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Renal Tubular Acidosis Type 1

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==========/Credit========== No copyright infringement intended and I DO NOT OWN THIS. This video is just a TRANSLATED version, which is ONLY for sharing. /Oringnal Video: https://www.youtube.com/watch?v=R2mhS... Link to creator's channel: https://www.youtube.com/channel/UCgyr... ==========/Credit========== /Original Song: The Chainsmokers & Coldplay - Something Just Like This

Renal Tubular Acidosis: The Clinical Entity

Renal Tubular Acidosis: The Clinical Entity Department of Pediatrics, Hospital de Cruces, Vizcaya, Spain. Correspondence to Professor J. Rodrguez-Soriano, Department of Pediatrics, Hospital de Cruces, Plaza de Cruces s/n, Baracaldo, 48903 Vizcaya, Spain. Phone: 34-94-6006357; Fax: 34-94-6006044; E-mail: jsoriano{at}hcru.osakidetza.net The term renal tubular acidosis (RTA) is applied to a group of transport defects in the reabsorption of bicarbonate (HCO3), the excretion of hydrogen ion (H+), or both. This condition was first described in 1935 ( 1 ), confirmed as a renal tubular disorder in 1946 ( 2 ), and designated renal tubular acidosis in 1951 ( 3 ). The RTA syndromes are characterized by a relatively normal GFR and a metabolic acidosis accompanied by hyperchloremia and a normal plasma anion gap. In contrast, the term uremic acidosis is applied to patients with low GFR in whom metabolic acidosis is accompanied by normo- or hypochloremia and an increased plasma anion gap. The renal acid-base homeostasis may be broadly divided into two processes: (1) reabsorption of filtered HCO3, which occurs fundamentally in the proximal convoluted tubule; and (2) excretion of fixed acids throu Continue reading >>

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  1. __ish

    So, long story short, I remember reading somewhere that a great way to help speed your body's entry into ketosis is to do a 24 hour fast and some low intensity exercise. Trouble is, I can't seem to find where it was that I read that! Did I dream it up? Is this a thing?

  2. __loridcon

    i believe what you read is this:
    http://josepharcita.blogspot.com/2011/03/guide-to-ketosis.html#33EE

  3. __ish

    Yes! Thank you!
    I stopped eating at 3PM and did the wrong kind of exercise tonight instead of tomorrow morning, but that can be remedied.
    Has anyone had success with this method? This will be my first real go at keto.

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What is renal tubular acidosis (RTA)? RTA is a type of metabolic acidosis caused by the kidneys failure to properly acidify the urine. Find more videos at http://osms.it/more. Study better with Osmosis Prime. Retain more of what youre learning, gain a deeper understanding of key concepts, and feel more prepared for your courses and exams. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways and more when you follow us on social: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Thank you to our Patreon supporters: Sumant Nanduri Omar Berrios Alex Wright Sabrina Wong Suzanne Peek Arfan Azam Mingli Fng Osmosis's Vision: Empowering the worlds caregivers with the best learning experience possible.

Orphanet: Distal Renal Tubular Acidosis

Only comments seeking to improve the quality and accuracy of information on the Orphanet website are accepted. For all other comments, please send your remarks via contact us . Only comments written in English can be processed. Check this box if you wish to receive a copy of your message Distal renal tubular acidosis (dRTA) is a disorder of impaired net acid secretion by the distal tubule characterized by hyperchloremic metabolic acidosis. The classic form is often associated with hypokalemia whereas other forms of acquired dRTA may be associated with hypokalemia, hyperkalemia or normokalemia. Inheritance: Autosomal dominantorAutosomal recessiveorNot applicable Prevalence of dRTA is unknown but is often underreported. The hereditary forms of dRTA are more prevalent in areas of high consanguinity (Arabic peninsula and North Africa) whereas acquired dRTA has been reported more frequently in Western countries. Disease onset can occur at any age, depending on cause. Hereditary dRTA subtypes include autosomal dominant (AD) and autosomal recessive (AR) dRTA (see these terms). A recessive subtype of dRTA associated with anemia has also been described in Southeast Asia. AR forms are frequ Continue reading >>

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  1. rumSmuggler

    I recently (couple weeks ago) started weights and also taking some protein powder (low carb - 1,8g per serving).
    As suggested I raised my protein intake and I'm somewhere around 1 to 1.2 protein ratio. Last couple days I used ketostix to test my ketones and it was negative. I'm sure I didn't exceed 20g carbs per day because I track all my food.
    Could that higher protein intake kick me out of ketosis?

  2. gupe

    As far as I've been able to discover, there are no absolutely definitive answers to the excess protein => additional glucose => inhibition of ketosis? causal chain question.
    This is a good article: "If You Eat Excess Protein, Does It Turn Into Excess Glucose?" on ketotic.org.
    And here is a recent discussion on "After workout protein needs" on /r/ketogains.
    An important unresolved question is: is gluconeogenesis (the manufacture of new glucose by the liver using proteins and fat) a supply-driven process or a demand-driven process?
    If it is a supply-driven process, then it seems more plausible that excess consumption of protein will lead to higher blood sugar levels.
    But if it's demand-driven, then excess glucose might just be due to the slower removal of glucose from the blood-stream after protein has been eaten, causing a bit of a build-up.
    I think that it might vary a lot from person to person. The best is to measure your own blood ketone concentration before and after eating protein. (The ketostix method is not as reliable, particularly if you've just finished a work-out.)
    Edit: fixed link.

  3. darthluiggi

    It can, but it depends on various factors such as weight, activity level, etc.
    I asked the science behind it to to /u/gogge and he gave a very good explanation in another post.
    Fact is, if you are doing strength training you will need to increase your protein intake, otherwise you will not grow muscle. Also protein comes into play if you are eating at a deficit.
    If you are completely sure that protein is taking you out of ketosis, then drop your intake to 1.0 and see if you get back.
    How much do you weight, what % BF do you have, what kind of excercises are you doing and for how long?
    As a side note: don't rely on ketostsix to see if you are in or out of keto.
    *Edited for grammar.

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What Is Renal Failure: In this video, We will share information about what is renal failure - how to identify renal failure - symptoms of renal failure. Subscribe to our channel for more videos. Watch: (https://www.youtube.com/watch?v=ivQE7...) How to Identify Renal Failure Renal failure, also known as kidney failure, is a condition that can take two different forms: acute, when it presents itself very suddenly, and chronic, when it develops slowly over at least three months. Acute kidney failure has the potential to lead to chronic renal failure. During both types of renal failure your kidneys arent able to perform the necessary functions your body needs to stay healthy. Despite this similarity between types, the causes, symptoms, and treatments for the two kinds of renal failure vary significantly. Learning about the symptoms and causes of this disease and being able to differentiate between the two forms can be beneficial if you or a loved one have been diagnosed with renal failure. Thanks for watching what is renal failure - how to identify renal failure - symptoms of renal failure video and don't forget to like, comment and share. Related Searches: acute renal failure dr najeeb, acute renal failure explained clearly, acute renal failure kaplan, acute renal failure khan academy, acute renal failure lecture, acute renal failure management, acute renal failure medcram, acute renal failure nursing, acute renal failure treatment, acute renal failure usmle, chronic renal failure explained clearly, chronic renal failure khan academy, chronic renal failure lecture, chronic renal failure nursing, chronic renal failure treatment, chronic renal failure usmle, end stage renal failure, michael linares renal failure, pathophysiology of renal failure, renal failure, renal failure and abgs, renal failure and bone health, renal failure and dialysis, renal failure and electrolyte imbalances, renal failure and hyperkalemia, renal failure and hypocalcemia, renal failure and massage, renal failure and phosphorus, renal failure anemia, renal failure animation, renal failure bolin, renal failure calcium, renal failure care plan, renal failure case study presentation, renal failure cat, renal failure catheter, renal failure causes, renal failure causes hyperkalemia, renal failure chronic, renal failure concept map, renal failure cure, renal failure definition, renal failure diagnosis, renal failure diet, renal failure diet for humans, renal failure diet therapy, renal failure disease, renal failure dog, renal failure dr najeeb, renal failure due to ace inhibitor, renal failure electrolyte imbalance, renal failure examination, renal failure explained, renal failure fluid retention, renal failure for dummies, renal failure for nursing students, renal failure from ace inhibitor, renal failure funny, renal failure grinding, renal failure home remedy, renal failure homeopathic treatment, renal failure humans, renal failure hyperkalemia pathophysiology, renal failure icd 10, renal failure in cats, renal failure in children, renal failure in dogs, renal failure in hindi, renal failure in malayalam, renal failure in neonates, renal failure in sepsis, renal failure in the emergency department, renal failure in urdu, renal failure khan, renal failure khan academy, renal failure lab values, renal failure lecture, renal failure loss of appetite, renal failure made easy, renal failure malayalam, renal failure management, renal failure meaning in urdu, renal failure medcram, renal failure medications, renal failure metabolic acidosis, renal failure natural remedies, renal failure natural treatment, renal failure nclex, renal failure nclex questions, renal failure nucleus, renal failure nursing, renal failure nursing care plan, renal failure on dialysis, renal failure osce station, renal failure osmosis, renal failure pathology, renal failure pathophysiology, renal failure pathophysiology animation, renal failure patient, renal failure pbds, renal failure pharmacology, renal failure phases, renal failure physiology, renal failure prerenal intrarenal postrenal, renal failure pronunciation, renal failure quiz, renal failure registered nurse rn, renal failure shaking, renal failure skin itching, renal failure stage 3, renal failure stages, renal failure stories, renal failure support groups, renal failure swollen feet, renal failure symptoms, renal failure symptoms in cats, renal failure transplant, renal failure treatment, renal failure treatment in ayurveda, renal failure treatment in homeopathy, renal failure ultrasound, renal failure urine, renal failure usmle, renal failure vascular calcification, renal failure video, renal failure volume overload, renal failure vs ckd, renal failure youtube, renal kidney failure, stages of renal failure, symptoms of renal failure, types of renal failure, what is renal failure

Renal Tubular Disorders

Renal tubular disorders are a very heterogeneous group of hereditary and acquired diseases that involve singular or complex dysfunctions of transporters and channels in the renal tubular system. The disorders may lead to fluid loss and abnormalities in electrolyte and acid-base homeostasis. Renal tubular acidosis ( RTA ) refers to normal anion gap (hyperchloremic) metabolic acidosis in the presence of normal or almost normal renal function. The various types of RTA include proximal tubular bicarbonate wasting (type II), distal tubular acid secretion (type I), very rarely carbonic anhydrase deficiency (type III) , and aldosterone deficiency/resistance (type IV). X-linked hypophosphatemic rickets , the most common form of hereditary hypophosphatemic rickets , is caused by phosphate wasting and presents with hypophosphatemia and symptoms related to rickets . Bartter syndrome , Liddle, and Gitelman syndrome are inherited disorders of tubular function that are characterized by hypokalemia and metabolic alkalosis . Renal tubular disorders are suspected when characteristic clinical features and/or laboratory findings are present. The diagnosis of hereditary conditions is usually confirme Continue reading >>

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  1. spoiltmomof2

    For me it doesn't So by eating high fat if I eat until I'm full I lose weight very, very, very slowly because I'm taking in too many calories I'm assuming.
    I know everyone says Atkins needs to be high fat but why?
    Even if I stay below 20 grams of carbs a day I don't lose weight unless I stay below 1600 calories.
    Staying low Carb does control my blood sugar and my Carb/sugar cravings so I don't want to stop but I do need to keep losing. right now I can't get below 175

  2. ouizoid

    see, I think we are all different. I need to eat very low fat with slightly higher carbs to feel appetite control. When I eat high fat low carb, I get hungry, almost bingey. If I eat a more vegetable based diet, I am free of cravings and hunger. I just think we all have different genetic organizations. I am definitely in favour of lowish carb--but I had to find my way of having appetite suppression, and it is not the same as others.

  3. Ntombi

    Ketosis definitely suppresses my appetite. What's more, not eating carby food doesn't lead to blood sugar highs and lows, so I don't feel starving three hours after eating something like oatmeal. So the combination of eating very low carb and being in ketosis makes a difference for my hunger pattern.
    I eat very low carb (fewer than 20g total), moderate protein (~120g--enough to nourish my LBM), and enough fat to make up the rest of my calories, because fat is the macronutrient that won't affect blood sugar nor be stored as fat. So yes I eat high fat, but that doesn't mean that I'm eating a ton of calories. I don't count calories, but I generally average about 1800-2000 calories per day, with some outliers.
    If you need to keep your calories below 1600, do that.

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