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Alcoholic Ketoacidosis Cause Of Death

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Diet Of Alcohol Killed High-flier

A RETIRED stock market trader died from the effects of chronic alcoholism after turning to drink to ease the stress of her job. Could not subscribe, try again laterInvalid Email A RETIRED stock market trader died from the effects of chronic alcoholism after turning to drink to ease the stress of her job. Jill Elliot, otherwise known as Ann Margaret Elliot, 50, was found dead in her flat on Bridge Street Row, Chester, on February 2. Cheshire coroners court heard that Mrs Elliot had led a high-flying career as an oil trader in London, but developed an alcohol problem because of the high level of pressure involved. Her family last heard from her in a telephone call on January 31, when Mrs Elliot said she was feeling ill and was going to take the telephone off the hook and try to rest. Her sister Julia Kay Slack, of Rochdale, told the inquest that she became worried when three days later she still could not contact her and phoned Cheshire Police. Officers had to force entry to the locked flat and found Mrs Elliot slumped on the floor at the bottom of the stairs. Dr Sally Ann Hales, of the Countess of Chester Hospital pathology department, told the court that a post mortem investigatio Continue reading >>

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  1. Santosh Anand

    Insulin plays a key role in helping sugar (glucose) enter your cells, thus providing them energy. When your cells don't get the glucose they need for energy, your body begins to burn fat for energy, which produces ketones. Ketones are acidic and so when they build up in the blood, they make the blood more acidic, leading to the condition called diabetic ketoacidosis (DKA).
    Now, in type-1 diabetes, there is no insulin production whereas in type-2, there is impairment of insulin production. Thus why Type-2 diabetic people hardly get DKA.
    Note: Diabetic ketoacidosis is a serious condition that might lead to diabetic coma or even death.

  2. Lucas Verhelst

    In order for the cells in your body to access the glucose in your bloodstream so they can use it as energy they need insulin. Insulin acts like a key, opennin the cell door to allow the entry of glucose. Type 1 diabetics produce no insulin and need to inject it, thus the amount of insulin they have is strictly limited. Once they run out of insulin the glucose remains in the blood stream. If this occurs over a long period of time their blood glucose levels will rise due to the release of glucose from the liver. High blood sugar levels causes ketoacidosis which leads to coma and death.

  3. Keith Phillips

    Although type 2 diabetics suffer from insulin resistance, the condition rarely has an absolute negative effect on the bodies ability to convert glucose to usable energy. Type 1 diabetics have little or no ability to produce insulin. With the exception of neural cells, the rest of the body which without insulin is experiencing starvation, will consume its own tissues. (this is how people have endured periods of famine). This process however produces by products that eventually overwhelm the body's ability to process toxins.

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The Postmortem Diagnosis Of Alcoholic Ketoacidosis.

Alcohol Alcohol. 2014 May-Jun;49(3):271-81. doi: 10.1093/alcalc/agt177. Epub 2013 Dec 10. The postmortem diagnosis of alcoholic ketoacidosis. Corresponding author: University Center of Legal Medicine, Rue du Bugnon 21, 1011 Lausanne, Switzerland. [email protected] The aim of this article is to review the forensic literature covering the postmortem investigations that are associated with alcoholic ketoacidosis fatalities and report the results of our own analyses. Eight cases of suspected alcoholic ketoacidosis that had undergone medico-legal investigations in our facility from 2011 to 2013 were retrospectively selected. A series of laboratory parameters were measured in whole femoral blood, postmortem serum from femoral blood, urine and vitreous humor in order to obtain a more general overview on the biochemical and metabolic changes that occur during alcoholic ketoacidosis. Most of the tested parameters were chosen among those that had been described in clinical and forensic literature associated with alcoholic ketoacidosis and its complications. Ketone bodies and carbohydrate-deficient transferrin levels were increased in all cases. Biochemical markers of generalized in Continue reading >>

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  1. lorrained8

    Metformin and lactic acidosis

    Has anyone here had lactic acidosis because of taking metformin
    I have leg and muscular pain I feel tired and weak
    I've discontinued my met er
    I will talk to my dr tomorrow

  2. Sweetpoison

    Originally Posted by lorrained8
    I have leg and muscular pain I feel tired and weak
    I have leg pain and bad night cramps but I had that occasionally before I even started Met. I am sorry, Lorraine, this is happening to you!
    How long have you been on the Met ER before you realized this?

  3. Lynnw

    Originally Posted by lorrained8
    Has anyone here had lactic acidosis because of taking metformin
    I have leg and muscular pain I feel tired and weak
    I've discontinued my met er
    I will talk to my dr tomorrow Are you on any other medications that could cause that? How long have you been on the Metformin? Metformin made me incredibly fatigued, to the point where I couldn't do anything but drag myself from my bed to my chair (I'm normally a very active person). I never figured out if that was lactic acidosis, Vit B-12 deficiency (which Metformin can cause), or something else. Others have had the same experience.

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Alcoholic Hypoglycemia - Effects, Causes, Symptoms, and Treatment What is Hypoglycemia? This is the opposite of Hyperglycemia; it is when you have chronic low levels of blood glucose. Causes include: Drinking too much alcohol Skipping meals or not eating enough at mealtime Overdosing on diabetes medication Overexertion What are the Symptoms of Hypoglycemia? Symptoms include: Hunger Nervousness and shakiness Perspiration Dizziness or light-headedness Sleepiness Confusion Difficulty speaking Feeling anxious or weakness Treatment for Hypoglycemia Some of the treatments for hypoglycemia include raising your blood sugar levels by eating glucose or simple carbohydrates. That means eating something like some raisins, regular soda (the sugary kind), some hard sweets or jellies, or even eating pure table sugar, honey, molasses, or maple/corn/golden syrup. You can then recheck your levels after a quarter hour, if they havent risen sufficiently, eat more. In more advanced cases, the patient will need an injection of glucagon, a hormone which will stimulate glucose release from the liver. Outlook The best way of dealing with the problem is early detection. You can better control the condition

Alcoholic Ketoacidosisclinical Presentation

Alcoholic KetoacidosisClinical Presentation Author: George Ansstas, MD; Chief Editor: Romesh Khardori, MD, PhD, FACP more... Patients with alcoholic ketoacidosis (AKA) almost always are alcoholics who, prior to the development of ketoacidosis, have engaged in a period of very heavy drinking, with subsequent abrupt cessation of alcohol consumption 1-2 days before presentation. Such presentations typically result from physical complaints, such as the following: Nausea, vomiting, and abdominal pain (each found in 60-75% of patients) Dyspnea, tremulousness, and/or dizziness (10-20% each) Muscle pain, diarrhea, syncope, and seizure (1-8% each) These symptoms usually are attributed to alcoholic gastritis or pancreatitis. Example case of alcoholic ketoacidosis: A 35-year-old man who chronically abuses alcohol presents with abdominal pain and intractable emesis for the past 2 days. The pain and emesis developed after 5 days of heavy drinking. Since their onset, he stopped eating and drinking altogether. He complains of epigastric pain that radiates through to his back. He is afebrile, tachycardic, and borderline hypotensive. He is sleepy, but awakens easily to verbal stimuli. Generally, t Continue reading >>

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  1. kryptomancer

    Experienced fasters: How long does it take you to reach ketosis?

    It seems that it takes about 3 days for the average person to get into a state of ketosis when fasting.
    For those of you who are veterans of fasting, especially longer fasts (5+ days), how long does it take you to get into ketosis?
    I'm curious about this due to this guy going into ketosis in only 2 days on his second 5 day fast. Was wondering if it was due to the avocado or his body just adapting faster.

  2. Glarsie

    You should be in ketosis once your liver glycogen stores are depleted. In a fast this should occur in 24-48hrs after your last meal and will be influenced by your insulin sensitivity (which affects you basal insulin level) and the amount of glycogen in your liver (ie what you ate leading up to the fast). It's entirely possible (some would argue desirable) to be in ketosis before fasting. Nutritional ketosis generally starts when you have a betahydroxybuterate level greater than .5 mmol/L which can't be accurately measured through urine ketostix which only show excreted acetoacetate (not blood concentrations of BHB). You can turn those keto sticks dark purple just by restricting fluid intake and becoming slightly dehydrated or equally make them not register by drinking half a gallon of water.
    Ketosis is brought on by a lack of carbohydrates/insulin and not by the presence of fat.
    Eat under 20g of net carbs per day (don't count fibre) for 3 days and you will be in ketosis. You will probably be in ketosis sooner, but 3 days is pretty true for everyone.
    Remember that as you produce ketones you will start to use them as well as free fatty acids for fuel (less is excreted in urine and blood levels don't increase indefinitely). In the end ketones will be used primarily for your brain and other tissues not able to use FFA while glucose will be reserved for cells that can only use glucose (eg red blood cells and some parts of the brain). The breakdown of triglycerides into FFAs also releases glycerol that is converted into glucose to supply the now reduced demand for glucose. This doesn't happen as soon as you produce ketones but over a few days as your levels increase.
    Edit: nutritional ketosis defined as starting at .5 rather than .3 mmol/l

  3. kryptomancer

    this should occur in 24-48hrs after your last meal
    Actually in the link I posted it took 3 days initially for the guy to go into ketosis, then on the second time through it only took him 2. I'm very interested in to why this was as I am planning on doing a series of longer fasts and want to make it as easy as possible.
    and will be influenced by your insulin sensitivity
    So perhaps the reason why the 2nd fast was quicker to get into ketosis with due to increased insulin sensitivity from his initial fast?
    and the amount of glycogen in your liver (ie what you ate leading up to the fast).
    This is my current plan: eat under 20g carbs for two days, start water fasting on the third and lifting heavy squats and dead lifts; then taking apple cider vinegar before bed.

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