Conversion Of Excess Glucose To Fat Is Called

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Video by Ulf Smith, MD, PhD, Professor of Internal Medicine, The Lundberg Laboratory for Diabetes Research, Center of Excellence for Cardiovascular and Metabolic Research, Sahlgrenska Academy, Gteborg University, Gteborg, Sweden Produced by the International Chair on Cardiometabolic Risk

Conversion Of Carbohydrate To Fat In Adipose Tissue: An Energy-yielding And,therefore, Self-limiting Process.

Conversion of carbohydrate to fat in adipose tissue: an energy-yielding and,therefore, self-limiting process. A theoretical analysis of the energy metabolism associated with the conversion ofglucose to fat is presented. In tissues where the pentose cycle furnishes some ofthe NADPH required for fatty acid synthesis, this conversion is an ATP-yieldingprocess. In rat adipose tissue the maximal rate of glucose conversion to fat can be quantatively predicted on the basis of the tissue's ability to use the ATPwhich is generated in excess during this conversion. The energy-generating natureof this process provides the means for a type of regulation which depends onmetabolic state and which, during fasting, contributes to the sparing ofcarbohydrate. Impairment of lipogenesis in the fasting state is attributed to adecrease in the activity of the malate cycle and to the presence of free fattyacids. However, rather than by inhibiting specific enzymes, it is by virtue oftheir quality as substrates for energy production that free fatty acids and theirCoA derivatives appear to inhibit de novo lipogenesis. The regulatory phenomenadiscussed here may explain the failure of the attempts made to ide Continue reading >>

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  1. Saphire

    So the last few days I have been sleeping really well. Barely got up, was able to sleep in the morning which is something I have always done, I am a night person and I like it that way. However, while I was not cheating, between all the supplements I have been taking for some aches and pains and the extra nuts I have been snacking on I suspected I was not in ketosis. Yesterday I skipped lunch unintentionally and started what is now a 40 hour fast. Last night my sleep went back to how it is on keto/fasting. I woke up several times to use the facilities and this morning woke up an hour earlier than usual and could not go back to sleep (no matter how much I wanted to). I do not feel refreshed but I do feel alert. On the flip side I usually fall asleep earlier than usual with keto.

    I have always had a bit of a delayed circadian issue but I like it and am used to it. When on keto it seems to fix it? Is that possible?

  2. David_Driver

    I am going to have to give up on Keto. I have had insomnia for almost a year and I discovered that when I cheat I sleep all night. This week I have bumped up the carbs significantly and I have slept all the way through every night. If you google low carb and insomnia you will find that it is fairly common.

  3. Loulou

    Perhaps that is why my insomnia has been so bad recently. Only slept 4 hours last night. However, if I eat more than 20 grams carbs I immediately put on 1 or 2 lbs over night which is a no no for me,

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A&p 2 Ch. 25a Flashcards | Quizlet

contains twice as much energy as carbs, they are harder to catabolize. is a form of lipid catabolism. catabolized into glycerol and fatty acids by bile and lipases. This is promoted by cortisol, epinephrine and norepinephrine. is converted to glyceraldehyde 3 phosphate, part of the glycolysis pathway. If individual DOES NOT need ATP, G 3-P converted into glucose to be stored for later! if individual DOES need ATP, G 3-P will undergo glycolysis, producing pyruvic acid which will continue through the steps of cellular respiration. is called beta oxidation (occurs in inner matrix of mitochondria) fatty acid chain broken down into small fragments consisting on two carbon atoms. The two carbon fragments are attached to coenzymes A to form acetyl coenzyme A which can enter the krebs cycle and continue through cell respiration. hepatocytes can take two acetyl Co A molecules and condense them into X acid this will occur if there are many acetyl CoA molecules in blood. Some acetoacetic acid is converted into B-hydroxybutyric acid and acetone (all three substances called ketone bodies) Detone bodies diffuse into the blood and can be used by other cells to make acetyl CoA again. excessive pr Continue reading >>

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  1. iwannabeayummymummy

    Where do you buy ketone sticks/strips from on the net? i'm from UK so need a UK site not exspensive either lol

  2. elliebird

    You can buy them in Boots, they will be behind the counter so ask the pharmacist, they are about £5 - I hope this helps

  3. Yambabe

    I got mine from sainsburys, again you have to go to the pharmacy and ask.

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Lipid Metabolism

on on Fats (or triglycerides) within the body are ingested as food or synthesized by adipocytes or hepatocytes from carbohydrate precursors ([link]). Lipid metabolism entails the oxidation of fatty acids to either generate energy or synthesize new lipids from smaller constituent molecules. Lipid metabolism is associated with carbohydrate metabolism, as products of glucose (such as acetyl CoA) can be converted into lipids. Lipid metabolism begins in the intestine where ingested triglycerides are broken down into smaller chain fatty acids and subsequently into monoglyceride molecules (see [link]b) by pancreatic lipases, enzymes that break down fats after they are emulsified by bile salts. When food reaches the small intestine in the form of chyme, a digestive hormone called cholecystokinin (CCK) is released by intestinal cells in the intestinal mucosa. CCK stimulates the release of pancreatic lipase from the pancreas and stimulates the contraction of the gallbladder to release stored bile salts into the intestine. CCK also travels to the brain, where it can act as a hunger suppressant. Together, the pancreatic lipases and bile salts break down triglycerides into free fatty acids. The Continue reading >>

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  1. RedhairedNurse

    Your nursing text should point out the difference. I would tell you, but I'd just have to look it up and my books are in storage. I could also google it, but something you can also do as well. Sorry.

  2. RedhairedNurse


  3. Ilithya

    In HHNS, blood sugar levels rise, and your body tries to get rid of the excess sugar by passing it into your urine, your body tries to compensate. This usually happens to type 2s
    In DKA there is little to no circulating insulin. DKA occurs mainly, but not exclusively, in Type 1 diabetes because Type 1 diabetes is characterized by a lack of insulin production in the pancreas. It is much less common in Type 2 diabetes because the latter is closely related to cell insensitivity to insulin, not -- at least initially -- to a shortage or absence of insulin. Some Type 2 diabetics have lost their own insulin production and must take external insulin; they have some susceptibility to DKA. You get acidosis in DKA because ketones lower the bloods pH.
    Does that help?

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