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Renal Tubular Acidosis Workup

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Should Blood Gas Analysis Be Part Of The Diagnostic Workup Of Short Children? Auxological Data And Blood Gas Analysis In Children With Renal Tubular Acidosis

Should Blood Gas Analysis Be Part of the Diagnostic Workup of Short Children? Auxological Data and Blood Gas Analysis in Children with Renal Tubular Acidosis Mul D.a Grote F.K.a Goudriaan J.R.a de Muinck Keizer-Schrama S.M.P.F.b Wit J.M.a Oostdijk W.a I have read the Karger Terms and Conditions and agree. I have read the Karger Terms and Conditions and agree. Buy a Karger Article Bundle (KAB) and profit from a discount! If you would like to redeem your KAB credit, please log in . Save over 20% compared to the individual article price. Buy Cloud Access for unlimited viewing via different devices Access to all articles of the subscribed year(s) guaranteed for 5 years Unlimited re-access via Subscriber Login or MyKarger Unrestricted printing, no saving restrictions for personal use * The final prices may differ from the prices shown due to specifics of VAT rules. For additional information: Background: Renal tubular acidosis (RTA) is a rare cause of growth failure, therefore it is uncertain whether routine screening with blood gas analysis of short infants and children is cost-effective. Objective: To investigate the clinical, growth and laboratory parameters in children with RTA to Continue reading >>

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  1. I.M.MAGIC

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  2. MIMISOCKS

    Thank you all for your expressions of concern. My brother is still in a coma, and on dialysis because of the DKA. One thing which has come out of this ordeal is the dialogue among his friends who have no idea things like this can happen to the young. His crisis is drawing the people who love him together in a positive way. They have been an inspiration to me to keep strong for his sake.
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This video provides you with a quick overview on not only the workup but also the treatment of hyperkalemia. Watch my video on hypokalemia! https://youtu.be/ous3RN7MKgo ECG changes in hyperkalemia: https://youtu.be/kfr19odmFEI Get it touch! Website http://www.crit-ic.com Twitter http://www.twitter.com/Crit_IC Facebook http://www.facebook.com/critic.medicine Instagram http://www.instagram.com/crit.ic

Workup For Mild Hyperkalemia

A 63-year-old asymptomatic man has had mild hyperkalemia (5.6-6.0 mEq/L) for the past six months. His physical examination and lab work are remarkable only for an elevated cholesterol level. He is a heavy smoker. Does this patient require workup for Addisons disease? How would you proceed? Embedded within this complex question are two separate but potentially interrelated issues: persistent hyperkalemia and the specter of adrenal insufficiency. First, hyperkalemia is rare in normal subjects. Your history should include a thorough search for medication use, both OTC and prescription (e.g., nonsteroidal anti-inflammatory drugs and diuretics). A set of normal laboratory values in this case should, at a minimum, exclude renal dysfunction, diabetes, and metabolic acidosis. In the absence of these abnormalities or rarer causes of hyperkalemia, such as selective defects in renal potassium handling and type I renal tubular acidosis, hypoaldosteronism should be considered. Hypoaldosteronism in the context of Addisons disease results from primary failure of the adrenal glands. While we most often consider this diagnosis with more dramatic presentations of hypotension and/or shock (adrenal c Continue reading >>

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  1. Eadem298

    Hey, how much have you lot on a strict low carb diet? Like Atkins or ketogenic diet in general...
    Avoiding carbs and sugar like fruits and simple carbs.
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  2. til_then

    This time around,23 pounds. It has taken a while though. About 3 months. But I'm older and have a really slow metabolism.

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    In my experience, not worth it. All the veggies out of my garden rotted in my fridge because I couldn't eat them.
    But you got to make that decision yourself. Myfitnesspal has Keto sub groups you could learn a lot on there from 'non-disordered' people.

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Hello, my name is Evgeny Romanov, I am third year medical student and in this video I gonna share with you an easy way to remember renal tubular acidosis for USMLE step 1 exam which I successfully passed this year. Subscribe and share with your mates; many more videos with mnemonics and simple ways to remember tough material are coming!

Renal Tubular Acidosis

Proximal RTA, defect in HCO3- reabsorption, mild hypokalemia results, Fanconi syndrome Mild hypokalemia comes from secondary aldosterone response to HCO3- loss Urine pH in acute vs. clinical presentation of Type II RTA Acute pH > 6.5 because of bicarbonate wasting Clinical pH < 5.5 because all of filtered HCO3- is removed and serum HCO3- is lower Multiple myeloma, heavy metals, carbonic anhydrase inhibitors, hereditary Weakness, bone pain, fractures due to osteomalacia (decreased synthesis of active vitamin D and phosphate wasting), imparied growth Hypokalemic distal RTA, inability to lower urine pH < 5.5. Decreased H+ secretion causes negative CCD and increased K+ secretion Alpha intercolated cell blockade: mutations in H+/K+ ATPase, carbonic anhydrase and AE1 bicarbonate/chloride antiport Calcium phosphate kidney stones, low urine citrate excretion Aldosterone deficient hyperkalemic RTA, ENaC expression decreases, decreased K+ and H+ secretion, usually mild/asymptomatic, urine pH < 5.5 b/c hyperkalemia supresses ammoniagenesis Aldosterone blockade: ACE inhibitors, ARBs, K+ sparing diuretics, abnormal aldosterone receptor. Also heparin ENaC deficiency or severe hypovolemia causes Continue reading >>

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  1. Smilescarlet

    I posted this in the woman's health section but got no replies and am still wondering about this. There is very little info about this online from what I found. I have been in ketosis for 24 days and have my lady friend almost two weeks early? Has this happened to anyone else? This never happens to me, I am like clockwork. Any thoughts?

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  3. AmyLiz

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    Proximal RTA, defect in HCO3- reabsorption, mild hypokalemia results, Fanconi syndrome Mild hypokalemia comes from secondary aldosterone response to HCO3- loss Urine pH in acute vs. clinical presentation of Type II RTA Acute pH > 6.5 because of bicarbonate wasting Clinical pH < 5.5 because all of filtered HCO3- is removed and serum HCO3- is lower Multiple myeloma, heavy metals, carbonic anhydrase inhibitors, hereditary Weakness, bone pain, fract ...

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