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Ketosis Prone Diabetes Ppt

Director, Clinical Documentation Improvement Program

Director, Clinical Documentation Improvement Program

Adelaide M. La Rosa, RN, BSN, CCDS, AHIMA-Approved ICD-10-CM/PCS Trainer/Ambassador St. Francis Hospital Roslyn, NY ICD-10 … The Journey Continues Flow of Physician’s Documentation Converts into codes, ICD-9 now and ICD-10 Oct. 2013? Physician’s documentation Feds RAC Insurance companies HealthGrades LeapFrog “Tier status†and credentialing Internet Your patient Newsday Oversight and recovery Ranking and reimbursement Diabetes DM due to underlying condition Drug- or chemical-induced DM Type 1 DM Type 2 DM Other specified DM Manifestations Complication (Kidney) (Circulatory) (Retinopathy) (Neurological) 5 categories 5 Categories (E08) DM due to underlying condition (E09) Drug or Chemical Induced DM (E10) Type I DM (E11) Type II DM (E13) Other specified DM Diabetes Mellitus Type 1 diabetes mellitus (E10 – ICD-10-CM category) Also known as ketosis-prone, juvenile-type, juvenile-onset, or juvenile diabetes Condition usually develops before reaching puberty Failure to produce insulin at all or decrease in production Requires regular insulin injections to sustain life Diabetes Mellitus (cont.) Type 2 diabetes mellitus (E11 – ICD-10-CM category) Ketosis-resistant Insulin produced in insufficient quantity or the body does not use it adequately Usually does not require insulin Managed with oral hypoglycemic agents, exercise, and diet Temporarily may require insulin coverage to control patient’s blood glucose level during hospitalization (Note: Do not use long-term use of insulin if during encounter only) Secondary Diabetes Caused by: Underlying conditions (E08 – ICD-10-CM category) Congenital rubella Cushing’s syndrome Cystic fibrosis Malignant neoplasm Malnutrition Pancreatitis * Code underlying condition first Di Continue reading >>

The Fat Burning Kitchen - Foods That Burn Fat, Foods That Make You Fat

The Fat Burning Kitchen - Foods That Burn Fat, Foods That Make You Fat

Attention Men & Women Over Age 50Struggling To Lose Weight... Discover How The FoodsYou're Eating Every Day Are Making Your Fat Cells SICK... Making it IMPOSSIBLE to lose weight, while also damaging your joints, disrupting your hormones, rapidly aging your skin, and even leading to Diabetes. Which means if you've been struggling to lose weight, whether it's months, days, or even years, then please pay close attention to the article below because you're going to discover it's NOT your fault... Please do yourself a favor and take 2-minutes out of your busy day to read this very important health article... Also please make sure you read to the end, because there's a very special surprise, just for YOU, that I know you'll love... by Mike Geary, aka 'The Nutrition Watchdog' Certified Nutrition Specialist, Best-Selling Author How did you feel when you woke up this morning? Were you refreshed and able to jump out of bed or did it take a while to force yourself up and out of bed, after being rudely shaken out of your slumber by that stupid alarm clock buzz, buzz, buzzing in your ear? Did your body feel young and energetic as you got up, or... ...Were you achy and groggy from another restless nights sleep? When you looked yourself in the mirror, did you see a fresh, young face, or Did you stagger into the bathroom, ankles and knees creaking, hunched over from back pain and tightness, and see an old version of yourself staring back through the mirror? With bags under your eyes, dry & graying hair, wrinkled & dry skin? When you looked down as you got dressed did you see a fit, lean, and healthy body, or Did you see an ever-growing stomach, soft, flabby fat, and a body thats become weak, tired and soft? What goes through your mind as you look at yourself? Are you happy with the wa Continue reading >>

Ketosis Prone Type 2 Diabetes Ppt

Ketosis Prone Type 2 Diabetes Ppt

If IR is a determinant of susceptibility to pasture-associated nhs diabetes blood glucose monitoring laminitis then what triggers the lamini- enth.: Cetirizin-2HCl 10 mg. I found the hero character to be a frustrating one but perhaps that was part of komplikasi diabetes melitus dengan hipertensi the books type 2 diabetes exercises to avoid charm. Ketosis Prone Type 2 Diabetes Ppt to accomplished endurance athletes who are happy with their current strength-training regimen probably not though useful insights could be gleaned from the aforementioned section on the non-physical benefits and the Fuel for Fitness chapter including the training recipes. Question: What is the differences between all the different insulins? With so many different types of insulin products and delivery devices its hard to keep everything straight. diabetes yoga in tamil diabetes.nhs.uk/safe use of insulin/elearning course I need someone who has dedicated their LIFE to doing PSAs thats for sure: I likes this story right from the very start This article was written by Sarfraz Zaidi MD FACE This is the bodys reaction to the irritation and vomiting helps expel the irritants from the stomach NPH insulin has a peak effect 4-12 hours after diabetes foundation donate car injection and a duration of action of 18-26 hours Insulin should be injected into fat to do its job properly. My stylist has one; her advice was just to keep the wand moving dont stop it or itll burn your hair. The answer is usually that the ADA is a private organization and they may do as they choose. Introduction and need help Last Post by claras 2 Replies 124 Views. Having that Call off the wedding checklist as like the 5th page of the book (before you een understand what youre going through) will scare the pants of anyone who is em Continue reading >>

Ketosis-prone Diabetes

Ketosis-prone Diabetes

Does presenting with diabetic ketoacidosis (DKA) mandate indefinite insulin treatment? Not always. Since the mid-1990s, weve increasingly observed and recognized patients that dont neatly fit into either type 1 diabetes (T1DM) or T2DM. Ketosis-prone type 2 diabetes mellitus (KPDM) is underrecognized and distinctive. First described by Winter and colleagues in 1987, 12 African-American patients initially presented with DKA, but their disease course unfolded more like that of an individual with T2DM.1 KPDM was initially thought to be a variant of maturity onset diabetes of the young (MODY). Other names include Flatbush diabetes (named for the part of Brooklyn, NY where young African-Americans were described to have these clinical features of KPDM), type 1.5 diabetes, and atypical diabetes. 1. A large number of KPDM patients present without a previous diagnosis of DM and without a known precipitating cause for the DKA. >75% of KPDM patients fit this description. Most patients are African-American or Hispanic, overweight or obese, male (theres a two- to three-fold greater prevalence in men compared with women), in their 40s or 50s at the time of diagnosis. 2. If the patients insulin requirements rapidly decline in the first several weeks after presenting, think of possible KPDM. i. Patients test pre-meal glucose at least 2 times/day, and check in with their health care professional team every 2 weeks for the first 2 months after being discharged from the hospital to titrate insulin, and subsequently every 2 or 3 months, as extent of control warrants. ii.Clinicians begin tapering insulin by 25% at each visit, once fasting glucose declines below 130 mg/dL for 2 weeks, or if the patient develops hypoglycemia. 3. Many patients with KPDM will spontaneously remit. Most patients Continue reading >>

Acylation Stimulating Protein, Complement C3 And Lipid Metabolism In Ketosis-prone Diabetic Subjects

Acylation Stimulating Protein, Complement C3 And Lipid Metabolism In Ketosis-prone Diabetic Subjects

Acylation Stimulating Protein, Complement C3 and Lipid Metabolism in Ketosis-Prone Diabetic Subjects Affiliations Centre de Recherche de lInstitut Universitaire de Cardiologie & Pneumologie de Qubec, Universit Laval, Qubec, Canada, Department of Pediatrics, Tongji Hospital, HuaZhong University of Science and Technology, Wuhan, Hubei, P. R. China Affiliation Centre de Recherche de lInstitut Universitaire de Cardiologie & Pneumologie de Qubec, Universit Laval, Qubec, Canada Affiliation Centre de Recherche de lInstitut Universitaire de Cardiologie & Pneumologie de Qubec, Universit Laval, Qubec, Canada Affiliation Division of Endocrinology and Metabolism, Department of Medicine, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand Affiliation Division of Endocrinology and Metabolism, Department of Medicine, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand * E-mail: [email protected] Affiliation Centre de Recherche de lInstitut Universitaire de Cardiologie & Pneumologie de Qubec, Universit Laval, Qubec, Canada Acylation Stimulating Protein, Complement C3 and Lipid Metabolism in Ketosis-Prone Diabetic Subjects Ketosis-prone diabetes (KPDM) is new-onset diabetic ketoacidosis without precipitating factors in non-type 1 diabetic patients; after management, some are withdrawn from exogenous insulin, although determining factors remain unclear. Twenty KPDM patients and twelve type 1 diabetic patients (T1DM), evaluated at baseline, 12 and 24 months with/without insulin maintenance underwent a standardized mixed-meal tolerance test (MMTT) for 2 h. At baseline, triglyceride and C3 were higher during MMTT in KPDM vs. T1DM (p<0.0001) with no differences in non-esterified fatty acids (NEFA) while Acylation Stimulating Protein (ASP) tended Continue reading >>

Brookes Dissertation Deadline Clothing! Order Dissertation Proposal

Brookes Dissertation Deadline Clothing! Order Dissertation Proposal

Brookes dissertation deadline clothing! Order dissertation proposal You know i considered applying once but you have to write like an essay? fuck that i'm not in school! differenzenquotient berechnen beispiel essay Et merci, je vais essayer de deplacer tout ca et on va voir. La fermentation est sensible aux vibrations ou pas (Genre sous un escalier avec des gamins qui tapent des pieds au dessus)? better education system essay active assignments dumpster diving essay summary and response thesis statement for college research paper.. Essay about recycling benefits wildlife big government vs small government essays opinion essay writing ppt background woodstock documentary review essay, psychology essays on self-identity anastatica hierochuntica research papers one of your fondest childhood memories essay what is the main purpose of gage's essay literature review of research paper uk. research paper basics you tube write a good essay quickly usa. Pro assisted suicide essay essay on crime and deviance nuclear energy and its uses essay. dumpster diving essay summary and response essay about adventure tourism about myself essay 300 words dissertation sur les passions gf flammarion ferreira, albert barrow essay prendre de la hauteur sur un sujet de dissertation apush long essay andrew jackson shanmuganathan engineering college graduation day essay short essay on nature conservation officer fairy doll synthesis essay capital punishment argumentative essays people around us essay sujet de dissertation le roman essay on clean water in africa six characteristic of research paper the donkey gk chesterton essays congenital heart disease essay sandra nitz dissertations dissertation social media hospitality on writing the college application essay harry bauld epub, giotto di bondone Continue reading >>

Ketosis-prone Type 2 Diabetes

Ketosis-prone Type 2 Diabetes

The original schema for classifying diabetes mellitus (DM) consisted of 2 categories known as type 1 diabetes mellitus and type 2 diabetes mellitus . Type 1 diabetes was also known as insulin-dependent diabetes. Patients with this type of diabetes were considered prone to develop diabetic ketoacidosis (DKA) . Patients with type 1 diabetes were found to have an absolute insulin deficiency due to autoimmune destruction of pancreatic beta cells. Patients with type 2 diabetics, or noninsulin-dependent diabetes, were not considered to be at risk for DKA. Type 2 diabetes is strongly associated with obesity and a family history of diabetes. These patients have peripheral insulin resistance with initially normal or elevated circulating levels of endogenous insulin. Since the mid-1990s, the number of patients who presented with DKA but did not require long-term insulin therapy has increased. Many such patients had conditions that resembled traditionally defined type 2 diabetes, in that they were obese and often had a family history of diabetes. Subsequent to these observations, new ways to classify diabetes were devised. The system of classification that most accurately predicts the need for insulin treatment 12 months after presentation with DKA is known as the A system. This system classifies diabetics into 4 groups as follows: A+- - Autoantibodies present, cell function absent A++ - Autoantibodies present, cell function present A-- - Autoantibodies absent, cell function absent A-+ - Autoantibodies absent, cell function present The commonest ketosis-prone diabetes (KPD) subgroup in a longitudinal study was A-+ (54%), followed by A-- (20%) A+- (18%) and A++ (8%). [ 1 ] As noted above, in the A-+ subgroup of patients with KPD cell antibodies are absent and cell function is pres Continue reading >>

Atypical Ketosis-prone Diabetes

Atypical Ketosis-prone Diabetes

S. Ali Imran , MB BS FRCP FRCPC and Ehud Ur , MB BS FRCP Dr Imran is an Associate Professor and Dr Ur is a Professor in the Division of Endocrinology and Metabolism at Dalhousie University in Halifax, NS Correspondence: Dr S.A. Imran, Division of Endocrinology and Metabolism, Dalhousie University, 7th Floor, North Victoria Building, VG Site, 1278 Tower Rd, Halifax, NS B3H 2Y9; telephone 902 473-8277; fax 902 473-3726; e-mail [email protected] Copyright the College of Family Physicians of Canada This article has been cited by other articles in PMC. Atypical diabetes is a rare form of diabetes mellitus (DM) that presents with diabetic ketoacidosis (DKA). However, in contrast to type 1 DM, patients with atypical DM undergo spontaneous remission and maintain long-term insulin independence. Family physicians must maintain a high index of suspicion to diagnose and manage such cases. A 44-year-old, previously healthy South Asian woman presented to her family physician with progressively worsening dry mouth, polyuria, and polydipsia for 6 weeks. She had also lost 7 kg in weight over the past 3 months. At the time of initial presentation, her fasting glucose was 18.1 mmol/L and her hemoglobin A1c was 13.4% (normal 4.5% to 6.5%). Results of the urinalysis were positive for glucose (> 55 mmol/L) and ketones (> 7.8 mmol/L). The family physician made a clinical diagnosis of type 1 DM and referred her to the local diabetes management centre. She was seen the same day at the diabetes centre and started on intensive insulin therapy, with multiple daily injections, after consultation with the endocrinologist. Upon presentation her weight was 63 kg with a calculated body mass index of 23.4 kg/m2. She had no family history of DM, and test results were negative for anti-islet cel Continue reading >>

Barker's Type 2 Ketosis Prone Diabetes / Atypical Diabetes T1b/ Flatbush Diabetes

Barker's Type 2 Ketosis Prone Diabetes / Atypical Diabetes T1b/ Flatbush Diabetes

Barker's Type 2 Ketosis Prone Diabetes / Atypical Diabetes T1b/ Flatbush Diabetes The accepted knowledge is that Diabetes destroys gradually over years. Ketosis Prone Type 2 diabetes is an acute form of type 2. This type 2 can reach fasting blood sugars of 300 or higher in months. This blog brings together all the documentation that I could find in the world and my speculation of what it means for KPDs in specific and diabetics in general. I ask you to leave your stories about what happened to you so that we can all gain a better understanding of what we are dealing with. Downloadable Scientific Ketosis Prone Type 2 Diabetes powerpoint presentation Guillermo E. Umpierrez, Dawn Smiley, and Abbas E. Kitabchi There is no news for you KPD fans in this presentation. This was done in 2006. This blog has a lot more current information and speculation on that information. This, however, represents the best thoughts of the current researchers at that time and these people have undisputed credentials. Whether people have heard of Ketosis Prone Type 2 diabetes or disbelieve its existence becomes moot because of this document. Here you have a complete presentation telling all about it. I post this in that light. You can simply download it and give it to family and friends. I found this presentation on the web and these are some of the chief writers and researchers of Ketosis Prone Type 2 diabetes. It seems to be a presentation in China and might be a bit dated but it is very thorough. I spend a good bit of time introducing people to this subject and so I started to develop a presentation on it. Low and behold, this pops up! You no longer have to go through the long discussions. This power point will do nicely. This would be a good thing to pass around, post or show to your diabete Continue reading >>

Frequently Asked Questions & Answers For Ketosis-prone Diabetes Mellitus, What Is Ketosis-prone Diabetes Mellitus? | Lybrate - Page 970

Frequently Asked Questions & Answers For Ketosis-prone Diabetes Mellitus, What Is Ketosis-prone Diabetes Mellitus? | Lybrate - Page 970

MBBS, DNB (General Medicine), Certified in Evidence Based Diabetes Management, MNAMS, MRCP (UK) Thank you for the query.As per the history you have provided, you are hypertensive (BP 130/90 mm of Hg, on Tenormin 25 and Stamlo 5 mg), have hyperlipidemia (raised cholesterol) and non-diabetic. It would be helpful, if you would have posted the reprots of the lipid profile. Itis advisable to maintain your Total cholesterol <200 mg%, LDL <120 mg% and HDL >45 mg%. The Triglyceride level should be <150 mg%. Since you have limited walking capacity, you need to modify on the diet. Reduce the intake of saturated fat (ghee, butter etc) and refined carbohydrate. The Statin (Atorvastatin, Rosuvastatin) group of medications is effective in reducing cholesterol, but should be taken only after consulting your physician.Regards. Mr. lybrate-user, Creatinine and protein excretion increase occurs when kidney function is affected. In long standing diabetes, filtering membrane of the nephron is damaged leading to proteinuria and also increase in creatinine. Consistent excellent blood glucose control over the years is needed to prevent damage to kidney. Apart from strict blood glucose control, protein intake also has to be reduced to 0.6 g per kg per day. Reduce excess physical activity, plus avoid foods more in creatinine. BP should be maintained at normal level. TSH 8.6 mU/L is high, but still falls in sub-clinical stage, so no treatment is advised. You should avoid soy beans, soy containing products, radish, cabbage, cauliflower, broccoli, mustard, peanuts and coffee. But it very important to maintain a very strict blood glucose control like FBS closer to 100 mg, PP 170 to 180 mg & HbA1c%<7. Thanks. Continue reading >>

Syndromes Of Ketosis-prone Diabetes Mellitus

Syndromes Of Ketosis-prone Diabetes Mellitus

Syndromes of Ketosis-Prone Diabetes Mellitus Ashok Balasubramanyam , Ramaswami Nalini , Christiane S. Hampe , and Mario Maldonado Translational Metabolism Unit (A.B., R.N., M.M.), Division of Diabetes, Endocrinology and Metabolism, Baylor College of Medicine, Houston, Texas 77030; Endocrine Service (A.B., R.N.), Ben Taub General Hospital, Houston, Texas 77030; Robert H. Williams Laboratory (C.S.H.), University of Washington, Seattle, Washington 98195; and Novartis, Inc. (M.M.), CH-4002 Basel, Switzerland Address all correspondence and requests for reprints to: Ashok Balasubramanyam, M.D., Translational Metabolism Unit, Division of Diabetes, Endocrinology and Metabolism, Baylor College of Medicine, Room 700B, One Baylor Plaza, Houston, Texas 77030. E-mail: [email protected] Received 2007 Aug 13; Accepted 2008 Jan 9. Copyright 2008 by The Endocrine Society This article has been cited by other articles in PMC. Ketosis-prone diabetes (KPD) is a widespread, emerging, heterogeneous syndrome characterized by patients who present with diabetic ketoacidosis or unprovoked ketosis but do not necessarily have the typical phenotype of autoimmune type 1 diabetes. Multiple, severe forms of -cell dysfunction appear to underlie the pathophysiology of KPD. Until recently, the syndrome has lacked an accurate, clinically relevant and etiologically useful classification scheme. We have utilized a large, longitudinally followed, heterogeneous, multiethnic cohort of KPD patients to identify four clinically and pathophysiologically distinct subgroups that are separable by the presence or absence of -cell autoimmunity and the presence or absence of -cell functional reserve. The resulting A classification system of KPD has proven to be highly accurate and predictive of such clinically importan Continue reading >>

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