Metabolic Acidosis Shock

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Understand shock with this medical lecture from Roger Seheult, MD of http://www.medcram.com. Includes illustrations of the different types of shock: cardiogenic shock, hypovolemic shock, and septic shock. This is video 1 of 2 on shock (the types of shock & shock treatment) and sepsis and is part of the "MedCram Remastered" series: A video we've re-edited/sped up to make learning even more efficient. Visit https://www.MedCram.com for this entire course and over 100 free lectures. This is the home for all new and updated MedCram medical videos (many videos, medical lectures, and quizzes are not on YouTube). Speaker: Roger Seheult, MD Co-Founder of MedCram.com (https://www.medcram.com) Clinical and Exam Preparation Instructor Board Certified in Internal Medicine, Pulmonary Disease, Critical Care, and Sleep Medicine. MedCram: Medical education topics explained clearly including: Respiratory lectures such as Asthma and COPD. Renal lectures on Acute Renal Failure and Adrenal Gland. Internal medicine videos on Oxygen Hemoglobin Dissociation Curve and Medical Acid Base. A growing library on critical care topics such as Shock and sepsis, Diabetic Ketoacidosis (DKA), and Mechanical Ventilation. Cardiology videos on Hypertension, ECG / EKG Interpretation, and heart failure. VQ Mismatch and Hyponatremia lectures have been popular among medical students and physicians. The Pulmonary Function Tests (PFTs) videos and Ventilator associated pneumonia bundles and lectures have been particularly popular with RTs. NPs and PAs have given great feedback on Pneumonia Treatment and Liver Function Tests among many others. Many nursing students have found the Asthma and shock lectures very helpful. Subscribe to the official MedCram.com YouTube Channel: https://www.youtube.com/subscription_... Recommended Audience - medical professionals and medical students: including physicians, nurse practitioners, physician assistants, nurses, respiratory therapists, EMT and paramedics, and many others. Review and test prep for USMLE, MCAT, PANCE, NCLEX, NAPLEX, NBDE, RN, RT, MD, DO, PA, NP school and board examinations. More from MedCram.com medical videos: MedCram Website: https://www.medcram.com Facebook: https://www.facebook.com/MedCram Google+: https://plus.google.com/u/1/+Medcram Twitter: https://twitter.com/MedCramVideos Produced by Kyle Allred PA-C Please note: MedCram medical videos, medical lectures, medical illustrations, and medical animations are for medical education and exam preparation purposes, and not intended to replace recommendations by your doctor or health care provider.

Metabolic Acidosis: Pathophysiology, Diagnosis And Management: Management Of Metabolic Acidosis

Recommendations for the treatment of acute metabolic acidosis Gunnerson, K. J., Saul, M., He, S. & Kellum, J. Lactate versus non-lactate metabolic acidosis: a retrospective outcome evaluation of critically ill patients. Crit. Care Med. 10, R22-R32 (2006). Eustace, J. A., Astor, B., Muntner, P M., Ikizler, T. A. & Coresh, J. Prevalence of acidosis and inflammation and their association with low serum albumin in chronic kidney disease. Kidney Int. 65, 1031-1040 (2004). Kraut, J. A. & Kurtz, I. Metabolic acidosis of CKD: diagnosis, clinical characteristics, and treatment. Am. J. Kidney Dis. 45, 978-993 (2005). Kalantar-Zadeh, K., Mehrotra, R., Fouque, D. & Kopple, J. D. Metabolic acidosis and malnutrition-inflammation complex syndrome in chronic renal failure. Semin. Dial. 17, 455-465 (2004). Kraut, J. A. & Kurtz, I. Controversies in the treatment of acute metabolic acidosis. NephSAP 5, 1-9 (2006). Cohen, R. M., Feldman, G. M. & Fernandez, P C. The balance of acid base and charge in health and disease. Kidney Int. 52, 287-293 (1997). Rodriguez-Soriano, J. & Vallo, A. Renal tubular acidosis. Pediatr. Nephrol. 4, 268-275 (1990). Wagner, C. A., Devuyst, O., Bourgeois, S. & Mohebbi, N. R Continue reading >>

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Popular Questions

  1. P70D1GY

    Ughhhhh, i feel like crap. On the keto diet i had super energy, was motivated and driven....
    Now, im tired, lazy and depressed.
    The total duration of my keto diet was 2 weeks.
    I went from zero carbs to insane nothing but carbs (and some fats).
    Its been 3 days now and i cant take this horrible feeling.
    To be honest id rather deal with the nasty and repetative foods eaten in ketosis.
    Somebody help!!!! Idk whats wrong with me.
    Should also note i think i started to devolope kidney failure on my last day in ketosis.
    (i had a red rash in pathes all over my body bilaterally, and they went away after i drank 5 glasses of water. and now my kidneys occationally hurt even when im not in ketosis.)

  2. Eileen

    To start with, keto does not cause kidney failure. There is not one single reported instance of that happening, and the whole idea of why it might is based on the idea that keto-ers eat nothing but protein. In fact, most BB-ers eat far more protein then keto-ers. And they don't get kidney failure. So forget that.
    The fact that you drank water and your symptoms went away tells the true story. You just weren't drinking enough. Don't blame keto.
    You eat an insane amount of carbs and now you feel crappy and depressed. Gee, I wonder what the answer to that might be?
    You don't have to go back to keto if you don't want, but I think you've just had a clear warning that you don't handle huge amounts of carbs well. So if you do a mixed diet, keep your carbs clean and unprocessed.

  3. P70D1GY

    So what im getting from that thread is that keto diets deplete seratonin....
    Is it possible im getting too much of it now and am expiriencing seratonin overdose?

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Anion gap usmle - anion gap metabolic acidosis normal anion gap metabolic acidosis

Metabolic Acidosis: Causes, Symptoms, And Treatment

The Terrible Effects of Acid Acid corrosion is a well-known fact. Acid rain can peel the paint off of a car. Acidifying ocean water bleaches and destroys coral reefs. Acid can burn a giant hole through metal. It can also burn holes, called cavities, into your teeth. I think I've made my point. Acid, regardless of where it's at, is going to hurt. And when your body is full of acid, then it's going to destroy your fragile, soft, internal organs even more quickly than it can destroy your bony teeth and chunks of thick metal. What Is Metabolic Acidosis? The condition that fills your body with proportionately too much acid is known as metabolic acidosis. Metabolic acidosis refers to a physiological state characterized by an increase in the amount of acid produced or ingested by the body, the decreased renal excretion of acid, or bicarbonate loss from the body. Metabolism is a word that refers to a set of biochemical processes within your body that produce energy and sustain life. If these processes go haywire, due to disease, then they can cause an excess production of hydrogen (H+) ions. These ions are acidic, and therefore the level of acidity in your body increases, leading to acidem Continue reading >>

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Popular Questions

  1. carwinski

    I've been on Keto for about 3 months. Not completely strict but I've done a pretty good job. Started at 205 and I am down to 180. I was never particularly heavy...I've always been athletic and an avid gym goer. So now that I am down to 180...I feel like I need some "fluff".

    I've been watching Jason Whittrock on YouTube the last 3 weeks and his intake of 4k cals per day. Then I watched him lose 2 lbs and 0.2% of body fat! Pretty amazing anyway you want to look at it. My involvement in Keto started as a need to lean out and is transforming in to a need to live longer and be healthy! I have no interest in stopping Keto but I would rather not be as lean as I am....if that makes any sense. Has anyone ever done a successful "bulk" using strict Keto?

  2. stacy

    Similar thought here. I have been keto 3.5 years, was never overweight much, went from 175 to 160 which is my high school weight. I am 16% body fat with some muscle (not a whole lot) as I work out regularly.
    I sometimes feel this is too lean but I figure it is where my body wants to be. I feel great and just plan to keep working out and maybe add some more muscle weight.

    Do you feel good and have energy? I don’t know anything really about bulking up so you might check the Keto Gains subreddit as I think they focus on that. reddit.com/r/ketogains

  3. akirby83

    Are you sure you need more fat, and do you really just need more muscle? IDK how successful you would be bulking on keto, since as Jason Wittrock has proven it doesn't matter if you eat a ton of calories on LCHF, you will not gain fat. So that means adding crappy carbs back into your diet.

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What is INSULIN SHOCK THERAPY? What does INSULIN SHOCK THERAPY mean? INSULIN SHOCK THERAPY meaning - INSULIN SHOCK THERAPY definition - INSULIN SHOCK THERAPY explanation. Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. Insulin shock therapy or insulin coma therapy (ICT) was a form of psychiatric treatment in which patients were repeatedly injected with large doses of insulin in order to produce daily comas over several weeks. It was introduced in 1927 by Austrian-American psychiatrist Manfred Sakel and used extensively in the 1940s and 1950s, mainly for schizophrenia, before falling out of favour and being replaced by neuroleptic drugs in the 1960s. It was one of a number of physical treatments introduced into psychiatry in the first four decades of the twentieth century. These included the convulsive therapies (cardiazol/metrazol therapy and electroconvulsive therapy), deep sleep therapy and psychosurgery. Insulin coma therapy and the convulsive therapies are collectively known as the shock therapies. Insulin coma therapy was a labour-intensive treatment that required trained staff and a special unit. Patients, who were almost invariably diagnosed with schizophrenia, were selected on the basis of having a good prognosis and the physical strength to withstand an arduous treatment. There were no standard guidelines for treatment; different hospitals and psychiatrists developed their own protocols. Typically, injections were administered six days a week for about two months. The daily insulin dose was gradually increased to 100150 units until comas were produced, at which point the dose would be levelled out. Occasionally doses of up to 450 units were used. After about 50 or 60 comas, or earlier if the psychiatrist thought that maximum benefit had been achieved, the dose of insulin was rapidly reduced before treatment was stopped. Courses of up to 2 years have been documented. After the insulin injection patients would experience various symptoms of decreased blood glucose: flushing, pallor, perspiration, salivation, drowsiness or restlessness. Sopor and comaif the dose was high enoughwould follow. Each coma would last for up to an hour and be terminated by intravenous glucose. Seizures sometimes occurred before or during the coma. Many would be tossing, rolling, moaning, twitching, spasming or thrashing around. Some psychiatrists regarded seizures as therapeutic and patients were sometimes also given electroconvulsive therapy or cardiazol/metrazol convulsive therapy during the coma, or on the day of the week when they didnt have insulin treatment. When they were not in a coma, insulin coma patients were kept together in a group and given special treatment and attention; one handbook for psychiatric nurses, written by British psychiatrist Eric Cunningham Dax, instructs nurses to take their insulin patients out walking and occupy them with games and competitions, flower-picking and map-reading, etc. Patients required continuous supervision as there was a danger of hypoglycemic aftershocks after the coma. In "modified insulin therapy", used in the treatment of neurosis, patients were given lower (sub-coma) doses of insulin. A few psychiatrists (including Sakel) claimed success rates for insulin coma therapy of over 80 percent in the treatment of schizophrenia; a few others argued that it merely sped up remission in those patients who would undergo remission anyway. The consensus at the time was somewhere in between - claiming a success rate of about 50 percent in patients who had been ill for less than a year (about double the spontaneous remission rate) with no influence on relapse. Sakel suggested the therapy worked by "causing an intensification of the tonus of the parasympathetic end of the autonomic nervous system, by blockading the nerve cell, and by strengthening the anabolic force which induces the restoration of the normal function of the nerve cell and the recovery of the patient." The shock therapies in general had developed on the erroneous premise that epilepsy and schizophrenia rarely occurred in the same patient. Another theory was that patients were somehow "jolted" out of their mental illness.

Mortality And The Nature Of Metabolic Acidosis In Children With Shock

, Volume 29, Issue2 , pp 286291 | Cite as Mortality and the nature of metabolic acidosis in children with shock Mortality in children with shock is more closely related to the nature, rather than the magnitude (base deficit/excess), of a metabolic acidosis. To examine the relationship between base excess (BE), hyperlactataemia, hyperchloraemia, 'unmeasured' strong anions, and mortality. Prospective observational study set in a multi-disciplinary Paediatric Intensive Care Unit (PICU). Forty-six children, median age 6months (1.514.4), median weight 5kg (3.28.8), admitted to PICU with shock. Predicted mortality was calculated from the paediatric index of mortality (PIM) score. The pH, base excess, serum lactate, corrected chloride, and 'unmeasured' strong anions (Strong Ion Gap) were measured or calculated at admission and 24h. Observed mortality (n=16) was 35%, with a standardised mortality ratio (SMR) of 1.03 (95% CI 0.711.35). There was no significant difference in admission pH or BE between survivors and nonsurvivors. There was no association between elevation of 'unmeasured' anions and mortality, although there was a trend towards hyperchloraemia in survivors (P=0.08). Admission Continue reading >>

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Popular Questions

  1. Patrizia_B

    Good morning,
    I am on day 5 of my induction phase today and think I am doing well with the food.
    I used a ketostix last evening and it showed a colour between the two first pink ones. This morning it was a bit lighter, but still pink-ish.
    Now: does that mean I am in Ketosis or not? Does the colour need to be dark purple to suggest i lose more than when it is pink?
    I cruised around the forum, but could not find any answer.
    Thank you for helping,

  2. Kisal

    You're either in ketosis, or you're not. (It's sort of like being pregnant. :lol: ) The color of the stix makes no difference. Here's some information that explains it further:

  3. lisabinil

    My Ketostix have registered negative all week but I have lost 2.2 lbs. I usually only show light pink and the only time I ever show purple is after I have a few drinks.

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