Untangling One Cause Of Prediabetes
Endocrinology Researcher, University of ColoradoDenver School of Medicine, Aurora, Colo. The biggest risk factor for type 2 diabetes is not obesity. Nor is it genetics, gender, race, or diet. It's prediabetes , a collection of conditions that show up as high blood glucose levels. In America alone, 79 million peoplea quarter of the entire populationhave prediabetes. For doctors and public health experts, it's a terrifying number. "Not everyone who has prediabetes gets to [type 2] diabetes, but 50 to 70 percent of them will," says Leigh Perreault, a researcher at the University of ColoradoDenver School of Medicine. Yet recent research has shown that not all prediabetes is created equal. Though the resultshigh blood glucose and insulin resistance look similar, the underlying causes are different. About a third of Americans with prediabetes, or about 27 million people, have impaired fasting glucose, something the American Diabetes Association (ADA) recognized as a prediabetic condition in 2004. This is how impaired fasting glucose works: Between meals, the body normally produces and stores a certain amount of insulin. After the next meal, the insulin is called upon to turn the glucose into energy to fuel the body's cells. When we eat, those insulin reserves are released and used up. If the stored insulin isn't enough to get all the sugar in the bloodstream absorbed, the body senses it and makes morelike a thermostat automatically turning on the air-conditioning when the house gets too hot. But for people with impaired fasting glucose, "that's exactly what's not happening," Perreault says. "Something's wrong with the glucose thermostat." For people with this kind of prediabetes, the signals that tell the body to make more insulin when its stores run dry don't turn on until Continue reading >>
2018 Icd-10-cm Diagnosis Code R73.01
R00-R99 Symptoms, signs and abnormal clinical and laboratory findings, not elsewhere classified R70-R79 Abnormal findings on examination of blood, without diagnosis R73.01 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes. The 2018 edition of ICD-10-CM R73.01 became effective on October 1, 2017. This is the American ICD-10-CM version of R73.01 - other international versions of ICD-10 R73.01 may differ. The following code(s) above R73.01 contain annotation back-references In this context, annotation back-references refer to codes that contain: Symptoms, signs and abnormal clinical and laboratory findings, not elsewhere classified This chapter includes symptoms, signs, abnormal results of clinical or other investigative procedures, and ill-defined conditions regarding which no diagnosis classifiable elsewhere is recorded. Signs and symptoms that point rather definitely to a given diagnosis have been assigned to a category in other chapters of the classification. In general, categories in this chapter include the less well-defined conditions and symptoms that, without the necessary study of the case to establish a final diagnosis, point perhaps equally to two or more diseases or to two or more systems of the body. Practically all categories in the chapter could be designated 'not otherwise specified', 'unknown etiology' or 'transient'. The Alphabetical Index should be consulted to determine which symptoms and signs are to be allocated here and which to other chapters. The residual subcategories, numbered .8, are generally provided for other relevant symptoms that cannot be allocated elsewhere in the classification. The conditions and signs or symptoms included in categories R00 - R94 consist of: (a) cases for which n Continue reading >>
Diet For Impaired Fasting Glucose
M. Gideon Hoyle is a writer living outside of Houston. Previously, he produced brochures and a wide variety of other materials for a nonprofit educational foundation. He now specializes in topics related to health, exercise and nutrition, publishing for various websites. Fruits and vegetables are important parts of an impaired fasting glucose diet. A fasting glucose test is a procedure designed to measure your blood levels of glucose after you go without eating for at least eight hours. Moderately elevated results from this test indicate the presence of a condition called impaired fasting glucose, which greatly increases your risk for developing diabetes. You can help reverse an impaired glucose finding by making certain changes to your diet. If your doctor suspects that you have dangerously high blood glucose levels, he can order a fasting glucose test to help definitively diagnose your condition, according to the National Diabetes Information Clearinghouse. Typically, you will fast overnight and undergo testing during the morning. If a fasting glucose test reveals levels of glucose between 100 and 125mg per deciliter of blood, or mg/dL, you have impaired fasting glucose. The condition is also commonly known as pre-diabetes. You can help prevent pre-diabetes from progressing to full-blown diabetes by altering several aspects of your everyday lifestyle, the American Academy of Family Physicians reports at FamilyDoctor.org. With respect to your diet, recommended changes to help lower your glucose levels include substituting whole grains for processed products that contain white flour; increasing your intake of vegetables, fruits, poultry, fish and beans; and restricting your intake of sugar and other sweeteners such as molasses and honey. In addition to these general gu Continue reading >>
- Pre-diabetes Impaired Glucose Tolerance
- Antidepressant Medication as a Risk Factor for Type 2 Diabetes and Impaired Glucose Regulation
- Postprandial Blood Glucose Is a Stronger Predictor of Cardiovascular Events Than Fasting Blood Glucose in Type 2 Diabetes Mellitus, Particularly in Women: Lessons from the San Luigi Gonzaga Diabetes Study
Impaired Fasting Glucose
Impaired fasting glucose, or Impaired Fasting Glycemia (IFG) is a type of prediabetes, in which a person's blood sugar levels during fasting are consistently above the normal range, but below the diagnostic cut-off for a formal diagnosis of diabetes mellitus.[1] Together with impaired glucose tolerance, it is a sign of insulin resistance. In this manner, it is also one of the conditions associated with Metabolic Syndrome. Those with impaired fasting glucose are at an increased risk of vascular complications of diabetes, though to a lesser extent. The risks are cumulative, with both higher blood glucose levels, and the total amount of time it spends elevated, increasing the overall complication rate. IFG can eventually progress to type 2 diabetes mellitus without intervention, which typically involves lifestyle modification. Those with impaired fasting glucose have a 1.5 fold increased risk of developing clinical diabetes within 10 years, when compared to the general population. Some studies suggest that without lifestyle changes, IFG will progress to clinically diagnosable diabetes in just under 3 years, on average.[2] Impaired fasting glucose is often, though not always, associated with impaired glucose tolerance, though it may occur in isolation, with such persons having a normal response to a glucose tolerance test. Signs and Symptoms[edit] Impaired fasting glucose is often without any signs or symptoms, other than higher than normal glucose levels being detected in an individual's fasting blood sample. There may be signs and symptoms associated with elevated blood glucose, though these are likely to be minor, with significant symptoms suggestive of complete progression to type 2 diabetes. Such symptoms include:[3] Increased thirst Increased urination, especially wak Continue reading >>
- Pre-diabetes Impaired Glucose Tolerance
- Antidepressant Medication as a Risk Factor for Type 2 Diabetes and Impaired Glucose Regulation
- Postprandial Blood Glucose Is a Stronger Predictor of Cardiovascular Events Than Fasting Blood Glucose in Type 2 Diabetes Mellitus, Particularly in Women: Lessons from the San Luigi Gonzaga Diabetes Study
Impaired Glucose Tolerance Is A Risk Factor For Cardiovascular Disease, But Not Impaired Fasting Glucose. The Funagata Diabetes Study.
Impaired glucose tolerance is a risk factor for cardiovascular disease, but not impaired fasting glucose. The Funagata Diabetes Study. Department of Laboratory Medicine, Yamagata University School of Medicine, Japan. [email protected] To determine whether the new category of impaired fasting glucose (IFG) recently proposed by the Expert Committee of the American Diabetes Association is a risk factor for cardiovascular disease. Death certificates and residence transfer documents from the cohort population consisting of participants of the diabetes prevalence study in Funagata, Yamagata prefecture, Japan, 1990-1992, were analyzed up through the end of 1996. First, the cohort population was classified into three groups: normal glucose tolerance (NGT) (n = 2,016), impaired glucose tolerance (IGT) (n = 382), and diabetic (n = 253). Then the same population was reclassified into normal fasting glucose (NFG), IFG, and diabetic. The cumulative survival rates among the groups were compared using the classical life-table method, and age-adjusted analyses, the person-year method, and Cox's proportional hazard model were adopted. At the end of seven observed years, the cumulative survival rates from cardiovascular disease of IGT and diabetes were 0.962 and 0.954, respectively, both significantly lower than that of NGT (0.988). The Cox's proportional hazard model analysis showed that the hazard ratio of IGT to NGT on death from cardiovascular disease was 2.219 (95% CI 1.076-4.577). However, the cumulative survival rate of IFG from cardiovascular disease was 0.977, not significantly lower than that of NFG (0.985). The Cox's hazard ratio of IFG to NFG on death from cardiovascular disease was 1.136 (0.345-3.734), which was not significant either. Continue reading >>
- Antidepressant Medication as a Risk Factor for Type 2 Diabetes and Impaired Glucose Regulation
- Postprandial Blood Glucose Is a Stronger Predictor of Cardiovascular Events Than Fasting Blood Glucose in Type 2 Diabetes Mellitus, Particularly in Women: Lessons from the San Luigi Gonzaga Diabetes Study
- Whole fruits protect against diabetes, but juice is risk factor, say researchers
Impaired Fasting Glucose And Impaired Glucose Tolerance: Implications For Care
Type 2 diabetes is now epidemic. In the U.S., there has been a 61% increase in incidence between 1990 and 2001.[ 1 ] There are currently 1.5 million new cases per year, and the prevalence in 2005 was almost 21 million.[ 2 ] The epidemic has affected developed and developing countries alike, and the worldwide prevalence of diabetes is projected to increase dramatically by 2025.[ 3 ] The increase in type 2 diabetes is related to lifestyle changes that have resulted in overweight, obesity, and decreased physical activity levels. These environmental changes, superimposed on genetic predisposition, increase insulin resistance, which, in concert with progressive -cell failure, results in rising glycemia in the nondiabetic range. In addition to the risk for diabetes, insulin resistance and impaired insulin secretion are accompanied by a host of major cardiovascular disease (CVD) risk factors including hypertension and dyslipidemia. Further reduction in insulin secretion over time results in increasing glycemia and the development of diabetes, which in turn is associated with the development of microvascular and cardiovascular complications. The transition from the early metabolic abnormalities that precede diabetes, impaired fasting glucose (IFG) and impaired glucose tolerance (IGT), to diabetes may take many years; however, current estimates indicate that most individuals (perhaps up to 70%) with these pre-diabetic states eventually develop diabetes.[ 410 ] During the pre-diabetic state, the risk of a CVD event is modestly increased.[ 1122 ] With the development of diabetes, however, there is a large increase in risk for CVD, as well as for long-term complications affecting the eyes, kidneys, and nervous system. The complications of diabetes, which are the cause of major mor Continue reading >>
Diagnosing Impaired Glucose Tolerance (igt)
People with IGT have blood glucose levels that are higher than normal but not high enough to say they have diabetes. This condition is diagnosed using the oral glucose tolerance test (OGTT). After a fast of 8 to12 hours, a person's blood glucose is measured before and 2 hours after drinking a glucose-containing solution. In normal glucose tolerance, blood glucose rises no higher than 140 mg/dl 2 hours after the drink. In impaired glucose tolerance (IGT), the 2-hour blood glucose is between 140 and 199 mg/dl. If the 2-hour blood glucose rises to 200 mg/dl or above, a person has diabetes. How does the fasting blood glucose test differ from the oral glucose tolerance test? In the fasting blood glucose test, a person's blood glucose is measured after a fast of 8 to 12 hours: A person with normal blood glucose has a blood glucose level below 100. A person with impaired fasting glucose has a blood glucose level between 100 and 125 mg/dl. If the fasting blood glucose level rises to126 mg/dl or above, a person has diabetes. The OGTT includes measures of blood glucose levels after a fast and after a glucose challenge. In 1997, an American Diabetes Association (ADA) expert panel recommended that doctors use the fasting blood glucose test to screen their patients for diabetes because the test is easier and less costly than the OGTT. Though the fasting glucose test detects most diabetes cases, the OGTT is more sensitive in identifying people with blood glucose problems that may first appear only after a glucose challenge. For a person with IGT, what is the risk of developing type 2 diabetes? As few as 1 to as many as 10 of every 100 persons with IGT will develop diabetes per year. The risk of getting diabetes rises as people become more overweight and more sedentary, have a stronge Continue reading >>
A Prospective Study Of Impaired Fasting Glucose And Type 2 Diabetes In China: The Kailuan Study
The worldwide prevalence and incidence of chronic noncommunicable diseases, particularly diabetes and obesity, are increasing in pandemic proportions. [1] South and southeast Asian countries are predicted to represent a disproportionately large burden of the global incidence of diabetes. This is particularly relevant for China, where an extremely large population is growing, aging, and urbanizing. The epidemiologic forecast predicts that the prevalence of diabetes in China will double between 2000 and 2030. [1,2] Perhaps more alarming is the fact that the prevalence of prediabetes cases is expected to be several fold higher than the number of diabetes cases, thereby dramatically magnifying the pool of at-risk individuals. [3] The ramifications of uncontrolled increases in adiposity and consequent increases in the incidence of impaired fasting glucose (IFG) and diabetes in China are expected to be disastrous: higher body mass index (BMI) and impaired glycemic control contribute to micro- and macrovascular complications [4] and predict mortality. [5,6] Addressing the epidemic of diabetes and diabetes-associated complications has been identified as a public policy priority for China to reduce death, disability, and improve quality of life measures. [7] A failure to address these outcomes may have immeasurable effects on China's domestic economy, and global economies, by influencing human health and healthcare allocations. Fortunately, interventions to modify risk factors that result in impaired glycemic control and diabetes have shown the potential for success by mitigating the incidence of diabetes in the United States [8] and in China. [9] In order for large-scale public health initiatives in China to better identify populations at risk for developing IFG and diabetes, Continue reading >>
- Improved pregnancy outcomes in women with type 1 and type 2 diabetes but substantial clinic-to-clinic variations: a prospective nationwide study
- The UK Prospective Diabetes Study (UKPDS): clinical and therapeutic implications for type 2 diabetes
- Postprandial Blood Glucose Is a Stronger Predictor of Cardiovascular Events Than Fasting Blood Glucose in Type 2 Diabetes Mellitus, Particularly in Women: Lessons from the San Luigi Gonzaga Diabetes Study
Impaired Fasting Glycaemia
This factsheet is for people whose blood test has shown a marginally high fasting glucose (blood sugar) level or who would like information about impaired fasting glycaemia. Impaired fasting glycaemia (IFG) means that your body isn't able to regulate glucose as efficiently as it should. About impaired fasting glycaemia Complications Causes Diagnosis Treatment Further information Sources Related topics About impaired fasting glycaemia Glucose enters the blood from your intestines, where it's absorbed from food and drinks as a natural part of digestion. When glucose reaches your body tissues, such as muscle, it's made available to cells where it is needed for energy by the hormone insulin. Insulin regulates the level of glucose in the blood so that it doesn't go too high or too low. What is the normal range for blood glucose? Blood glucose concentrations change throughout the day. They are typically higher after eating and lower during fasting. A fasting blood glucose level lower than 6mmol/l is normal. A fasting blood glucose of 7mmol/l or higher may indicate diabetes. Diabetes is a long-term condition where the body is not able to control the amount of glucose in the blood. If your fasting blood glucose level is below 7mmol/l but above 6mmol/l you may have IFG or impaired glucose tolerance (IGT). IFG and IGT are not diabetes but suggest that your body is not converting glucose to energy as efficiently as it should. For the range of blood glucose concentrations associated with IFG, see Diagnosis. Complications Some people with IFG will go on to develop type 2 diabetes. Others may develop diabetes only if they don't take steps to reduce the chances of this happening (see Self-help). Diabetes is 10 to 20 times more likely in those with IGT or IFG. Since IFG can be an early Continue reading >>
- Advice to walk after meals is more effective for lowering postprandial glycaemia in type 2 diabetes mellitus than advice that does not specify timing: a randomised crossover study
- Pre-diabetes Impaired Glucose Tolerance
- Antidepressant Medication as a Risk Factor for Type 2 Diabetes and Impaired Glucose Regulation
Contributions Of Beta-cell Dysfunction And Insulin Resistance To The Pathogenesisof Impaired Glucose Tolerance And Impaired Fasting Glucose.
Contributions of beta-cell dysfunction and insulin resistance to the pathogenesisof impaired glucose tolerance and impaired fasting glucose. Abdul-Ghani MA(1), Tripathy D, DeFronzo RA. (1)Division of Diabetes, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr., San Antonio, TX 78229, USA. [email protected] Impaired glucose tolerance (IGT) and impaired fasting glucose (IFG) areintermediate states in glucose metabolism that exist between normal glucosetolerance and overt diabetes. Epidemiological studies demonstrate that the twocategories describe distinct populations with only partial overlap, suggestingthat different metabolic abnormalities characterize IGT and IFG. Insulinresistance and impaired beta-cell function, the primary defects observed in type 2 diabetes, both can be detected in subjects with IGT and IFG. However, clinical studies suggest that the site of insulin resistance varies between the twodisorders. While subjects with IGT have marked muscle insulin resistance withonly mild hepatic insulin resistance, subjects with IFG have severe hepaticinsulin resistance with normal or near-normal muscle insulin sensitivity. BothIFG and IGT are characterized by a reduction in early-phase insulin secretion,while subjects with IGT also have impaired late-phase insulin secretion. Thedistinct metabolic features present in subjects with IFG and IGT may requiredifferent therapeutic interventions to prevent their progression to type 2diabetes. Continue reading >>
The Prevalence And Determinants Of Impaired Fasting Glucose In The Population Of Taiwan
The prevalence and determinants of impaired fasting glucose in the population of Taiwan Chen and Yeh; licensee BioMed Central Ltd.2013 A current prevalence and relevant risk factors for impaired fasting glucose (IFG) have been reported by various ethnic groups and countries. By contrast, nationwide data for the incidence of IFG in Taiwan have not been presented in the past 15years. The aim for this manuscript was to estimate the prevalence of IFG and associated risk factors in the population of Taiwan. For this cross-sectional research, we used a nationally representative sample (N = 2604) obtained from the 20052008 Nutrition and Health Survey in Taiwan (NAHSIT), and adopted a stratified multistage sampling design. The tools employed for data collection included questionnaire interviews, anthropometry measurements, and laboratory analysis. The prevalence of IFG among adults in Taiwan is 35.8% (a fasting glucose level between 100 and 125mg/dl is considered abnormal). An estimated number of people with IFG is 6.5 million. A higher prevalence of IFG is observed in men younger than 65years compared to women. However, this trend is reversed for the elderly population. The factors significantly associated with IFG include the following: sex, age, overweight (27 > BMI 24), obesity (BMI 27), waist circumference (men 90cm, women 80cm), hypertension, and hyperlipidemia. IFG among adults in Taiwan is a health concern that requires attention. We recommend targeting the younger population, especially overweight and obese men between the ages of 19 and 40years, to provide applicable healthy lifestyle counseling and services. Furthermore, appropriate screening of elderly people is required to detect undiagnosed IFG cases and provide early intervention and treatment. Impaired fasting Continue reading >>
Diagnosis, Prognosis, And Treatment Of Impaired Glucose Tolerance And Impaired Fasting Glucose: Summary - Ahrq Evidence Report Summaries - Ncbi Bookshelf
NCBI Bookshelf. A service of the National Library of Medicine, National Institutes of Health. AHRQ Evidence Report Summaries. Rockville (MD): Agency for Healthcare Research and Quality (US); 1998-2005. This publication is provided for historical reference only and the information may be out of date. This publication is provided for historical reference only and the information may be out of date. 128Diagnosis, Prognosis, and Treatment of Impaired Glucose Tolerance and Impaired Fasting Glucose: Summary PL Santaguida, C Balion, D Hunt, K Morrison, H Gerstein, P Raina, L Booker, and H Yazdi. Diabetes mellitus (DM) and its associated disease outcomes are a growing concern worldwide. The current global prevalence of DM for all ages has been estimated at 2.8 percent and is predicted to reach 4.4 percent by 2030. 1 There is intense interest in identifying and treating risk factors that may prevent the onset of this disease and minimize morbidity. Impaired fasting glucose (IFG) and impaired glucose tolerance (IGT) are the intermediate metabolic states between normal and diabetic glucose homeostasis. These conditions are thought to be the precursors of DM, but the progression to overt disease is not straight-forward. The risk for both macrovascular and microvascular complications increases across the distribution of blood glucose concentrations well below the overt DM, and the risk is more strongly associated with post-challenge hyperglycemia than fasting glucose levels. However, it is unclear whether this glucose effect is independent of classical risk factors, such as blood pressure and lipids, or occurs due to abnormalities of other metabolites, such as free fatty acids. The goal of this systematic review is to evaluate the state of the evidence in the areas of the diagnosis Continue reading >>
How Can We Keep Impaired Glucose Tolerance And Impaired Fasting Glucose From Progressing To Diabetes?
How can we keep impaired glucose tolerance and impaired fasting glucose from progressing to diabetes? J Fam Pract. 2010 September;59(9):532-533 1. Orozco LJ, Buchleitner AM, Gimenez-Perez G, et al. Exercise or exercise and diet for preventing type 2 diabetes mellitus. Cochrane Database Syst Rev. 2008;3:CD003054.- 2. Gillies CL, Abrams KR, Lambert PC, et al. Pharmacological and lifestyle interventions to prevent or delay type 2 diabetes in people with impaired glucose tolerance: systematic review and meta-analysis. BMJ. 2007;334:299.- 3. Van de Laar FA, Lucassen PL, Akkermans RP, et al. Alpha-glucosidase inhibitors for people with impaired glucose tolerance or impaired fasting blood glucose. Cochrane Database Syst Rev. 2006;4:CD005061.- 4. Gerstein HC, Yusuf S, Bosch J, et al. DREAM (Diabetes Reduction Assessment with ramipril and rosiglitazone Medication) Trial Investigators Effect of rosiglitazone on the frequency of diabetes in patients with impaired glucose tolerance or impaired fasting glucose: a randomised controlled trial. Lancet. 2006;368:1096-1105. 5. Lago RM, Singh PP, Nesto RW. Congestive heart failure and cardiovascular death in patients with prediabetes and type 2 diabetes given thiazolidinediones: a meta-analysis of randomised clinical trials. Lancet. 2007;370:1129-1136. 6. Abuissa H, Jones PG, Marso SP, et al. Angiotensin-converting enzyme inhibitors or angiotensin receptor blockers for prevention of type 2 diabetes: a meta-analysis of randomized clinical trials. J Am Coll Cardiol. 2005;46:821-826. 7. Bosch J, Yusuf S, Gerstein HC. DREAM Trial Investigators Effect of ramipril on the incidence of diabetes. N Engl J Med. 2006;355:1551-1562. 8. American Diabetes Association. Standards of medical care in diabetes2009. Diabetes Care. 2009;32:S13-S61. LIFESTYLE Continue reading >>
- Pre-diabetes Impaired Glucose Tolerance
- Antidepressant Medication as a Risk Factor for Type 2 Diabetes and Impaired Glucose Regulation
- Postprandial Blood Glucose Is a Stronger Predictor of Cardiovascular Events Than Fasting Blood Glucose in Type 2 Diabetes Mellitus, Particularly in Women: Lessons from the San Luigi Gonzaga Diabetes Study
Prevalence And Control Of Diabetes And Impaired Fasting Glucose In New York City.
Prevalence and control of diabetes and impaired fasting glucose in New York City. New York City Department of Health and Mental Hygiene, New York, New York, USA. [email protected] Diabetes Care. 2009 Jan;32(1):57-62. doi: 10.2337/dc08-0727. OBJECTIVE: To determine the prevalence of diabetes and impaired fasting glucose (IFG) and to assess clinical management indicators among adults with diabetes in a representative sample of New York City adults. RESEARCH DESIGN AND METHODS: In 2004, New York City implemented the first community-level Health and Nutrition Examination Survey (NYC HANES), modeled after the National Health and Nutrition Examination Survey (NHANES). We used an interview to determine previously diagnosed diabetes and measured fasting plasma glucose to determine undiagnosed diabetes and IFG in a probability sample of 1,336 New York City adults. We assessed glycemic control and other clinical indicators using standardized NHANES protocols. RESULTS: The prevalence of diabetes among New York City adults was 12.5% (95% CI 10.3-15.1): 8.7% diagnosed and 3.8% undiagnosed. Nearly one-fourth (23.5%) of adults had IFG. Asians had the highest prevalence of impaired glucose metabolism (diabetes 16.1%, IFG 32.4%) but were significantly less likely to be obese. Among adults with diagnosed diabetes, less than one-half (45%) had A1C levels <7%; one-half (50%) had elevated blood pressure measures at interview, 43% of whom were not on antihypertensive medications; nearly two-thirds (66%) had elevated LDL levels, and only 10% had their glucose, blood pressure, and cholesterol all at or below recommended levels. Most adults (84%) with diagnosed diabetes were on medication, but only 12% were receiving insulin. CONCLUSIONS: In New York City, diabetes and IFG are widespread. Continue reading >>
- Antidepressant Medication as a Risk Factor for Type 2 Diabetes and Impaired Glucose Regulation
- Pre-diabetes Impaired Glucose Tolerance
- Postprandial Blood Glucose Is a Stronger Predictor of Cardiovascular Events Than Fasting Blood Glucose in Type 2 Diabetes Mellitus, Particularly in Women: Lessons from the San Luigi Gonzaga Diabetes Study
Impaired Fasting Glycemia
A person with impaired fasting glycemia isn't able to process glucose as efficiently as they should be able to Impaired fasting glycemia (IFG) may also be known as pre-diabetes or metabolic syndrome. Impaired fasting glycemia occurs when blood glucose levels in the body are elevated during periods of fasting , but not enough to prompt a diagnosis of diabetes. Effectively, a person with impaired fasting glycemia isnt able to process glucose as efficiently as they should be able to. What are the health implications of impaired fasting glycemia? People with impaired fasting glycemia face a greater risk of developing type 2 diabetes, despite having less of a risk than those with impaired glucose tolerance. The risk of developing cardiovascular disease is also lower than for people with impaired glucose tolerance . What does impaired fasting glycemia mean? Impaired fasting glycemia (IFG) means that the body cannot regulate glucose as efficiently as it should be able to. Glucose is usually carried around the body where it is absorbed and made into energy. Insulin regulates the concentration of glucose in the blood. IFG occurs when this process isnt functioning as effectively as it could, and effects millions of people in the UK. What are the symptoms of impaired fasting glycemia? Unfortunately, IFG may exhibit very little in the way of symptoms, meaning diagnosis often takes a long time. Many people diagnosed with IFG are overweight, have high blood pressure, increased cholesterol levels or a family history of IFG. So how do I know if my IFG becomes type 2 diabetes? IFG increases type 2 diabetes risk, so go straight to your doctor or healthcare professional if you feel unnaturally thirsty, pass more urine than usual, have recurrent infections, have blurred vision, or if your Continue reading >>
