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Dka Vs Hhs Ppt

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Hey, everyone! I wanted to share some bumps in the road that have been coming up with post-op and how Im doing some things for prevention :) Thanks for watching! xoxo, Erin Join my private gastric sleeve group community coaching program to be the best and have support on your journey!: http://bit.ly/MyLevel10LifePrivateGroup Mentioned: Terrazyme from doTERRA http://bit.ly/2HfD6zn Zendocrine Essential Oil Blend 10-15 drops in a roller bottle http://bit.ly/2HfD6zn Nioxin Shampoo & Conditioner http://amzn.to/2Cjg6Md Weekly Vitamin Organizer: http://amzn.to/2FNlWsc Work with Erin: Biz Coaching/Get out of Debt with me: [email protected] Get your FREE Top 10 Mistakes MLMs Make on Social Media: http://bit.ly/RBMGuideTopTEN Follow Me on IG: https://www.instagram.com/rusticbarnm... How Im Saving Money MY AMAZON FAVES: http://bit.ly/ShopMyLevel10Life $10 OFF WALMART GROCERIES: http://bit.ly/SHOPWALMARTFREE $10 BONUS with IBOTTA: https://ibotta.com/r/osjtrcs (referral code: osjtrcs) GET ONE MONTH FREE ON AUDIBLE: https://amzn.to/2JYrVAn GET $40 CREDIT WITH AIRBNB: https://abnb.me/e/K6wPogWbrP My Favorite Essential Oils To Order: http://bit.ly/2MNv9UX USE MY ID: 40395

Preventing Complications Of Dm Ppt.

-give 20-30g CHO or Glucagon 1mg (retest in 15 min) -Give Iglucagon SQ (repeat in 10 min if pt is still unresponsive) --at hospital give 25-50% dextrose IV bolus --slower adrenergic response to falling glucose (shaky, nervous, tachycardia) Acute complications of Poor Glycemic control -Hyperglycemic crisis associated with fluid and electrolyte loss -manifestation of insulin deficiency (severe) Characteristics: Hyperglycemia, ketoacids, hemoconcentration, acidosis, coma -Kussmal respiration (rapid and deep) --trying to expel CO2 to increase the pH 3. Proliferative (overgrowth of abnormal blood vessels on the retina --very thin and prone to aneurysm) regular ophthalmologic exams (with dilated pupils) report any floaters, flashing lights, or changes in vision --decreased blood flow to Bowman's capsule avoid nephrotoxic drugs and dye (hydrate after tests with contrast medium) check serum creatinine, albumin, and creatinine clearance (already have decreased blood supply and wound healing) -Decreased/altered Sensation (bilateral, symmetrical, lower extremities) Preventing Injury with decreased sensation -wear leather, canvas shoes (dry between wearing) -do not cross legs, wear restrictiv Continue reading >>

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Popular Questions

  1. rosehale

    Ate a teaspoon of peanut butter sat night yes I know I shouldn't of, what I'm wondering is if my body was going through the x amount of days to get to ketosis I presume my slip up will effect that and will my body be restarting the process to go into ketosis? And how long will it take approx?
    Many thanks in advance

  2. lynnwilliams

    3 days i think

  3. lynnwilliams

    It probably wont have an affect on your ketosis, you can buy ketostix from boots chemist and u can check yourself if you are in ketosis or not )

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This video will review our custom Infusion Advisors. We created these advisors in order to help improve insulin management for hyperglycemic states.

Dka/hhs. - Ppt Video Online Download

To view this video please enable JavaScript, and consider upgrading to a web browser that supports HTML5 video Published by Audrey Price Modified over 2 years ago 115,000 hospitalizations in U.S 10-30% of admissions with primary diagnosis of Diabetes Mortality fallen 22% over the past 20 years Lower incidence compared to DKA 1% of DM related admissions Much high mortality rate ~40% compared to <5% for DKA No significant improvement in recent past 5 Diagnosis Table with diagnostic parameters 11 Ketosis Prone T2DM Mostly minorities Presents with DKA African Americans and Hispanics Presents with DKA Require insulin treatment initially Can usually be transitioned quickly to Oral hypoglycemics as outpatient Due to Glucose toxicity or lipotoxicity to the Beta-Cells 12 Presentation Dehydration Weight loss Weakness Abdominal Pain polyuria, polydipsia Weight loss Weakness Abdominal Pain Correlates with severity of acidosis May be confused with Acute Abdomen Related to osmolality more than hyperglycemia or acidosis More common in HHS Signs & Symptoms related to underlying cause Infection, CV disease etc Diabetic patients may have silent cardiac ischemia precipitating DKA even without chest Continue reading >>

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Popular Questions

  1. MandyJ

    Hey everyone!
    I’ve been doing keto for roughly 4 months now and have been loving it. I’ve incorporated a 16/8 IF most days with a few 3-5 day fasts mixed in as well. I’ve been listening to a lot of podcasts that talk about fasting vs feasting, and making sure you eat enough on the feasting days. I’ve also heard these podcasts talk about really feasting and taking in enough calories in order to prevent metabolic damage. As someone who was an eternal yo-yo dieter before keto, I assume I would have some damage already. Since going keto, I’ve only lost ten pounds, though I definitely look slimmer. I’m 5’10”, plateaued at 174, and have always been a little more muscular as well. My question here is how do you guys take in enough calories, and is undereating something I should be concerned about? I’ve tried keto coffee in the morning with 1-2 tablespoons each of butter, hwc, and mct oil, and dinner will be a bun-less burger with cheese, broccoli, and sometimes bacon. If not that, then I’ll scramble some eggs with butter or bacon and melt cheese on top. My calories are always around 1100 or less, though my bmr according to online calculators is closer to 1800. The common them is to eat fat to satiety, though I feel like I’m over eating and overly stuffed at such a calorie deficit already; that is, I really only feel hungry enough for one smaller meal a day. From everything that I’ve read and listened to I’m assuming this undereating is the reason I haven’t been able to lose more than the ten pounds. If so, how do I go about fixing my metabolism in order to be able to eat more without feeling sick?

  2. MiKetoAF

    BPC or what you call “Keto coffee” can easily be 800-900 calories itself. On my feasting days I just make sure to eat lots of fat and not over do the protein. I also stick to 2 meals and avoid snacking at all costs.

  3. LizinLowell

    I hear you, it’s very confusing the advice not to undereat. BUT, as I asked this same question a dozen different ways the advice that seemed more important was not to eat if you aren’t hungry. If you are slowing your metabolism you will have more symptoms than just slowed weight loss, such as feeling cold, depressed, increased hair loss. You might have a day where your appetite knows no bounds! For me it’s always changing. The last pounds are notoriously hard to lose, but it sounds like you are doing everything right. KCKO!

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Hyperglycemic crises: Hyperglycemic hyperosmolar nonketotic coma (HHNK) versus DKA. See DKA video here: https://youtu.be/r2tXTjb7EqU This video and similar images/videos are available for instant download licensing here https://www.alilamedicalmedia.com/-/g... Voice by: Penelope Hammet Alila Medical Media. All rights reserved. All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition. Support us on Patreon and get FREE downloads and other great rewards: patreon.com/AlilaMedicalMedia Hyperosmolar hyperglycemic state, or HHS, is another ACUTE and life-threatening complication of diabetes mellitus. It develops slower than DKA, typically in the course of several days, but has a much higher mortality rate. Like DKA, HHS is triggered when diabetic patients suffer from ADDITIONAL physiologic stress such as infections, other illness, INadequate diabetic treatment or certain drugs. Similar to DKA, the RISE in COUNTER-regulatory hormones is the major culprit. These hormones

Management Of The Hyperosmolar Hyperglycaemic State (hhs) In Adults With Diabetes (aug 2012)

Management of the hyperosmolar hyperglycaemic state (HHS) in adults with diabetes (Aug 2012) Management of the hyperosmolar hyperglycaemic state (HHS) in adults with diabetes (Aug 2012) Unlike the other common diabetes emergency, diabetic ketoacidosis (DKA), guidelines on the management of the hyperglycaemic hyperosmolar state (HHS) in adults are uncommon and often there is little to differentiate them from the management of DKA. However, HHS is different and treatment requires a different approach. The person with HHS is often elderly, frequently with multiple co-morbidities but always very sick. Even when specific hospital guidelines are available, adherence to and use of these is variable amongst the admitting teams. In many hospitals these patients are managed by non-specialist teams, and it is not uncommon for the most junior member, who is least likely to be aware of the hospital guidance, to be given responsibility for the initial management of this complex and challenging condition. Diabetes specialist teams are rarely involved at an early stage and sometimes never at all. To address these issues the Joint British Diabetes Societies (JBDS) for inpatient care, supported by Continue reading >>

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Popular Questions

  1. Santosh Anand

    Insulin plays a key role in helping sugar (glucose) enter your cells, thus providing them energy. When your cells don't get the glucose they need for energy, your body begins to burn fat for energy, which produces ketones. Ketones are acidic and so when they build up in the blood, they make the blood more acidic, leading to the condition called diabetic ketoacidosis (DKA).
    Now, in type-1 diabetes, there is no insulin production whereas in type-2, there is impairment of insulin production. Thus why Type-2 diabetic people hardly get DKA.
    Note: Diabetic ketoacidosis is a serious condition that might lead to diabetic coma or even death.

  2. Lucas Verhelst

    In order for the cells in your body to access the glucose in your bloodstream so they can use it as energy they need insulin. Insulin acts like a key, opennin the cell door to allow the entry of glucose. Type 1 diabetics produce no insulin and need to inject it, thus the amount of insulin they have is strictly limited. Once they run out of insulin the glucose remains in the blood stream. If this occurs over a long period of time their blood glucose levels will rise due to the release of glucose from the liver. High blood sugar levels causes ketoacidosis which leads to coma and death.

  3. Keith Phillips

    Although type 2 diabetics suffer from insulin resistance, the condition rarely has an absolute negative effect on the bodies ability to convert glucose to usable energy. Type 1 diabetics have little or no ability to produce insulin. With the exception of neural cells, the rest of the body which without insulin is experiencing starvation, will consume its own tissues. (this is how people have endured periods of famine). This process however produces by products that eventually overwhelm the body's ability to process toxins.

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