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Dka Death Rate

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Respiratory acidosis #sign and symptoms of Respiratory acidosis Respiratory acidosis ABGs Analyse https://youtu.be/L5MWy1iHacI Plz share n subscribe my chanel is a condition that occurs when the lungs cant remove enough of the Suctioning https://youtu.be/hMJGkxvXTW0 carbon dioxide (CO2) produced by the body. Excess CO2 causes the pH of blood and other bodily fluids to decrease, making them too acidic. Normally, the body is able to balance the ions that control acidity. This balance is measured on a pH scale from 0 to 14. Acidosis occurs when the pH of the blood falls below 7.35 (normal blood pH is between 7.35 and 7.45).Rinku Chaudhary NSG officer AMU ALIGARH https://www.facebook.com/rinkutch/ Respiratory acidosis is typically caused by an underlying disease or condition. This is also called respiratory failure or ventilatory failure. Suctioning https://youtu.be/hMJGkxvXTW0 Normally, the lungs take in oxygen and exhale CO2. Oxygen passes from the lungs into the blood. CO2 passes from the blood into the lungs. However, sometimes the lungs cant remove enough CO2. This may be due to a decrease in respiratory rate or decrease in air movement due to an underlying condition such as: asth

Outcome Of Patients With Diabetic Ketoacidosis And Acute Respiratory Failure

Abstract SESSION TITLE: Respiratory Support Posters SESSION TYPE: Original Investigation Poster PURPOSE: Diabetic ketoacidosis (DKA) is a life threatening metabolic derangement. Acute respiratory failure in DKA is a strong mortality predictor and is usually secondary to both a decreased level of consciousness and severe acidosis. We assessed the outcome of patients admitted with DKA and acute respiratory failure. METHODS: A retrospective cohort analysis was done on patients admitted to our hospital with DKA and acute respiratory failure between January 2008 and December 2013. The baseline demographic information, clinical and laboratory characteristics were collected. The primary outcome of the study was in-hospital mortality. A univariate analysis was done to identify association of variables with the outcome. Continuous variables were reported as mean (SD) and compared via the student t test. Comparison of categorical data was made via the chi-square test. RESULTS: The cohort consisted of 76 patients admitted with diabetic ketoacidosis and acute respiratory failure. Of these patients, 28 deaths were noted resulting in a mortality rate of 37%. On univariate analysis, the presence Continue reading >>

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  1. manohman

    Why can't fat be converted into Glucose?

    So the reason cited is that beta oxidation/metabolism of fats leads to formation of acetyl coa, a 2 carbon molecule, and that because of that it cannot be converted back into glucose.
    Why exactly is that the case?
    If Glucogenic amino acids can be converted into citric acid cycle intermediates and then turn back into glucose via gluconeogensis, then why cant Fatty Acids which yield Acetyl Coa. Can't you just have Acetyl Coa enter the citric acid cycle and produce the same intermediates that the glucogenic amino acids creat?

  2. Czarcasm

    manohman said: ↑
    So the reason cited is that beta oxidation/metabolism of fats leads to formation of acetyl coa, a 2 carbon molecule, and that because of that it cannot be converted back into glucose.
    Why exactly is that the case?
    If Glucogenic amino acids can be converted into citric acid cycle intermediates and then turn back into glucose via gluconeogensis, then why cant Fatty Acids which yield Acetyl Coa. Can't you just have Acetyl Coa enter the citric acid cycle and produce the same intermediates that the glucogenic amino acids creat?
    Click to expand... Both glucose and fatty acids can be stored in the body as either glycogen for glucose (stored mainly in the liver or skeletal cells) or for FA's, as triacylglycerides (stored in adipose cells). We cannot store excess protein. It's either used to make other proteins, or flushed out of the body if in excess; that's generally the case but we try to make use of some of that energy instead of throwing it all away.
    When a person is deprived of nutrition for a period of time and glycogen stores are depleted, the body will immediately seek out alternative energy sources. Fats (stored for use) are the first priority over protein (which requires the breakdown of tissues such as muscle). We can mobilize these FA's to the liver and convert them to Acetyl-CoA to be used in the TCA cycle and generate much needed energy. On the contrary, when a person eats in excess (a fatty meal high in protein), it's more efficient to store fatty acids as TAG's over glycogen simply because glycogen is extremely hydrophilic and attracts excess water weight; fatty acids are largely stored anhydrously and so you essentially get more bang for your buck. This is evolutionary significant and why birds are able to stay light weight but fly for periods at a time, or why bears are able to hibernate for months at a time. Proteins on the other hand may be used anabolically to build up active tissues (such as when your working out those muscles), unless you live a sedentary lifestyle (less anabolism and therefore, less use of the proteins). As part of the excretion process, protein must be broken down to urea to avoid toxic ammonia and in doing so, the Liver can extract some of that usable energy for storage as glycogen.
    Also, it is worth noting that it is indeed possible to convert FA's to glucose but the pathway can be a little complex and so in terms of energy storage, is not very efficient. The process involves converting Acetyl-CoA to Acetone (transported out of mitochondria to cytosol) where it's converted to Pyruvate which can then be used in the Gluconeogenesis pathway to make Glucose and eventually stored as Glycogen. Have a look for yourself if your interested: http://www.ploscompbiol.org/article/info:doi/10.1371/journal.pcbi.1002116.g003/originalimage (and this excludes the whole glycogenesis pathway, which hasn't even begun yet).
    TLDR: it's because proteins have no ability to be stored in the body, but we can convert them to glycogen for storage during the breakdown process for excretion. Also, in terms of energy, it's a more efficient process than converting FA's to glycogen for storage.

  3. soccerman93

    This is where biochem comes in handy. Czarcasm gives a really good in depth answer, but a simpler approach is to count carbons. The first step of gluconeogenesis(formation of glucose) requires pyruvate, a 3 carbon molecule. Acetyl Co-A is a 2 carbon molecule, and most animals lack the enzymes (malate synthase and isocitrate lyase) required to convert acetyl co-A into a 3 carbon molecule suitable for the gluconeogenesis pathway. The ketogenic pathway is not efficient, as czarcasm pointed out. While acetyl co-A can indeed be used to form citric acid intermediates, these intermediates will be used in forming ATP, not glucose. Fatty acid oxidation does not yield suitable amounts of pyruvate, which is required for gluconeogenesis. This is part of why losing weight is fairly difficult for those that are overweight, we can't efficiently directly convert fat to glucose, which we need a fairly constant supply of. Sorry, that got a little long-winded

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National diabetes care home survey highlights 'concerning' results October 4th, 2013 Almost one in five care home residents with diabetes self-medicate without any checks, according to new research. Self-management is usually encouraged, but vulnerable people with diabetes, especially those on the potentially-dangerous drug insulin, should be checked to ensure they have taken their medicine to prevent complications, including descending into a coma. The first-ever National Care Home Diabetes Audit also revealed that more than 60 per cent of the 2,043 premises surveyed did not have a designated member of staff with responsibility for diabetes management. The audit was carried out by the Institute of Diabetes for Older People (IDOP) in partnership with ABCD (Association of British Clinical Diabetologists) and a group of further key collaborators (listed below). The audit results found only one in 10 care home residents were reported as having diabetes although this figure is at odds with previous research that showed as many as 26 per cent of all care home residents actually have diabetes. However, despite this high number, 63.2 per cent of homes did not have a designated member of s

National Diabetes Audit - 2011-12: Report 2

Summary 2011-2012 National Diabetes Audit (NDA) report. Report 2 shows Complications and Mortality This national report from the ninth year of the NDA, presents key findings on complications in 2010-2012 and deaths in 2012 for all age groups. This report presents statistics about diabetes outcomes including Diabetic Ketoacidosis (DKA), chronic kidney disease and treatment of end stage disease (renal replacement therapy, RRT), lower limb amputations, retinopathy treatment, heart disease, stroke and mortality. Due to the size of this publication, the data have been organised alphabetically by CCG/LHB profile and split into several pages, which are linked to above. Key Facts More than a quarter of admissions to hospital with heart failure involve a patient with diabetes (28 per cent, or 198,200 of 717,100 admissions during 2010-2012). The National Diabetes Audit published today recorded over two million patients with diabetes and shows people with diabetes have a 74 per cent greater risk of being admitted to hospital for heart failure compared to the rest of the population. Of the 198,100 people in the audit with type 1 diabetes in England and Wales in 2012, 3,300 died during the year Continue reading >>

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Popular Questions

  1. Frentzke

    I'm curious to hear the experiences of others with distance running while being Keto. I'm in the second week of training for a half marathon in April. I've had no problem running 3 miles fasted for around 22 hours, but I'm interested in the possible need to fuel with fat on longer runs. Last October I completed a 10k after doing a fat fast for 5 days and then the morning of the race ate three eggs fried in a ton of butter. I actually beat my best training time by a decent amount with this plan, but 13.1 miles could require a different strategy. Thanks for any tips and Keto on!

  2. bodwest

    Check this podcast out - it has information that should be very helpful: https://blog.bulletproof.com/102-extreme-endurance-training-and-ketosis-with-ben-greenfield-podcast/
    818

  3. Jacquie

    I think @trekkin1 is a keto long distance runner and @Emacfarland is a shorter keto distance runner. Hopefully, they will chime in soon with lots of good information.

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The Occurrence Of Diabetic Ketoacidosis In

Type 2 Diabetic Adults Chih-Hsun Chu; Jenn-Kuen Lee; Hing-Chung Lam; Chih-Chen Lu Division of Endocrinology and Metabolism, Department of Medicine, Veterans General Hospital-Kaohsiung, School of Medicine, National Yang-Ming University, Taipei, Taiwan. Running title: The Occurrence of Diabetic Ketoacidosis in Type 2 Diabetic Adults Abstract OBJECTIVE. To study the diabetic ketoacidosis (DKA) episodes which occurred in the type 2 diabetic adults. STUDY DESIGN. We reviewed retrospectively the charts of patients who were admitted to the division of endocrinology and metabolism from Jan. 1991 to Dec. 1997 due to DKA. RESULTS. Total 121 adult patients with 137 episodes (57 females and 80 males) of DKA, with mean age of 45.90 years. 98 episodes (71.5%) occurred in type 2 diabetes mellitus (DM) with mean age of 48.73, which was significantly older than type 1 diabetic patients. Among ten patients suffered from repeated episodes of DKA, 2 four patients belonged to type 2 DM. 33 episodes (24.1%) occurred in patients without a history of DM, however, up to 24 episodes were classified as in type 2. Infection was the most important precipitating factor in type 2 diabetic patients, with respirat Continue reading >>

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Popular Questions

  1. carissanbo

    I'm just wondering if there are ways to tell if you're in Ketosis without using the strips. We live in the Outback in Australia in a small town and I'm not sure I would be able to find them here. And to be honest, I really don't feel like checking. I feel that I finally found a WOE that works for me and I'm feeling great and don't want to focus too much on that aspect. So I'm wondering if there are signs to tell me.
    I'm finishing up my 3rd day of induction and to be honest, it's been pretty easy for me. I haven't really had any bad side effects and after supper I have no urges or cravings to eat anything (and no urges for carbs during the day). I have ALWAYS had a snack before bed my whole life, but I've really had no interest.
    I just want to make sure I'm doing this right. If you go to my journal you can check out my menu for today if you'd like to see what I'm eating. And I have lost a few pounds.
    Thanks!

  2. Sinbad

    Hiya!
    You should update your profile with your weight loss so we can share your happiness
    As for ketosis, I can tell without strips because I sometimes get an odd tinny taste in my mouth, I don't feel hungry or sluggish or headachey at all, and most convincing of all (for me) is that my pee smells almost of acetone... Sometimes it looks almost greenish too but that could be because of the supplements I'm taking (or all the green veggies I'm eating, which I never used to! )
    Of course, the best sign that you're in ketosis/lipolysis is that you're losing weight!
    Good luck with the plan!
    Steven

  3. btdude

    Agreed. Metal mouth prevails. You are in the bathroom every (at least) 35.4 minutes.

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