched by a corresponding fall in PaCO2, and rapid decompensation can occur. For example, in a patient with metabolic acidosis with a serum HCO3- level of 9 mEq/L and a maximally compensated PCO2 of 20 mm Hg, a drop in the serum HCO3- level to 7 mEq/L results in a change in pH from 7.28 to 7.16. A second situation in which HCO3- correction should be considered is in well-compensated metabolic acidosis with impending respiratory failure. As metabolic acidosis continues in some patients, the increased ventilatory drive to lower the PaCO2 m Continue reading >>

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  1. tigherblood

    So I've been trying to specify my macros more and I'm a little stuck on how much protein I should be eating. My weight is 184 and I would estimate myself to be about 45% body fat percentage and I'm going to put down sedentary to be conservative, but I'm getting a few different answers from different sources. I looked into a couple of formulas as well as a few keto calculators. I'm basically getting anywhere from 60g of protein a day (I find this really hard to believe because I just can't seem to figure out a way to eat 1300 cal, under 20g carbs, and under 60g protein.)
    At the same time, I'm nervous that eating up to 90g of protein will be too much! In addition on formula I did said that it should be .6 of your weight?? That's over 100g of protein for me!
    Can anyone shed light to this?

  2. anbeav

    Use the keto calculator in the side bar and set it based on activity and lean body mass
    Sedentary: 0.8 grams per lb LBM
    Moderate endurance/strength training: 0.8 to 1 gram per lb LBM
    What are your stats? Details would be helpful to know if 100 grams is too much
    It's best to follow your protein goal, but you have to massively overeat protein to affect ketosis so don't worry too much about this once it's set.

  3. tigherblood

    Keto Calculator for macros
    That's the first calculator I used and it gives me a range of 61-101g of protein. Now because I've chosen sedentary, it tells me I should go with 61g, but I found that really hard to stick with, so I looked at some other sources to help. My height is 5'4", by the way. Again, I'm just concerned that if I go up to 80 or 90g, which is more realistic (seriously 60g is like 4 pieces of chicken and a stalk of broccoli) that it'll be too much because I'm not being very active at the moment
    Thanks for your response, by the way!

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