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How Does Ketoacidosis Cause Hyperventilation

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Does Chronic Hyperventilation Cause Brain Damage?

“Does chronic hyperventilation cause brain damage?” “…since it causes cerebral hypoxia due to low CO2 level, can we deduce that it cause brain damage?” Voluntarily hyperventilating is unlikely to cause any problems. Your brain and body is generally too intelligent to allow you to damage yourself in such a manner. Low CO2 levels from Hyperventilation can cause cerebral vasoconstriction and ischemia. This usually is only a seen in settings of artificial respiration. Well it does not directly cause brain damage. However, you are correct that it can cause brain damage indirectly. When you hyper ventilate, your brain becomes depleted of oxygen. This is not good. Extened periods of lack of oxygen can cause cells (and neurons) to become damaged, or even die. This should not really be a problem unless it is a regular occurance. Hyperventilating for a few seconds every day wont do much, but if you are hyperventilating for very long periods of time multiple times daily, this could be a problem. I strongly suggest you speak to a doctor about this if you are concerned. If I can help, I suggest practising breathing excersises regularly. This will allow you over time to become more in Continue reading >>

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  1. White Wolf

    Hypocalcemia

    Hyperventilation

    Medical Conditions and Diseases


    Medicine and Healthcare



    How does hyperventilation cause hypocalcemia?




    1 Answer







    Hyperventilation will wash out the carbon dioxide from the blood, which results in respiratory alkalosis.
    If the kidney can regulate the pH by altering H+ and bicarbonate transfer across the tubules, the pH will return to normal.

    If not, then the increased pH will increase binding of free calcium to protein, hence reducing the concentration of calcium ion in blood manifested by tetanic spasms, etc.

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Dka And Cerebral Oedema Do We Really Know The Cause?

Cerebral oedema is the most feared complication in children presenting with Diabetic Ketoacidosis (DKA). It occurs in about 1% of cases but has a mortality rate of up to 90% (Waldorf J et al Diabetes Care 2006; 29:1150-9). Patients will have a decreased conscious state and may also have cranial nerve palsies, headache and/or bradycardia and hypertension. Its incidence has remained the same since it was described in 1936 and although we have clues as to what may contribute to it, and we know that some patients have subclinical cerebral oedema at presentation(Krane et al NEJM 1985;312:1147-51), we still cant predict who will get it, nor greatly affect its high rate of mortality. There are theories of causative factors, most of which are vasogenic or osmotically based, but there is really no great evidence out there. The studies are small or retrospective, or both. One theory relates to osmolytes accumulating in brain cells. These are the compounds that maintain normal cell volumes. As extracellular osmolality decreases rapidly with treatment, water flows rapidly onto these cells causing the brain to swell. Another theory relates to Na+ / H+ exchanger, such that a correction of acido Continue reading >>

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  1. Alan Young

    If you hyperventilate you ‘blow off’ the carbon dioxide in your blood very quickly. Carbon dioxide has an effect on cerebral blood vessels (the blood vessels in the brain).

  2. Jonas Grandt

    My best guess (which is better than nothing), is that rapid, shallow breaths use oxygen way faster than they collect, and they cannot effectively remove carbon dioxide from the bloodstream, leading to fainting through oxygen deprivation and carbon dioxide buildup. Once you've fainted, your breathing should return to normal, fixing both problems before much else happens.

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Chapter 16: Acid-base Imbalances

Sort What is the function of a buffer? a) To excrete weak acids b) To secrete hydrogen ions c) To convert strong acids to weak acids d) To convert ammonia to ammonium ions c) To convert strong acids to weak acids -Buffers convert strong acids to weak acids. Excretion of weak acids, secretion of hydrogen ions into the renal tubule, and conversion of ammonia to ammonium ions takes place in the kidneys. A nurse is caring for a patient three days after abdominal surgery who continues to have poorly controlled abdominal pain with green bilious nasogastric output. The patient's respiratory rate is 32 and heart rate is 128. Which acid-base imbalance does the nurse suspect is occurring? a) Mixed acidosis b) Mixed alkalosis c) Metabolic alkalosis d) Respiratory acidosis b) Mixed alkalosis -Mixed alkalosis can occur in a patient who is losing CO2 via hyperventilation (possibly related to pain) while also losing acid by another method, such as prolonged suctioning with a nasogastric tube. Respiratory acidosis occurs when the primary loss of acid is via a respiratory "blow off" of CO2. Metabolic alkalosis occurs with a systemic loss of acid via a metabolic process such as vomiting or suctionin Continue reading >>

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  1. Alan Young

    If you hyperventilate you ‘blow off’ the carbon dioxide in your blood very quickly. Carbon dioxide has an effect on cerebral blood vessels (the blood vessels in the brain).

  2. Jonas Grandt

    My best guess (which is better than nothing), is that rapid, shallow breaths use oxygen way faster than they collect, and they cannot effectively remove carbon dioxide from the bloodstream, leading to fainting through oxygen deprivation and carbon dioxide buildup. Once you've fainted, your breathing should return to normal, fixing both problems before much else happens.

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