Euglycemic Ketoacidosis Wiki

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What is DIABETIC KETOACIDOSIS? What does DIABETIC KETOACIDOSIS mean? DIABETIC KETOACIDOSIS meaning - DIABETIC KETOACIDOSIS definition - DIABETIC KETOACIDOSIS explanation. Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. SUBSCRIBE to our Google Earth flights channel - https://www.youtube.com/channel/UC6Uu... Diabetic ketoacidosis (DKA) is a potentially life-threatening complication of diabetes mellitus. Signs and symptoms may include vomiting, abdominal pain, deep gasping breathing, increased urination, weakness, confusion, and occasionally loss of consciousness. A person's breath may develop a specific smell. Onset of symptoms is usually rapid. In some cases people may not realize they previously had diabetes. DKA happens most often in those with type 1 diabetes, but can also occur in those with other types of diabetes under certain circumstances. Triggers may include infection, not taking insulin correctly, stroke, and certain medications such as steroids. DKA results from a shortage of insulin; in response the body switches to burning fatty acids which produces acidic ketone bodies. DKA is typically diagnosed when testing finds high blood sugar, low blood pH, and ketoacids in either the blood or urine. The primary treatment of DKA is with intravenous fluids and insulin. Depending on the severity, insulin may be given intravenously or by injection under the skin. Usually potassium is also needed to prevent the development of low blood potassium. Throughout treatment blood sugar and potassium levels should be regularly checked. Antibiotics may be required in those with an underlying infection. In those with severely low blood pH, sodium bicarbonate may be given; however, its use is of unclear benefit and typically not recommended. Rates of DKA vary around the world. About 4% of people with type 1 diabetes in United Kingdom develop DKA a year, while in Malaysia the condition affects about 25% a year. DKA was first described in 1886 and, until the introduction of insulin therapy in the 1920s, it was almost universally fatal. The risk of death with adequate and timely treatment is currently around 1–4%. Up to 1% of children with DKA develop a complication known as cerebral edema. The symptoms of an episode of diabetic ketoacidosis usually evolve over a period of about 24 hours. Predominant symptoms are nausea and vomiting, pronounced thirst, excessive urine production and abdominal pain that may be severe. Those who measure their glucose levels themselves may notice hyperglycemia (high blood sugar levels). In severe DKA, breathing becomes labored and of a deep, gasping character (a state referred to as "Kussmaul respiration"). The abdomen may be tender to the point that an acute abdomen may be suspected, such as acute pancreatitis, appendicitis or gastrointestinal perforation. Coffee ground vomiting (vomiting of altered blood) occurs in a minority of people; this tends to originate from erosion of the esophagus. In severe DKA, there may be confusion, lethargy, stupor or even coma (a marked decrease in the level of consciousness). On physical examination there is usually clinical evidence of dehydration, such as a dry mouth and decreased skin turgor. If the dehydration is profound enough to cause a decrease in the circulating blood volume, tachycardia (a fast heart rate) and low blood pressure may be observed. Often, a "ketotic" odor is present, which is often described as "fruity", often compared to the smell of pear drops whose scent is a ketone. If Kussmaul respiration is present, this is reflected in an increased respiratory rate.....

Diabetic Ketoacidosis

Print Overview Diabetic ketoacidosis is a serious complication of diabetes that occurs when your body produces high levels of blood acids called ketones. The condition develops when your body can't produce enough insulin. Insulin normally plays a key role in helping sugar (glucose) — a major source of energy for your muscles and other tissues — enter your cells. Without enough insulin, your body begins to break down fat as fuel. This process produces a buildup of acids in the bloodstream called ketones, eventually leading to diabetic ketoacidosis if untreated. If you have diabetes or you're at risk of diabetes, learn the warning signs of diabetic ketoacidosis — and know when to seek emergency care. Symptoms Diabetic ketoacidosis signs and symptoms often develop quickly, sometimes within 24 hours. For some, these signs and symptoms may be the first indication of having diabetes. You may notice: Excessive thirst Frequent urination Nausea and vomiting Abdominal pain Weakness or fatigue Shortness of breath Fruity-scented breath Confusion More-specific signs of diabetic ketoacidosis — which can be detected through home blood and urine testing kits — include: High blood sugar l Continue reading >>

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  1. legking

    Ketostix = negative! Wft!?

    I was browsing the forums and found several references to Ketostix, so I picked some up. However, ALL of my tests (tried yesterday about 5 times, and once this morning as soon as I woke up) came out negative!
    I'm on CKD right now, a day before my carb-up, and I'm flummoxed. I'm very diligent about what I take in; I mean, the vast majority of my diet consists of tuna & mayo, pepperettes, cheese, salmon and nuts.
    The only explanation I can think of is either I'm not using them correctly, or I drink so much water and pee so often that any ketones would be so diluted as to come up negative. Can anyone with experience with this gimme some feedback? Thanks in advance!

  2. Man2kx

    How long have you been dieting the CKD phase for?
    What do your workouts look like? Are you getting enough intensity?
    How much liquid do you drink a day?
    As far as using them correctly, you're supposed to urinate and wait for the color for 15 seconds. It usually shows up instantly though but for very slight changes in hue (i.e. to maybe see some pink) then you may have to wait your 15 secs.

  3. Jason762

    2.17: How can I tell if I'm in ketosis?
    A: The primary method: Ketostix (urine analysis strips) . Ketostix can be obtained at a pharmacy. If your pharmacy has a diabetics supply area, it will be there, otherwise simply ask the pharmicist. Ketostix measure the prescence of ketones in the urine. If the strips get dark, you're in ketosis. Ketosis is also evidenced by particuarly bad or "fruity" breath and foul smelling urine. A metallic taste in the mouth is also commonly noted. Many people notice a different mental state when they're in ketosis. For some, they get "foggy" about things while others are exactly the opposite: they feel more alert. This appears to vary considerably from individual to individual. Ketostix are not a completely reliable indication of ketosis, but it's the best we have. [HC]
    2.18: What if my ketostix aren't getting dark?
    A: There are varying degrees of ketosis. If the strips aren't changing color at all, you may still be in, just not excreting sufficient ketones to react the sticks. But as long as you are showing at least "trace", you are in ketosis. The correlation of the degree of darkness of the strips to fat loss is unclear, the strips represent the amount of ketones present in the urine (i.e. excreted). If you burned up all your ketones as energy , the strips won't show anything. Darker strips don't necessary indicate greater fat loss. Some individuals find that lesser degrees of ketosis are better for fat loss although this is not universal. [HC]
    Hope this helps. Got it from http://low-carb.org/faq/#Q1

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What is KETOACIDOSIS? What does KETOACIDOSIS mean? KETOACIDOSIS meaning - KETOACIDOSIS definition - KETOACIDOSIS explanation. Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. SUBSCRIBE to our Google Earth flights channel - https://www.youtube.com/channel/UC6Uu... Ketoacidosis is a metabolic state associated with high concentrations of ketone bodies, formed by the breakdown of fatty acids and the deamination of amino acids. The two common ketones produced in humans are acetoacetic acid and ß-hydroxybutyrate. Ketoacidosis is a pathological metabolic state marked by extreme and uncontrolled ketosis. In ketoacidosis, the body fails to adequately regulate ketone production causing such a severe accumulation of keto acids that the pH of the blood is substantially decreased. In extreme cases ketoacidosis can be fatal. Ketoacidosis is most common in untreated type 1 diabetes mellitus, when the liver breaks down fat and proteins in response to a perceived need for respiratory substrate. Prolonged alcoholism may lead to alcoholic ketoacidosis. Ketoacidosis can be smelled on a person's breath. This is due to acetone, a direct by-product of the spontaneous decomposition of acetoacetic acid. It is often described as smelling like fruit or nail polish remover. Ketosis may also smell, but the odor is usually more subtle due to lower concentrations of acetone. Treatment consists most simply of correcting blood sugar and insulin levels, which will halt ketone production. If the severity of the case warrants more aggressive measures, intravenous sodium bicarbonate infusion can be given to raise blood pH back to an acceptable range. However, serious caution must be exercised with IV sodium bicarbonate to avoid the risk of equally life-threatening hypernatremia. Three common causes of ketoacidosis are alcohol, starvation, and diabetes, resulting in alcoholic ketoacidosis, starvation ketoacidosis, and diabetic ketoacidosis respectively. In diabetic ketoacidosis, a high concentration of ketone bodies is usually accompanied by insulin deficiency, hyperglycemia, and dehydration. Particularly in type 1 diabetics the lack of insulin in the bloodstream prevents glucose absorption, thereby inhibiting the production of oxaloacetate (a crucial molecule for processing Acetyl-CoA, the product of beta-oxidation of fatty acids, in the Krebs cycle) through reduced levels of pyruvate (a byproduct of glycolysis), and can cause unchecked ketone body production (through fatty acid metabolism) potentially leading to dangerous glucose and ketone levels in the blood. Hyperglycemia results in glucose overloading the kidneys and spilling into the urine (transport maximum for glucose is exceeded). Dehydration results following the osmotic movement of water into urine (Osmotic diuresis), exacerbating the acidosis. In alcoholic ketoacidosis, alcohol causes dehydration and blocks the first step of gluconeogenesis by depleting oxaloacetate. The body is unable to synthesize enough glucose to meet its needs, thus creating an energy crisis resulting in fatty acid metabolism, and ketone body formation.

Diabetic Ketoacidosis

4 Evaluation 5 Management Defining features include hyperglycemia (glucose > 250mg/dl), acidosis (pH < 7.3), and ketonemia/ketonuria Leads to osmotic diuresis and depletion of electrolytes including sodium, magnesium, calcium and phosphorous. Further dehydration impairs glomerular filtration rate (GFR) and contributes to acute renal failure Due to lipolysis / accumulation of of ketoacids (represented by increased anion gap) Compensatory respiratory alkalosis (i.e. tachypnea and hyperpnea - Kussmaul breathing) Breakdown of adipose creates first acetoacetate leading to conversion to beta-hydroxybutyrate Causes activation of RAAS in addition to the osmotic diuresis Cation loss (in exchange for chloride) worsens metabolic acidosis May be the initial presenting of an unrecognized T1DM patient Presenting signs/symptoms include altered mental status, tachypnea, abdominal pain, hypotension, decreased urine output. Perform a thorough neurologic exam (cerebral edema increases mortality significantly, especially in children) Assess for possible inciting cause (especially for ongoing infection; see Differential Diagnosis section) Ill appearance. Acetone breath. Drowsiness with decreased reflex Continue reading >>

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  1. MechantMiaou

    Hello, I'm planning on eating mostly green beans (350g) and sauerkraut (350g) per day , more or less, everyday. I think I've read that too much this kind of food can lead to kidney stones, is there a safe limit or something ?

  2. ArentEnoughRocks

    I watched a video by an MD about the risk of stones in keto. Basically, his advice was: cruciferous veg and spinach add oxalates. Use the juice of one lemon per day (citrates) and have calcium from food (cheese, dairy, etc) as the calcium will bind in the digestive system w the high levels of oxalates (instead of in blood and out into the kidney/urine) . There's other videos on YT too on the subject. I just searched "keto kidney stones". Make sure you've got enough magnesium (if you're not getting it from food).
    Here's an article on it
    All I can tell you is that my dad got stones on keto. He was having a lot of protein shakes, he said, which was all the information I got out of him at the time bc I wasn't really interested in keto then.

  3. Forgetfultiger

    Kidney stones can be caused by high urine pH or low urine pH or a number of other things like bacteria. If you're worried about kidney stones go see your doctor.

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People always freak out when I tell them I am doing keto. I even have one friend that refers to my diet as "The Ketoacidosis Diet". I can't get him to understand the difference, but maybe this will help you! Find Me On Twitter: www.twitter.com/HeavyKevi Instagram: www.instagram.com/TheHeavyTruthTV Follow My Macros on MyFitnessPal @The HeavyTruth Facebook Group: Facebook.com/Groups/TheHeavyTruthTV Subscribe to my Essential Oils Channel: https://goo.gl/El053Q Send Questions or Testimonials By Mail: Kevin Gillem P.O.Box 291517 Phelan, CA 92329 My Favorite Low Carb Sweetener - http://amzn.to/2smCmDM I recommend Smackfat Ketone Strips - http://amzn.to/2laB9MG I use the Match DNA Milk Frother - http://amzn.to/2klHt4o I use NOW MCT Oil - http://amzn.to/2kOs48S I'm Kevin and I have used a Ketogenic diet, Intermittent Fasting and Extended Fasting to successfully lose 160 pounds and I am still shrinking. I hope to one day be half the man I was at 400 pounds. During this journey I have learned a lot about weight loss, Metabolic syndrome, Insulin Resistance, LCHF dieting and overall health in general. I am greatly interested in continuing to learn about health related topics and sharing what I learn with others so that all of our lives can be improved. Please like and subscribe to my channel so that I can share the things I learn that I think are important for all of our well being. I would also appreciate feedback from you all so I can share it with other subscribers and we can all grow as a community. I am not a doctor. I am not licensed. I do not hold any qualifications for giving medical advice. This is an account of my own experiences and does not apply to anyone else. All information, content, and material of this channel is for informational purposes only and are not intended to serve as a substitute for the consultation, diagnosis, and/or medical treatment of a qualified physician or healthcare provider. If you have questions about your own personal situation it is recommended that you discuss them with your own licensed healthcare professional. If you think you are having an emergency, dial 911 immediately.


GENERAL ketoacidosis is a high anion gap metabolic acidosis due to an excessive blood concentration of ketone bodies (keto-anions). ketone bodies (acetoacetate, beta-hydroxybutyrate, acetone) are released into the blood from the liver when hepatic lipid metabolism has changed to a state of increased ketogenesis. a relative or absolute insulin deficiency is present in all cases. CAUSES The three major types of ketosis are: (i) Starvation ketosis (ii) Alcoholic ketoacidosis (iii) Diabetic ketoacidosis STARVATION KETOSIS when hepatic glycogen stores are exhausted (eg after 12-24 hours of total fasting), the liver produces ketones to provide an energy substrate for peripheral tissues. ketoacidosis can appear after an overnight fast but it typically requires 3 to 14 days of starvation to reach maximal severity. typical keto-anion levels are only 1 to 2 mmol/l and this will usually not alter the anion gap. the acidosis even with quite prolonged fasting is only ever of mild to moderate severity with keto-anion levels up to a maximum of 3 to 5 mmol/l and plasma pH down to 7.3. ketone bodies also stimulate some insulin release from the islets. patients are usually not diabetic. ALCOHOLIC KE Continue reading >>

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  1. Lilhead

    Hey everyone, I would like some advice... I used to be very fat/keto adapted in 2012 to 2015 I was strict keto I started with a BMI of 45 to a BMI of 24 (I know BMI is crap however it gives an idea of what weight I was and went to)....so from 2015 till now I have slowly been putting weight on as I was on and off keto to where I'm now a BMI of >30! My question is I have tried to go back on to it without success and I have been told that once you miss the bandwagon then you can't go back into ketosis.... is this true? P.S I'm desperate now lol ...TIA

  2. LouiseReynolds

    Firstly, good for you for getting back on the keto-train! I haven't heard about 'missing the boat' that once you're out of ketosis that you can never return (a bit like you've been locked out for good??) What it will mean of course is switching back on your fuel usage system (that is burning fat again, rather than glucose). Of course, this will take the requisite three or so days for the fuel switch (carb withdrawal) and then the process to being fully fat adapted. So, to answer your question, I wouldn't think that your body would shut off the fuel usage system because you've used it once and changed back again.

  3. Lilhead

    Thank you Louise I hope so... I just wish I never stopped

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