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Alcoholic Ketoacidosis Sudden Death

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What is HYPOKALEMIA? What does HYPOKALEMIA mean? HYPOKALEMIA meaning - HYPOKALEMIA pronunciation - HYPOKALEMIA definition - HYPOKALEMIA explanation - How to pronounce HYPOKALEMIA? Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/... license. Hypokalemia, also spelled hypokalaemia, is a low level of potassium (K+) in the blood serum. Normal potassium levels are between 3.5 and 5.0 mmol/L (3.5 and 5.0 mEq/L) with levels below 3.5 mmol/L defined as hypokalemia. Mildly low levels do not typically cause symptoms. Symptoms may include feeling tired, leg cramps, weakness, and constipation. It increases the risk of an abnormal heart rhythm such as bradycardia and cardiac arrest. Causes of hypokalemia include diarrhea, medications like furosemide and steroids, dialysis, diabetes insipidus, hyperaldosteronism, hypomagnesemia, and not enough intake in the diet. It is classified as severe when levels are less than 2.5 mmol/L. Low levels can also be detected on an electrocardiogram (ECG). Hyperkalemia refers to a high level of potassium in the blood serum. The speed at which potassium should be replaced depends on whether or not there are symptoms or ECG changes

Sudden Cardiac Arrest Induced By Hypoglycemia And Hypokalemia In A Chronic Alcoholic Patient

Sudden cardiac arrest induced by hypoglycemia and hypokalemia in a chronic alcoholic patient Chair and Department of Internal Medicine, Medical University, Lublin, Poland Student Scientifi c Society, Medical University, Lublin, Poland Chair and Department of Nephrology, Medical University, Lublin, Poland A 44-year-old alcoholic patient was admitted to the Cardiac Intensive Care Unit due to sudden cardiac arrest in the mechanism of asystole. Additional examinations revealed hypoglycemia 13 mg/dl and hypokalemia 3.27 mmol/l. Thanks to prompt resuscitation and intravenous infusion of 40% glucose and potassium, the condition of the patient improved. Based on the case reported, causes of sudden cardiac death in alcoholics are discussed. Department of Internal Medicine,Medical University, Staszica 16, 20-081 Lublin, Poland. Sovari A, Kocheril AG, Baas AS: Sudden Cardiac Death, at: http:// emedicine.medscape.com/article... . Brown TM, Harvey AM: Spontaneous hypoglycemia in smoke drinkers. JAMA 941, 117, 12-15. Robinson RT, Harris ND, Ireland RH, Lee S, Newman C, Heller SR: Mechanisms of abnormal cardiac repolarisation during insulin-induced hypoglycemia. Diabetes 2003, 52, 1469-1474. Gor Continue reading >>

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  1. Polly_ana85

    Is keto worth the effort or would I lose just as much restricting 800 calories? Keto would be 1000.
    What's better?

  2. Procaffeinating

    I've wondered the same thing. There seem to be a lot of people who swear by keto, so I'm thinking of giving it a try. Following this thread

  3. Polly_ana85

    Yeah I'm wanting to lose weight! I'm over eating at the moment and hover at 1200 cals but a weekend away I was averaging 3000 over three days...alchol and food
    So I need a change!! I'm thinking keto because restricting makes me bingy

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Alcoholism is disease, heres some resources to help you fight back: Responsible Drinking: A Moderation Management Approach http://amzn.to/1ZdgP9f I Need to Stop Drinking!: How to get back your self-respect. http://amzn.to/1VEqbeU Why You Drink and How to Stop: A Journey to Freedom: http://amzn.to/1Q8pAv2 Alcoholics Anonymous: The Big Book: http://amzn.to/1N0rttl Alcoholics: Dealing With an Alcoholic Family Member, Friend or Someone You Love: http://amzn.to/1j9cvH4 Watch more How to Understand Alcoholism videos: http://www.howcast.com/videos/517398-... The question that has been asked of me is if alcoholism can lead to diabetes. And if so, how? The answer is chronic alcohol use can lead to diabetes. The way it leads to diabetes is that chronic alcohol use can cause inflammation of the pancreas, and chronic inflammation of the pancreas can affect the production of insulin in the body. And that's what causes diabetes. So that is why alcohol can be an actual primary determinate of diabetes. The other way that heavy alcohol use can lead to diabetes or exacerbate diabetes is that alcohol has a high content of sugar. So if one is already diabetic, alcohol is really not indicated because o

Homepage Of Awareness Of Sudden Death Syndrome In Alcoholism

Alcoholism is rampant in U.S. and world.People like you reading this page know someone who is alcoholic. They canbe your relatives, friends or co-workers. We are all familiar with thealcohol associated problems in our daily life. These include alcohol relatedsocial, occupational, or legal difficulties such as alcohol related blackouts,argument with friends/family, marital problems, poor job performance, decreasedlife quality and so on. Health professionals may be familiar with the definitionof alcohol abuse or alcohol dependence (as indicated in DSM-III), alcoholrelated health problems such as alcohol withdrawal, alcohol hepatitis,cirrhosis, GI bleeding, cardiomyopathy, and CNS complications (deliriumtremens, generalized seizures), etc. However, few people are aware of thesudden death syndrome associated with alcoholism. The sudden, unexpecteddeath associated with alcoholism is one of the most mystifying complicationsof the abuse of alcohol and it is an entity so poorly understood that itdoes not even bear a generally accepted label or name. In general, deathassociated with alcohol may be classified as following categories: 1. Acute alcoholism and acutealcohol poisoning 3. Increas Continue reading >>

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  1. LoveANurse

    ketosis signs?

    what are some obviouse physical signs that you may be in ketosis.
    breath? urine? mentality?

  2. stang1101

    bad breath, urine stinks. Get some ketostix to get a more accurate reading.

  3. New_York

    Brain fog, bad breath, urine stinks, urine is always yellow even when hydrated(spelling?)...

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Football is by far the world's most popular sport and because of this, the occurrence of sudden cardiac death appears more prevalent. It also seems to be African players who are more likely to fall victim to the problem...the question is why are healthy players dying and what needs to be done to stop it from happening again. CGTN's Sias du Plessis takes a look. Subscribe to us on YouTube: http://ow.ly/Zvqj30aIsgY Follow us on: Facebook: https://www.facebook.com/cgtnafrica/ Twitter: https://twitter.com/cgtnafrica

Sudden Unexpected Death In Alcohol Misusean Unrecognized Public Health Issue?

Int J Environ Res Public Health. 2009 Dec; 6(12): 30703081. Published online 2009 Dec 4. doi: 10.3390/ijerph6123070 Sudden Unexpected Death in Alcohol MisuseAn Unrecognized Public Health Issue? Alexa H. Templeton ,1,* Karen L.T. Carter ,1 Nick Sheron ,1,2 Patrick J. Gallagher ,3 and Clare Verrill 3 1 Liver Research Group, University of Southampton, Southampton General Hospital, Southampton University Hospitals NHS Trust, Tremona Road, Southampton, SO16 6YD, UK; E-Mails: [email protected] (K.L.T.C.); [email protected] (N.S.) 2 Clinical Hepatology, Division of Infection, Inflammation and Immunity, University of Southampton Medical School, Southampton, SO16 6YD, UK 3 Department of Histopathology, Southampton University Hospitals NHS Trust, Southampton, UK; E-Mails: [email protected] (P.J.G.); [email protected] (C.V.) * Author to whom correspondence should be addressed; E-Mail: [email protected] ; Tel.: 07841427173; Fax: +44(0)23 8079 6603. Received 2009 Oct 29; Accepted 2009 Nov 29. Copyright 2009 by the authors; licensee Molecular Diversity Preservation International, Basel, Switzerland. This article is an open-access article distributed under the ter Continue reading >>

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  1. manohman

    Why can't fat be converted into Glucose?

    So the reason cited is that beta oxidation/metabolism of fats leads to formation of acetyl coa, a 2 carbon molecule, and that because of that it cannot be converted back into glucose.
    Why exactly is that the case?
    If Glucogenic amino acids can be converted into citric acid cycle intermediates and then turn back into glucose via gluconeogensis, then why cant Fatty Acids which yield Acetyl Coa. Can't you just have Acetyl Coa enter the citric acid cycle and produce the same intermediates that the glucogenic amino acids creat?

  2. Czarcasm

    manohman said: ↑
    So the reason cited is that beta oxidation/metabolism of fats leads to formation of acetyl coa, a 2 carbon molecule, and that because of that it cannot be converted back into glucose.
    Why exactly is that the case?
    If Glucogenic amino acids can be converted into citric acid cycle intermediates and then turn back into glucose via gluconeogensis, then why cant Fatty Acids which yield Acetyl Coa. Can't you just have Acetyl Coa enter the citric acid cycle and produce the same intermediates that the glucogenic amino acids creat?
    Click to expand... Both glucose and fatty acids can be stored in the body as either glycogen for glucose (stored mainly in the liver or skeletal cells) or for FA's, as triacylglycerides (stored in adipose cells). We cannot store excess protein. It's either used to make other proteins, or flushed out of the body if in excess; that's generally the case but we try to make use of some of that energy instead of throwing it all away.
    When a person is deprived of nutrition for a period of time and glycogen stores are depleted, the body will immediately seek out alternative energy sources. Fats (stored for use) are the first priority over protein (which requires the breakdown of tissues such as muscle). We can mobilize these FA's to the liver and convert them to Acetyl-CoA to be used in the TCA cycle and generate much needed energy. On the contrary, when a person eats in excess (a fatty meal high in protein), it's more efficient to store fatty acids as TAG's over glycogen simply because glycogen is extremely hydrophilic and attracts excess water weight; fatty acids are largely stored anhydrously and so you essentially get more bang for your buck. This is evolutionary significant and why birds are able to stay light weight but fly for periods at a time, or why bears are able to hibernate for months at a time. Proteins on the other hand may be used anabolically to build up active tissues (such as when your working out those muscles), unless you live a sedentary lifestyle (less anabolism and therefore, less use of the proteins). As part of the excretion process, protein must be broken down to urea to avoid toxic ammonia and in doing so, the Liver can extract some of that usable energy for storage as glycogen.
    Also, it is worth noting that it is indeed possible to convert FA's to glucose but the pathway can be a little complex and so in terms of energy storage, is not very efficient. The process involves converting Acetyl-CoA to Acetone (transported out of mitochondria to cytosol) where it's converted to Pyruvate which can then be used in the Gluconeogenesis pathway to make Glucose and eventually stored as Glycogen. Have a look for yourself if your interested: http://www.ploscompbiol.org/article/info:doi/10.1371/journal.pcbi.1002116.g003/originalimage (and this excludes the whole glycogenesis pathway, which hasn't even begun yet).
    TLDR: it's because proteins have no ability to be stored in the body, but we can convert them to glycogen for storage during the breakdown process for excretion. Also, in terms of energy, it's a more efficient process than converting FA's to glycogen for storage.

  3. soccerman93

    This is where biochem comes in handy. Czarcasm gives a really good in depth answer, but a simpler approach is to count carbons. The first step of gluconeogenesis(formation of glucose) requires pyruvate, a 3 carbon molecule. Acetyl Co-A is a 2 carbon molecule, and most animals lack the enzymes (malate synthase and isocitrate lyase) required to convert acetyl co-A into a 3 carbon molecule suitable for the gluconeogenesis pathway. The ketogenic pathway is not efficient, as czarcasm pointed out. While acetyl co-A can indeed be used to form citric acid intermediates, these intermediates will be used in forming ATP, not glucose. Fatty acid oxidation does not yield suitable amounts of pyruvate, which is required for gluconeogenesis. This is part of why losing weight is fairly difficult for those that are overweight, we can't efficiently directly convert fat to glucose, which we need a fairly constant supply of. Sorry, that got a little long-winded

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