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Dka Protocol Uptodate

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What is DIABETIC KETOACIDOSIS? What does DIABETIC KETOACIDOSIS mean? DIABETIC KETOACIDOSIS meaning - DIABETIC KETOACIDOSIS definition - DIABETIC KETOACIDOSIS explanation. Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. SUBSCRIBE to our Google Earth flights channel - https://www.youtube.com/channel/UC6Uu... Diabetic ketoacidosis (DKA) is a potentially life-threatening complication of diabetes mellitus. Signs and symptoms may include vomiting, abdominal pain, deep gasping breathing, increased urination, weakness, confusion, and occasionally loss of consciousness. A person's breath may develop a specific smell. Onset of symptoms is usually rapid. In some cases people may not realize they previously had diabetes. DKA happens most often in those with type 1 diabetes, but can also occur in those with other types of diabetes under certain circumstances. Triggers may include infection, not taking insulin correctly, stroke, and certain medications such as steroids. DKA results from a shortage of insulin; in response the body switches to burning fatty acids which produces acidic ketone bodies. DKA is typically diagnosed when testing finds high blood sugar, low blood pH, and ketoacids in either the blood or urine. The primary treatment of DKA is with intravenous fluids and insulin. Depending on the severity, insulin may be given intravenously or by injection under the skin. Usually potassium is also needed to prevent the development of low blood potassium. Throughout treatment blood sugar and potassium levels should be regularly checked. Antibiotics may be required in those with an underlying infection. In those with severely low blood pH, sodium bicarbonate may be given; however, its use is of unclear benefit and typically not recommended. Rates of DKA vary around the world. About 4% of people with type 1 diabetes in United Kingdom develop DKA a year, while in Malaysia the condition affects about 25% a year. DKA was first described in 1886 and, until the introduction of insulin therapy in the 1920s, it was almost universally fatal. The risk of death with adequate and timely treatment is currently around 1–4%. Up to 1% of children with DKA develop a complication known as cerebral edema. The symptoms of an episode of diabetic ketoacidosis usually evolve over a period of about 24 hours. Predominant symptoms are nausea and vomiting, pronounced thirst, excessive urine production and abdominal pain that may be severe. Those who measure their glucose levels themselves may notice hyperglycemia (high blood sugar levels). In severe DKA, breathing becomes labored and of a deep, gasping character (a state referred to as "Kussmaul respiration"). The abdomen may be tender to the point that an acute abdomen may be suspected, such as acute pancreatitis, appendicitis or gastrointestinal perforation. Coffee ground vomiting (vomiting of altered blood) occurs in a minority of people; this tends to originate from erosion of the esophagus. In severe DKA, there may be confusion, lethargy, stupor or even coma (a marked decrease in the level of consciousness). On physical examination there is usually clinical evidence of dehydration, such as a dry mouth and decreased skin turgor. If the dehydration is profound enough to cause a decrease in the circulating blood volume, tachycardia (a fast heart rate) and low blood pressure may be observed. Often, a "ketotic" odor is present, which is often described as "fruity", often compared to the smell of pear drops whose scent is a ketone. If Kussmaul respiration is present, this is reflected in an increased respiratory rate.....

Alcoholic Ketoacidosis

Alcoholic Ketoacidosis Damian Baalmann, 2nd year EM resident A 45-year-old male presents to your emergency department with abdominal pain. He is conscious, lucid and as the nurses are hooking up the monitors, he explains to you that he began experiencing abdominal pain, nausea, vomiting about 2 days ago. Exam reveals a poorly groomed male with dry mucous membranes, diffusely tender abdomen with voluntary guarding. He is tachycardic, tachypneic but normotensive. A quick review of the chart reveals a prolonged history of alcohol abuse and after some questioning, the patient admits to a recent binge. Pertinent labs reveal slightly elevated anion-gap metabolic acidosis, normal glucose, ethanol level of 0, normal lipase and no ketones in the urine. What are your next steps in management? Alcoholic Ketoacidosis (AKA): What is it? Ketones are a form of energy made by the liver by free fatty acids released by adipose tissues. Normally, ketones are in small quantity (<0.1 mmol/L), but sometimes the body is forced to increase its production of these ketones. Ketones are strong acids and when they accumulate in large numbers, their presence leads to an acidosis. In alcoholics, a combination o Continue reading >>

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  1. Natalia & Enana

    Hi.
    I just put my cat in a veterinary emergency clinic with high ketoacidosis. They gave her an IV and insulin and other things I could afford. They told me to keep her there for 48 hours but I'm picking her up tomorrow and taking her to her vet. I dont know if my beautiful cat will survive. What are her chances? What are your suggestions.
    Thanks,
    Nat

  2. Sue and Oliver (GA)

    I am so sorry your kitty has DKA, Nat. It is possible to turn it around and she is in the best place to have that happen. We do have many cats who have survived and come home and lived long lives.
    What you can do - learn how to keep her safe at home. The best way to do that is to home test for blood glucose and for ketones. We test our cat's blood glucose levels just like we would our diabetic children. Here is a good site for beginning info: Newbie hometesting site and a video: Video for hometesting We have taught hundreds of people how to test over the internet. We would love to teach you. Testing for ketones will help you determine before DKA that he is heading into dangerous territory: ketones
    You can get the supplies for both these kinds of testing at any drug store. We use human glucometers and ketostix.
    Read about the best diet for your cat here: http://www.catinfo.org We feed wet lo carb food. BUT don't change the diet until you are hometesting. Oliver went down 100 points overnight when we switched from dry to wet. If we hadn't been hometesting, he would have overdosed.
    I am giving you a lot of info at once. The board is going down in 25 minutes and will be off for 2 hours for maintenance. Come back on later tonight or in the morning and post specifically for DKA. People who have dealt with it can give you lots of tips on how to care for your kitty when she gets home.

  3. Robert and Echo

    Nat, sending best wishes for your cat. Please keep us updated on her condition. Many cats do recover from DKA but, as you already know, it can be expensive treatment.
    _Rebecca

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Beste Online Kasinoer I Norge

Spilleautomater Dka Management Uptodate Pediatric diabetic ketoacidosis, uid therapy, and cerebral injury: Pediatric diabetic ketoacidosis, uid therapy, in DKA management continues to exist. Our general interest enewsletter keeps you up to date on a wide Mayo Clinic, MayoClinic. Diabetic ketoacidosis (DKA) is a common, serious, and preventable complication of type 1 diabetes, with a mortality of 35. Diabetic Ketoacidosis Abdelaziz Elamin Professor of Pediatric Endocrinology University of Khartoum, Sudan Management The management steps of DKA includes. Diabetes Management Uptodate Gestational Diabetes And Induction The 3 Step Trick that Reverses Diabetes Permanently in As Little as 11 Days. Diabetes Dka Symptoms Diabetes Type 1 Uptodate The 3 Step Trick that Reverses Diabetes medication management for diabetes type 2. Management of DKA in children the calculator, you must download it from the website to ensure that you are using the most uptodate version. Diabetes Treatment Uptodate: : We had been talking about pain management and the speaker launched a statement any. Diabetes Dka Talk to your doctor when you experience overwhelmed and work together to find a way to simplify your di Continue reading >>

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  1. GinaJ

    how long until ketosis begins?

    On a low carb diet, apporx how long does it take before you are in ketosis? I'm sure it varies, but just a rough idea.

  2. SandyR

    I've been told 3-4 days, but have not seen research on this.
    The pre-op LSd's need to be for a minimum of 2 weeks to be effective. - that IS proven and in the literature.
    Sandy r
    band educator

  3. Iam_with_the_Band

    It took me about 4 days. I checked with the Keto-strips from Walgreens. I also noticed that the first 4 days were the most difficult as far as exhaustion. I had absolutely NO energy. At the start of the 5th day, I was energized and I also noticed I was in Ketosis.

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Hyperglycemic crises: Hyperglycemic hyperosmolar nonketotic coma (HHNK) versus DKA. See DKA video here: https://youtu.be/r2tXTjb7EqU This video and similar images/videos are available for instant download licensing here https://www.alilamedicalmedia.com/-/g... Voice by: Penelope Hammet Alila Medical Media. All rights reserved. All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition. Support us on Patreon and get FREE downloads and other great rewards: patreon.com/AlilaMedicalMedia Hyperosmolar hyperglycemic state, or HHS, is another ACUTE and life-threatening complication of diabetes mellitus. It develops slower than DKA, typically in the course of several days, but has a much higher mortality rate. Like DKA, HHS is triggered when diabetic patients suffer from ADDITIONAL physiologic stress such as infections, other illness, INadequate diabetic treatment or certain drugs. Similar to DKA, the RISE in COUNTER-regulatory hormones is the major culprit. These hormones stimulate FURTHER production and release of glucose into the blood, causing it to overflow into urine, resulting in excessive LOSS of water and electrolytes. The major DIFFERENCE between HHS and DKA is the ABSENCE of acidosis in HHS. This is because, unlike DKA, the level of insulin in HHS patients is HIGH enough to SUPPRESS lipolysis and hence ketogenesis. This explains why HHS occurs more often in type 2 diabetics, who have more or less normal level of circulating insulin. Reminder: type 2 diabetics DO produce insulin but their cells do NOT respond to insulin and therefore cannot use glucose. Because symptoms of acidosis are NOT present, development of HHS may go UNnoticed until blood glucose levels become EXTREMELY high. Severe dehydration results in INcreased concentrations of solutes in the blood, raising its osmolarity. HyPERosmotic blood plasma drives water OUT of bodys tissues causing cellular dysfunction. Primary symptom of HHS is ALTERED consciousness due to excessive dehydration of brain tissues. This can range from confusion to coma. Emergency treatment consists of intravenous fluid, insulin and potassium similar to those used in DKA.

Diabetic Ketoacidosis And Hyperosmolar Hyperglycemic State In Adults: Epidemiology And Pathogenesis

INTRODUCTION Diabetic ketoacidosis (DKA) and hyperosmolar hyperglycemic state (HHS, also called hyperosmotic hyperglycemic nonketotic state) are two of the most serious acute complications of diabetes. They each represent an extreme in the hyperglycemic spectrum. The epidemiology and the factors responsible for the metabolic abnormalities of DKA and HHS in adults will be discussed here. The clinical features, evaluation, diagnosis, and treatment of these disorders are discussed separately. (See "Diabetic ketoacidosis and hyperosmolar hyperglycemic state in adults: Clinical features, evaluation, and diagnosis" and "Diabetic ketoacidosis and hyperosmolar hyperglycemic state in adults: Treatment".) EPIDEMIOLOGY Diabetic ketoacidosis (DKA) is characteristically associated with type 1 diabetes. It also occurs in type 2 diabetes under conditions of extreme stress such as serious infection, trauma, cardiovascular or other emergencies, and, less often, as a presenting manifestation of type 2 diabetes, a disorder called ketosis-prone diabetes mellitus. (See "Syndromes of ketosis-prone diabetes mellitus".) DKA is more common in young (<65 years) patients, whereas hyperosmolar hyperglycemic s Continue reading >>

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  1. beezala

    Hey everyone,
    I just wanted to tell people the cause of my hair loss issue, and to tell you to GO TO YOUR DOCTOR IF YOU ARE LOSING HAIR ON KETO. After 3 months I started losing hair and got a few greys, which I've never had before.
    Under everyone's advice here and on the web, I assumed it was telogen effluvium, which is common and temporary.
    After getting blood work, my doctor said I am very low in b12, which cab cause hair loss and loss of hair pigment. The solution for me: supplement daily with 1000mg b12. This is my first day supplementing.
    If left untreated, low b12 can be catastrophic. So please, do get checked out. B12 comes from animal products, so you must be wondering HOW I could possibly low on that if I eat keto.
    Apparently, it has something to do with grass fed animal products; the meat and cheese from these animals has higher levels of b12 than that of the grain fed variety. Also, it can be found in fruits and grains, which have been completely removed from my diet.
    Just some food for thought.
    EDIT: I've just read about how antibiotics can lead to b12 deficiency. I have posted before about antibiotics ruining my gut microbiome, and now I think it may have been the cause of my b12 deficiency. Apparently bad gut bacteria eats up the b12, so an overgrowth of it may be the culprit. But won't jump to conclusions until I speak with my doctor. The antibiotic I took was doxycycline for keto rash...
    UPDATE: Today is the first day I am noticing a little less hair loss than normal and it has been exactly 3 weeks since I started supplementing. There is less hair on my clothing/around me, but I am still not back to normal. Another symptom I've had is very dry/scaly/itchy scalp, which has appeared to calm down a bit the last couple of days. I'm hoping this means my body is readjusting to normal levels! Feeling optimistic.
    UPDATE 2: Confirming that after almost exactly 1 month of b12 supplementation, my hair loss is back to normal! Also, want to clarify that all of this is most likely NOT related to the Keto WOE, as my iron levels are also low and this is genetic, and apparently, they are related. Still waiting to hear from my doctor, but I may have a minor form of Thalassemia.

  2. hazeFL

    There is no B12 in fruit or grains.

  3. Addbutter

    I thought it was common knowledge since vegetarians always have to worry about not getting b12.

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