Escitalopram For Mood And Anxiety Disorders Cipralex
Escitalopram is prescribed as a once-daily dose. Try to take your doses at the same time of day each day. You can take escitalopram with or without food. Please tell your doctor if you feel that you are getting worse, or if you experience any troublesome side-effects. About escitalopram Type of medicine A selective serotonin reuptake inhibitor (SSRI) antidepressant Used for Depression, and anxiety disorders (such as panic disorder, social anxiety disorder, generalised anxiety disorder and obsessive-compulsive disorder) in adults Also called Cipralex® Available as Tablets, and oral liquid drops Escitalopram belongs to a group of medicines called SSRI antidepressants. It is prescribed for the treatment of some mood and anxiety disorders. These are depression, panic disorder, social anxiety disorder, generalised anxiety disorder and obsessive-compulsive disorder. Both depression and anxiety disorders can develop for no apparent reason, but they can also be triggered by a life event such as a relationship problem, a work-related problem, bereavement, or illness. Whatever the cause of the problem, when the symptoms are severe enough, they can interfere with normal day-to-day activities. When this happens, taking a medicine such as escitalopram can help to ease the symptoms and restore normal daily routines. Escitalopram works by regulating the level of a certain chemical in your brain, called serotonin. Before taking escitalopram Some medicines are not suitable for people with certain conditions, and sometimes a medicine can only be used if extra care is taken. For these reasons, before you start taking escitalopram it is important that your doctor knows: If you are pregnant, trying for a baby or breast-feeding. If you have any problems with the way your liver works, or any Continue reading >>
Neurotransmitter Repletion
Neurotransmitter Repletion Neurotransmitters are the naturally occurring chemicals inside your body that transmit messages between nerve cells. In the brain alone there are 183 different neurotransmitters. Two major neurotransmitters are serotonin and catecholamines, which includes norepinephrine, epinephrine, and dopamine. Although this is the focus of this webpage, sometimes additional neurotransmitters such as acetylcholine, histamine and GABA must be considered in a comprehensive successful program. For years it has been known in medicine that low levels of Serotonin and/or Norepinephrine can cause many diseases and illness. Some of the diseases and/or illnesses caused by or associated with low levels of Serotonin and/or Norepinephrine include: Depression/Moody Anxiety Panic Attacks Insomnia/Sleep disorders Premenstrual Tension Fibromyalgia Obesity Anorexia Bulimia “Hypoglycemia” Chronic pain states Migraines ADD/ADHD Restless Leg Syndrome In addition, over 60 diseases and illnesses may be caused by or associated with neurotransmitter deficiency. Low neurotransmitter levels is not only very common, it is epidemic. “How do the levels of serotonin and catecholamine neurotransmitters get to such critically low levels?” There are several explanations. The first is that neurotransmitter depletion is nutritionally based. Neurotransmitters are made from amino acids that must be obtained from protein in the diet. In addition, amino acids, vitamins and minerals eaten in food are required for the creation of the neurotransmitters. If the diet is deficient, neurotransmitter deficiency develops. There are multiple medications that have shown to cause depletion of serotonin and/or catecholamine in the urine. These are the medications prescribed to increase the activity o Continue reading >>
Intractable Fasting Hypoglycemia As A Manifestation Of Hepatocellular Carcinoma
Copyright © 2017 Justin J. Forde et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Abstract Non-islet cell tumor hypoglycemia (NICTH) is a rare and serious paraneoplastic complication of both malignant and benign tumors to consider when evaluating fasting hypoglycemia, especially in the setting of liver diseases. We present a case of NICTH in a 54-year-old male with hepatocellular carcinoma (HCC) who presented with symptomatic intractable hypoglycemia (IH) after bowel preparation and fasting for screening upper endoscopy and colonoscopy. 1. Introduction Hypoglycemia due to non-islet cell tumor hypoglycemia (NICTH) is an established paraneoplastic complication of hepatocellular carcinoma (HCC) that occurs in 4–27% of patients [1–3]. The actual prevalence and incidence are not known, likely due to a large number of cases that go unrecognized. Rarely, NICTH has been identified as the initial symptom of HCC in otherwise asymptomatic individuals [1, 4]. There are two types based on main etiologies for NICTH, types A and B. Type B is more common and is caused by overproduction of incompletely processed insulin-like growth factor 2 (IGF-2) by the tumor cells. Less common is type A, characterized by increased glucose demand from rapidly growing tumor, late in the disease process. Both types result in intractable fasting hypoglycemia and should be considered in patients with refractory hypoglycemia [1, 5–8]. NICTH occurs more commonly in patients with mesenchymal tumors, fibromas, carcinoid, myelomas, lymphomas, hepatocellular, and colorectal carcinomas [5, 9, 10]. 2. Case A 54-year-old male with long-s Continue reading >>
Lexapro Side Effects Center
Lexapro (escitalopram) is type of antidepressant called a selective serotonin reuptake inhibitor (SSRI) used to treat anxiety in adults and major depressive disorder in adults and adolescents who are at least 12 years old. Common side effects of Lexapro include: drowsiness, dizziness, sleep problems (insomnia), nausea, upset stomach, gas, constipation, weight changes, decreased sex drive, Lexapro is available in tablet form. The recommended adult dose of Lexapro is 10 mg once daily. Do not use escitalopram if you are using an MAO inhibitor such as isocarboxazid (Marplan), tranylcypromine (Parnate), phenelzine (Nardil), rasagiline (Azilect), or selegiline (Eldepryl, Emsam). SSRI antidepressants may cause serious or life-threatening lung problems in newborn babies whose mothers take the medication during pregnancy. However, you may have a relapse of depression if you stop taking your antidepressant during pregnancy. If you are planning a pregnancy, or if you become pregnant while taking escitalopram, do not stop taking the medication without first talking to your doctor. Escitalopram can pass into breast milk and may harm a nursing baby. Do not use this medication without telling your doctor if you are breastfeeding a baby. Our Lexapro Side Effects Drug Center provides a comprehensive view of available drug information on the potential side effects when taking this medication. This is not a complete list of side effects and others may occur. Call your doctor for medical advice about side effects. You may report side effects to FDA at 1-800-FDA-1088. Continue reading >>
Lexapro Side Effects By Likelihood And Severity
COMMON side effects If experienced, these tend to have a Less Severe expression Chronic Trouble Sleeping Constipation Diarrhea Dizzy Dry Mouth Excessive Sweating Feel Like Throwing Up Inability To Have An Erection Low Energy Problem With Ejaculation INFREQUENT side effects If experienced, these tend to have a Less Severe expression Flu-Like Symptoms Gas Indigestion Inflammation Of The Nose Loss Of Appetite Numbness And Tingling Sexual Problems Sinus Irritation And Congestion Stomach Cramps Toothache RARE side effects If experienced, these tend to have a Severe expression Abnormally Low Blood Pressure Acute Inflammation Of The Pancreas Aggressive Behavior Anemia Atrial Fibrillation Behaving With Excessive Cheerfulness And Activity Bleeding Bleeding Of The Stomach Or Intestines Blood Clot In A Deep Vein Of The Extremities Decreased Blood Platelets Decreased White Blood Cells Deficiency Of Granulocytes A Type Of White Blood Cell Extrapyramidal Reaction Giant Hives Hallucination Having Thoughts Of Suicide Heart Attack Hepatitis High Blood Pressure Inflammation Of The Lining Of The Stomach And Intestines Involuntary Quivering Kidney Failure Low Amount Of Sodium In The Blood Lung Embolism Neuroleptic Malignant Syndrome Prolonged QT Interval On EKG Psychosis Caused By A Drug Rapid Ventricular Heartbeat Rhabdomyolysis Secondary Angle-Closure Glaucoma Seizures Serotonin Syndrome - Adverse Drug Interaction Slow Heartbeat Stevens-Johnson Syndrome Syndrome Of Inappropriate Antidiuretic Hormone Secretion Toxic Epidermal Necrolysis Very Rapid Heartbeat - Torsades De Pointes If experienced, these tend to have a Less Severe expression Abnormal Manner Of Walking Altered Interest In Having Sexual Intercourse Blood Coming From Anus Blood Pressure Drop Upon Standing Blurred Vision Bronchit Continue reading >>
A Case Report On Escitalopram-induced Hyperglycaemia In A Diabetic Patient.
Abstract The incidence of depression in diabetic patients is quite high; moreover, it has been suggested that the presence of depression itself may increase the risk of diabetes mellitus. Hence, it follows that the simultaneous use of antidiabetic and antidepressant drugs is common. Some clinical evidence indicates that selective serotonin re-uptake inhibitors (SSRIs) could be very useful in treating overweight patients, both with and without diabetes. However, recent deregulation of glucidic metabolism was tested in diabetic subjects treated with antidepressants. Several cases of hyperglycaemia and hypoglycaemia associated with other SSRIs have been published, whereas only one case of escitalopram inducing hyperglycaemia has been noted. The exact mechanism of glucose control impairment in patients taking SSRIs--escitalopram in particular--still remains unclear. We describe a diabetic 83-year-old woman with good glycaemic control (as evinced by glycaemic and glycosylated haemoglobin assay--HbA1c--values) before escitalopram initiation in response to therapy with glibenclamide. Escitalopram resulted in a significantly increased glycaemia values 5 days following administration. Glycaemia values returned to normality only after suspension of escitalopram, despite antidiabetic dosage increase. We report this case to draw attention to escitalopram as a possible cause of glycaemic control loss. Continue reading >>
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Most of these supplements can be found on-line, at your local pharmacy or vitamin store, or through a number of supplement companies. We have included the link to a website for Xymogen, which is a supplement company that Dr. Jill Quigley and Eric Adrid, PA-C have reviewed and found to be a reliable source for supplements. Please check with your provider prior to starting supplements to make sure they will benefit you and will not interact with other medications or supplements you are taking. Your provider will inform you of the dose of each supplement that you should take. If you are interested in purchasing supplements online, please speak to your medical provider about how to create your online account. Alpha Lipoic Acid: Antioxidant, helps turn glucose into energy Can cause permanent hypothyroidism when dose greater than 1800mg/day is taken Beta-glucan: Fiber from cell walls; decreases hunger; also lowers LDL and A1c and increases HDL No known side effects Not available through Metagenics Chromium (Trivalent Chromium Picolinate): Improves the efficiency of insulin; improves fat loss with muscle sparing Can cause hypoglycemia so do not use if not overweight Green Tea Extract (Epigallocatechin Gallate (EGCG): Releases norepinephrine at the hypothalamus (like phentermine) Can cause abnormal liver function tests (AST and ALT) Requires a good supplement company to ensure extraction of ingredient Green Coffee Extract (Chlorogenic Acid): Increases production of norepinephrine at the hypothalamus (like phentermine) No serious side effects noted; side effects are those associated with caffeine Standardization and extraction is important, so make sure a good company makes it; should say chlorogenic acids, not green coffee extracts on ingredients 5-Hydroxytryptophan (5-HTP): Co Continue reading >>
Antidepressants Cause 40,000 Deaths A Year - But They're Handed Out Like Candy
My recent article on the dangers of psychiatric drugs ignited a firestorm of controversy. Part of the outrage may have been caused by some general misunderstandings, which I hope to clear up here. This is particularly important at this time as it is abundantly clear that suicide rates rise as the economy worsens. The image of people jumping from windows after the stock market crash of 1929 graphically illustrates this risk. Many may not agree, but I have been studying the economy for over 30 years now and it seems crystal clear to me that the economy has yet to hit bottom, and this will only serve to increase the risk of depression. My Personal Experiences with Depression First of all, I would like to set the record straight as many were confused about my personal experiences with depression. They believed I had none, and therefore there is no way I could understand this disease. Well let me tell you, nothing could be further from the truth. Mental and emotional problems exact an extreme toll on family units and in some cases extended circles of friends. I've personally been a witness to the struggles of two people near and dear to me who suffered from deep chronic depression for a number of years that actually resulted in multiple suicide attempts. Suicide is a common complication of depression, and is one of the primary reasons why it must be taken seriously as it can become a terminal illness. Many also might be unaware that I was a full-time practicing physician for over 20 years before I determined that I could help more people by committing myself full time to this newsletter and web site, than treating patients one on one. Before making that choice however, I treated tens of thousands of people for all sorts of problems, and I've seen my fair share of depressed p Continue reading >>
The Selective Serotonin Reuptake Inhibitor Sertraline Enhances Counterregulatory Responses To Hypoglycemia
Go to: Selective serotonin reuptake inhibitors (SSRIs) are widely prescribed for patients with comorbid diabetes and depression. Clinical case studies in diabetic patients, however, suggest that SSRI therapy may exacerbate hypoglycemia. We hypothesized that SSRIs might increase the risk of hypoglycemia by impairing hormonal counterregulatory responses (CRR). We evaluated the effect of the SSRI sertraline on hormonal CRR to single or recurrent hypoglycemia in nondiabetic rats. Since there are time-dependent effects of SSRIs on serotonin neurotransmission that correspond with therapeutic action, we evaluated the effect of 6- or 20-day sertraline treatment on hypoglycemia CRR. We found that 6-day sertraline (SERT) treatment specifically enhanced the epinephrine response to a single bout of hypoglycemia vs. vehicle (VEH)-treated rats (t = 120: VEH, 2,573 ± 448 vs. SERT, 4,202 ± 545 pg/ml, P < 0.05). In response to recurrent hypoglycemia, VEH-treated rats exhibited the expected impairment in epinephrine secretion (t = 60: 678 ± 73 pg/ml) vs. VEH-treated rats experiencing first-time hypoglycemia (t = 60: 2,081 ± 436 pg/ml, P < 0.01). SERT treatment prevented the impaired epinephrine response in recurrent hypoglycemic rats (t = 60: 1,794 ± 276 pgl/ml). In 20-day SERT-treated rats, epinephrine, norepinephrine, and glucagon CRR were all significantly elevated above VEH-treated controls in response to hypoglycemia. Similarly to 6-day SERT treatment, 20-day SERT treatment rescued the impaired epinephrine response in recurrent hypoglycemic rats. Our data demonstrate that neither 6- nor 20-day sertraline treatment impaired hormonal CRR to hypoglycemia in nondiabetic rats. Instead, sertraline treatment resulted in an enhancement of hypoglycemia CRR and prevented the impaired adr Continue reading >>
The Serotonin Connection
By Jurriaan Plesman, BA(Psych), Post Grad Dip Clin Nutr I will attempt to describe briefly The Serotonin Connection as asequence of psychological and biochemical events in the development of emotional disorders. Most of the statements below can be verified by scientific studies, but some are still controversial, especially the relationship between insulin resistance and absorption of amino acids. Much research needs to be done in this area. The events appear to follow a predetermined sequence as follows; 1) An extended period of physical or psychological stress, will produce stress hormones such as cortisol and adrenaline, that can interfere with the synthesis of the brain neurotransmitter, Serotonin. 2) A neurotransmitter is any one of numerous chemicals that occupy the gap (synapse) between two or more nerve cells (neurons) and thereby allows the triggering of a tiny electrical currents in adjacent cells. Each neurotransmitter fits into a unique receptor – like a key fitting into a lock – thus allowing messages to be carried along nerve pathways. See Figure 1 3) Serotonin is a neurotransmitter that conveys the positive sensations of satiety, satisfaction and relaxation. It regulates appetite and when converted to melatonin helps us to sleep. 4) A deficiency of Serotonin in the brain can cause endogenous depression, upsets the appetite mechanism and may lead to obesity or other eating disorders such as anorexia and bulimia nervosa and may be responsible for insomnia. Doctors usually prescribe Selective Serotonin Reuptake Inhibitors (SSRIs) which have the effects of increasing the amount Serotonin and thereby medically treat the above conditions. Unfortunately, SSRIs may have side effects in some patients, and generally do not address the underlying biochemical caus Continue reading >>
For Health And Quality Of Life
Cortisol Deficiency (a.k.a. hypocortisolism, adrenal fatigue, adrenal insufficiency) I had decided not to give patients labels and drugs, but only to prescribe natural hormones and nutrients, I thought it would be easy work--just replacing sex hormones that to correct age-related losses. From Dr. Rouzier's course I had learned about diagnosing hypothyroidism according to symptoms and relatively low FT4 and FT3 levels, and optimizing T3 levels/effects with natural desiccated thyroid (NDT). I helped many people so much that they referred their suffering friends and relatives to me. They had fatigue, brain fog, depression, anxiety, headaches, etc. and had gotten little help from the drugs prescribed by physicians. They were mostly women. I tried to help with ovarian hormone replacement when needed, and with NDT. Many of responded very well to NDT. Some, however, had no improvement at all. Some actually felt much worse, sometimes even after just one dose. Some immediately better on NDT, but later felt worse. Some of them also felt worse when I tried to correct their estradiol or DHEA deficiencies. NDT and other hormones worsened their underlying, long-standing symptoms. I understood that these negative reactions to physiological doses/levels of natural hormones were not “side effects”. They indicated that there was some other endocrine or metabolic disorder/deficiency that was being strongly affected by potent T4/T3 therapy, usually for the worse. The cause was not hard to find. All physicians are taught that thyroid replacement can worsen adrenal insufficiency; but they think that adrenal insufficiency is extremely rare—confined to persons with obvious disease or damage affecting their adrenal glands (Addison’s Disease) or their hypothalamic-pituitary system. They Continue reading >>
Antidepressants That Cause High Blood Sugar
High blood sugar--abnormal, even dangerous levels of sugar in the blood--is known as hyperglycemia and is most often associated with the disease diabetes mellitus. However, different types of medications may also cause high blood sugar; this is sometimes known as medication-induced, or drug-induced, diabetes. Within the list of drugs are several medications that are used to treat depression. While only one medication is officially considered an "antidepressant," certain antipsychotics--which may also be used in the treatment of depression--may cause hyperglycemia as well. Video of the Day Fluoxetine is a commonly-used antidepressant; it belongs to a class called the “serotonin-specific reuptake inhibitors,” and may also be known by one of its brand names, Prozac. It is used to treat both depression that occurs as a result of major depressive disorder and, in combination with the antipsychotic medication olanzepine, to treat the depression that may occur in patients with bipolar disorder. In addition to its effects on blood sugar levels, fluoxetine may Medline Plus reports that fluoxetine can cause nausea, lack of appetite, weight loss, nervousness, and changes in sex drive. The medical reference UpToDate reports that in patients with diabetes, fluoxetine affects blood sugar regulation in an interesting way. Patients who take fluoxetine may have more episodes of hypoglycemia, or low blood sugar--but when they stop taking fluoxetine, instead of their blood sugar reaching normal levels, patients’ blood sugar level goes above normal. This is called hyperglycemia, and this observation suggests that fluoxetine is somehow impacting the blood sugar regulation mechanism of patients with diabetes. Because of this, a patient who is taking medication for his diabetes and is a Continue reading >>
