What's Causing Your Joint And Nerve Pain?
It’s natural to feel a little discomfort in your hands, fingers, feet, and ankles from time to time. Joint pain is a part of getting older and can have a number of causes. But that ache in your foot or arm could also be a problem with a nerve caused by your diabetes. And that’s an issue that could be serious and require quick attention. So how do you tell the difference? It’s the leading cause of disability in the U.S. It affects more than 50 million adults. Often referred to as arthritis, it’s broadly defined as discomfort where two or more bones meet. Though often mild, sometimes sporadic, and rarely an emergency, the pain can be severe, making it hard to move the joint. If you have it, you’ll probably notice changes to your joint like: Stiffness Less range in motion Swelling Redness Tenderness or warmth A tougher time using it A difference in shape The causes of joint pain vary greatly. It could be: Muscle strains or sprains A broken or dislocated bone Gout Hypothyroidism Leukemia Lupus Osteoarthritis Rickets Lyme disease Rheumatoid arthritis Your doctor might call it diabetic neuropathy. It’s pain in your nerves, not in your bones. It happens when high blood sugar harms the nerve fibers. You can get it anywhere in your body, but it most often affects your legs and feet. Anywhere from 60%-70% of people with diabetes have some sort of neuropathy. Most get it after having the disease for 10 years or more. There are many types. But the two most likely to cause problems with your joints are peripheral and autonomic neuropathy. This is the most common form of diabetic joint pain. It affects your legs, arms, hands, feet, fingers, and toes. With ongoing diabetes, joints can no longer respond like they should to the strain and stress placed on them. As a result, Continue reading >>
- Is it possible to catch diabetes? It sounds absurd, but that's what a reputable new study suggests. And it may be that other 'lifestyle' diseases such as joint pain and even obesity are contagious, too
- Do Simvastatin Side Effects Include Diabetes and Joint Pain?
- 14 Ways to Reduce Joint Pain With Diabetes
Diabetes Is A Risk Factor For Knee Osteoarthritis Progression
Summary Recent studies have suggested that metabolic factors (obesity, diabetes, hypertension and dyslipidemia) and their clustering in metabolic syndrome (MetS) might be involved in the pathophysiology of knee osteoarthritis (OA). We investigated their impact on radiographic progression by an annualised measure of the joint space narrowing (JSN) of the medial tibiofemoral compartment. 559 patients older than 50 years with symptomatic knee OA were recruited for the placebo arm of the SEKOIA trial. The presence of diabetes, hypertension and dyslipidemia was determined at baseline interview. Body mass index (BMI) was calculated, obesity was considered >30 kg/m2. MetS was defined by the sum of metabolic factors ≥3. Minimal medial tibiofemoral joint space on plain radiographs was measured by an automated method at baseline and then annually for up to 3 years. The mean age of patients was 62.8 [62.2–63.4] years; 392 were women. A total of 43.8% was obese, 6.6% had type 2 diabetes, 45.1% hypertension, 27.6% dyslipidemia and 13.6% MetS. Mean annualised JSN was greater for patients with type 2 diabetes than without diabetes (0.26 [−0.35 to −0.17] vs 0.14 [−0.16 to −0.12] mm; P = 0.001). This association remained significant after adjustment for sex, age, BMI, hypertension and dyslipidemia (P = 0.018). In subgroup analysis, type 2 diabetes was a significant predictor of JSN in males but not females. The other metabolic factors and MetS were not associated with annualised JSN. Type 2 diabetes was a predictor of joint space reduction in men with established knee OA. No relationships were found between MetS or other metabolic factors and radiographic progression. Continue reading >>
Links Between Osteoarthritis And Diabetes:implications For Management From A Physical Activity Perspective
Go to: INTRODUCTION Osteoarthritis (OA) and Type 2 Diabetes Mellitus (T2DM) are two prevalent chronic diseases in the United States, Osteoarthritis (OA) affects 14% of adults aged 25 years and older and 34% of those above the age of 65.1 OA is a leading cause of disability and economic burden - around 40% of adults with OA report arthritis related limitations in daily activities and 30% report difficulties in work-related tasks.1 Diabetes affects 12% of adults 20 years and older and 26% of those above the age of 65.2 Diabetes is associated with mortality and serious complications such as heart disease and stroke, kidney failure, and lower-limb amputation.2 In the aging population, the co-existence of both OA and T2DM is frequent and can be a source of greater disability and economic burden.3,4 There seems to be an increased susceptibility to develop OA in those with T2DM.5,6 A recent report in adults ranging from 18–64 years showed that the prevalence of arthritis was 52% in those with T2DM compared to 27% in those without T2DM.7 The reason for the high prevalence of arthritis in those with T2DM is not entirely clear. OA and T2DM share common risk factors such as obesity and advanced aging, which may explain the higher prevalence of OA in the diabetic population.8 More recently, OA has been associated with systemic metabolic disturbances commonly seen in T2DM, suggesting that diabetes in and of itself influences the pathophysiology of OA independently of obesity or aging per se. These metabolic alterations have been proposed to serve as an underlying link between OA and T2DM. With the growing prevalence of older persons diagnosed with both OA and T2DM, adequate prevention and management of these combined conditions becomes necessary. Optimal care of these patients dep Continue reading >>
Link Between Diabetes And Osteoarthritis
Obesity blamed for the tendency of diabetes patients to develop osteoarthritis. Being overweight is a common trait in people with type 2 diabetes and has been blamed for the tendency of these patients to develop osteoarthritis (OA). A French research team has found a stronger correlation between the 2 ailments. The link between type 2 diabetes and the increased risk of osteoarthritis was first described in the early 1960s, but a December 2016 review of research data sets by Alice Courties, MD, and Jérémie Sellam (MD, PhD), from the rheumatology department of the Saint-Antoine Hospital in Paris, France, reveals an even stronger link between type 2 diabetes and OA that is independent of weight (or BMI) as a confounding factor. The meta-analysis looks at the effects of metabolic disturbances on OA, and posits the existence of a diabetes-induced form of OA as a subset, or "sub-phenotype" of metabolic OA. Although there is certainly a mechanical impact on the joints of many individuals with type 2 diabetes as a result of obesity, the data Courties and Sellam interpret supports that the connection between OA and obesity cannot explain all factors of OA development in individuals with type 2 diabetes. The joint ailment OA is characterized by osteophytes, synovial inflammation, cartilage degradation, and bone sclerosis, all of which are exacerbated by the "systemic metabolic disturbances and low-grade inflammation" associated with hyperglycemia and insulin resistance in individuals with Type 2 diabetes. Courties and Sellam remark that elevated fasting glucose concentration has also been associated with bone marrow lesions which, they said, are known to predict OA structural damages." In diabetic patients there is an added risk in the loss of subcondral bone through lower bone Continue reading >>
Diabetes And Arthritis: Is There A Connection?
Diabetes mellitus (also known as ‘sugar’ diabetes) interferes with the body’s ability to use sugar. It is a long-term condition requiring treatment by diet, pills and often injections of insulin. Generally doctors recognise two types of diabetes. Type I typically occurs in younger people and often requires treatment with insulin. Type 2 occurs in older overweight people and is treated with tablets but there can be a lot of overlap between the two types. There is often a family history of diabetes in both. Early symptoms of diabetes include thirst and passing a lot of urine, and some people lose a lot of weight. The problems with handling sugar, and specifically high blood sugar levels, can eventually lead to complications in the blood vessels, kidneys, eyes, and the nerves to the hands and feet. These complications can be delayed and minimised by controlling the blood sugar with treatment. People with diabetes are also prone to a number of musculoskeletal complications but the relationship between these complications and the diabetic control is not clear. Many of these problems are not unique to diabetes but occur more frequently in this condition. This short article describes the complications and offers advice on treatment and prevention. Shoulder problems Shoulder pain is probably the most common musculoskeletal disorder which I see associated with diabetes. Specifically the shoulder becomes stiff and painful due to inflammation and thickening of the tissue surrounding the shoulder joint – sometimes known as frozen shoulder. The pain may start following a minor injury or just come out of the blue. Typically the pain builds up to a constant nagging pain which limits the movement of the joint and causes sleep disturbance. The pain is worse in the first 3 months Continue reading >>
Arthritis & Diabetes
What do diabetes and arthritis have in common? Plenty. People with diagnosed diabetes are nearly twice as likely to have arthritis, indicating a diabetes-arthritis connection. Diabetes occurs when the body does not produce or use the hormone insulin sufficiently. Insulin shuttles glucose from foods into cells so it can be converted into energy. Without insulin, glucose remains in your blood (raising blood glucose levels), your cells create less energy and you feel fatigued. What starts off as a hormonal problem can evolve into joint problems, in addition to the widely known cardiovascular problems. Diabetes causes musculoskeletal changes that lead to symptoms such as joint pain and stiffness; swelling; nodules under the skin, particularly in the fingers; tight, thickened skin; trigger finger; carpal tunnel syndrome; painful shoulders; and severely affected feet. After having had diabetes for several years, joint damage – called diabetic arthropathy – can occur. Continue reading >>
Identifying And Treating Diabetes Joint Pain
Diabetes and joint pain are considered to be independent conditions. Joint pain may be a response to an illness, injury, or arthritis. It can be chronic (long-term) or acute (short-term). Diabetes is caused by the body not using the hormone insulin correctly, or insufficient production of it, which affects blood sugar levels. What would a hormone and blood sugar-related condition have to do with joint health? Diabetes is associated with widespread symptoms and complications. According to the Centers for Disease Control and Prevention, 47 percent of people with arthritis also have diabetes. There is an undeniably strong link between the two conditions. Diabetes can damage joints, a condition called diabetic arthropathy. Unlike pain caused by immediate trauma, the pain of arthropathy happens over time. Other symptoms include: thick skin changes in the feet painful shoulders carpal tunnel syndrome A joint is the place where two bones come together. Once a joint wears down, the protection it provides is lost. Joint pain from diabetic arthropathy comes in different forms. Charcot’s joint occurs when diabetic nerve damage causes a joint to break down. Also called neuropathic arthropathy, this condition is seen in the feet and ankles in people with diabetes. Nerve damage in the feet is common in diabetes, which may lead to Charcot’s joint. A loss of nerve function leads to numbness. People who walk on numb feet are more likely to twist and injure ligaments without knowing it. This places pressure on the joints, which can eventually cause them to wear down. Severe damage leads to deformities in the foot and other affected joints. Bone deformities in Charcot’s joint may be prevented through early intervention. Signs of the condition include: painful joints swelling or redn Continue reading >>
- Is it possible to catch diabetes? It sounds absurd, but that's what a reputable new study suggests. And it may be that other 'lifestyle' diseases such as joint pain and even obesity are contagious, too
- Do Simvastatin Side Effects Include Diabetes and Joint Pain?
- 14 Ways to Reduce Joint Pain With Diabetes
Diabetes Is An Independent Predictor For Severe Osteoarthritis
Abstract OBJECTIVE To evaluate if type 2 diabetes is an independent risk predictor for severe osteoarthritis (OA). RESEARCH DESIGN AND METHODS Population-based cohort study with an age- and sex-stratified random sample of 927 men and women aged 40–80 years and followed over 20 years (1990–2010). RESULTS Rates of arthroplasty (95% CI) were 17.7 (9.4–30.2) per 1,000 person-years in patients with type 2 diabetes and 5.3 (4.1–6.6) per 1,000 person-years in those without (P < 0.001). Type 2 diabetes emerged as an independent risk predictor for arthroplasty: hazard ratios (95% CI), 3.8 (2.1–6.8) (P < 0.001) in an unadjusted analysis and 2.1 (1.1–3.8) (P = 0.023) after adjustment for age, BMI, and other risk factors for OA. The probability of arthroplasty increased with disease duration of type 2 diabetes and applied to men and women, as well as subgroups according to age and BMI. Our findings were corroborated in cross-sectional evaluation by more severe clinical symptoms of OA and structural joint changes in subjects with type 2 diabetes compared with those without type 2 diabetes. CONCLUSIONS Type 2 diabetes predicts the development of severe OA independent of age and BMI. Our findings strengthen the concept of a strong metabolic component in the pathogenesis of OA. Osteoarthritis (OA) is among the most frequent chronic diseases in the industrialized world, with estimation for the lifetime prevalence ranging from 30–50% (1,2). Moreover, OA is associated with a substantial disease burden due to pain, functional decline and increased mortality (2). In a proportion of individuals, OA progresses toward joint failure requiring total joint replacement (arthroplasty). In the U.S., 200,000 hip joints are replaced every year, and intervention rates for hip and knee OA Continue reading >>
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Osteoarthritis In Hip Or Knee Can Increase Diabetes Risk
Individuals who have osteoarthritis in the hip or knee are significantly more likely to develop diabetes than are people without the condition, according to findings from a population-based cohort study. The relationship between osteoarthritis (OA) and new-onset diabetes was largely explained by the walking limitations brought on by OA, which was unsurprising to lead author Tetyana Kendzerska, MD, PhD, of the University of Toronto, who presented the study at the annual meeting of the Canadian Rheumatology Association. “Given that walking is critical for physical activity, this is not surprising perhaps [because] we know that physical activity is a key preventive measure for all chronic conditions, including diabetes,” Dr. Kendzerska said in an interview. But Dr. Kendzerska noted that even though “the World Health Organization has determined that osteoarthritis is the fastest growing chronic disease and the single most common cause of disability in older adults [and] the majority of people with OA have at least one other chronic condition – usually diabetes, high blood pressure, [or] heart disease ... few studies have examined the impact of OA on these other conditions, including on the development of diabetes.” Dr. Kendzerska and her coauthors used existing data to study a population of 16,362 adults aged 55 or older who did not have diabetes at baseline enrollment during 1996-1998 and were then followed until 2014 for a median period of 13 years. The adults’ median age was 68 years and median body mass index was 25.3 kg/m2; 61% were female. Cox regression modeling was used to quantify any association found between osteoarthritis and diabetes. A total of 1,637 (10%) had hip osteoarthritis, 2,431 (15%) had knee osteoarthritis, and 3,908 (24%) had some type of Continue reading >>
Rheumatoid Vs. Osteoarthritis And Diabetes
Though they both share connections with diabetes, rheumatoid arthritis (RA) and osteoarthritis (OA) are related to the disease in different ways. Let's look at a few of the connections: Autoimmunity and Type 1 Diabetes Type 1 diabetes is an autoimmune disease, as is rheumatoid arthritis. In people who have type 1 diabetes, the body attacks the pancreas, the organ where insulin is made, just as RA attacks the synovial tissue lining the joints. Inflammation is the common culprit. Levels of inflammatory markers, such as C-reactive protein (CRP) and interleukin-6 (IL-6), which often are high in people with rheumatoid arthritis, also are increased in those with type 1 diabetes. A study of people who had type 1 diabetes for longer than five years shows an increase in tumor necrosis factor-alpha (TNF-α), another inflammatory marker often elevated in people with inflammatory forms of arthritis. Inhibiting TNF-α with drugs such as adalimumab (Humira), etanercept (Enbrel) and infliximab (Remicade) is the goal of treating arthritis and related conditions. As scientists learn more about the roots of inflammation, some treatments for inflammatory arthritis may wind up helping to control other inflammation-related conditions. Researchers already are testing the possibilities. Reducing inflammation with Remicade improved insulin sensitivity in people who had inflammatory diseases and were insulin resistant, according to a small study published in the journal Annals of the Rheumatic Diseases. And in a study of 70 people who had type 2 diabetes, the arthritis drug anakinra (Kineret) brought down the glucose level, improved function of the pancreas and decreased levels of CRP and IL-6. Osteoarthritis and Type 2 Diabetes Go above your ideal weight, and your lower-body joints feel the bu Continue reading >>
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Causes Of Osteoarthritis
Primary osteoarthritis is mostly related to aging. With aging, the water content of the cartilage increases and the protein makeup of cartilage degenerates. Repetitive use of the joints over the years causes damage to the cartilage that leads to joint pain and swelling. Eventually, cartilage begins to degenerate by flaking or forming tiny crevasses. In advanced cases, there is a total loss of the cartilage cushion between the bones of the joints. Loss of cartilage cushion causes friction between the bones, leading to pain and limitation of joint mobility. Damage to the cartilage can also stimulate new bone outgrowths (spurs) to form around the joints. Osteoarthritis occasionally can be found in multiple members of the same family, implying an heredity (genetic) basis for this condition. Rarely, some of these hereditary cases of osteoarthritis are caused by defects in collagen, which is an important component of cartilage. Secondary osteoarthritis is caused by another disease or condition. Conditions that can lead to secondary osteoarthritis include obesity, repeated trauma or surgery to the joint structures, abnormal joints at birth (congenital abnormalities), gout, rheumatoid arthritis, diabetes, and other hormone disorders. Obesity causes osteoarthritis by increasing the mechanical stress on the cartilage. In fact, next to aging, obesity is the most powerful risk factor for osteoarthritis of the knees. The early development of osteoarthritis of the knees among weight lifters is believed to be in part due to their high body weight. Repeated trauma to joint tissues (ligaments, bones, and cartilage) is believed to lead to early osteoarthritis of the knees in soccer players. Interestingly, recent studies have not found an increased risk of osteoarthritis in long-distance Continue reading >>
Bone And Joint Problems Associated With Diabetes
If you have diabetes, you're at increased risk of various bone and joint disorders. Certain factors, such as nerve damage (diabetic neuropathy), arterial disease and obesity, may contribute to these problems — but often the cause isn't clear. Learn more about various bone and joint disorders, including symptoms and treatment options. Charcot joint What is it? Charcot (shahr-KOH) joint, also called neuropathic arthropathy, occurs when a joint deteriorates because of nerve damage — a common complication of diabetes. Charcot joint primarily affects the feet. What are the symptoms? You might have numbness and tingling or loss of sensation in the affected joints. They may become warm, red and swollen and become unstable or deformed. The involved joint may not be very painful despite its appearance. How is it treated? If detected early, progression of the disease can be slowed. Limiting weight-bearing activities and use of orthotic supports to the affected joint and surrounding structures can help. Diabetic hand syndrome What is it? Diabetic hand syndrome, also called diabetic cheiroarthropathy, is a disorder in which the skin on the hands becomes waxy and thickened. Eventually finger movement is limited. What causes diabetic hand syndrome isn't known. It's most common in people who've had diabetes for a long time. What are the symptoms? You may be unable to fully extend your fingers or press your palms together flat. How is it treated? Better management of blood glucose levels and physical therapy can slow the progress of this condition, but the limited mobility may not be reversible. Osteoporosis What is it? Osteoporosis is a disorder that causes bones to become weak and prone to fracture. People who have type 1 diabetes have an increased risk of osteoporosis. What are Continue reading >>
- Joint Disorders Associated with Diabetes
- Is it possible to catch diabetes? It sounds absurd, but that's what a reputable new study suggests. And it may be that other 'lifestyle' diseases such as joint pain and even obesity are contagious, too
- Do Simvastatin Side Effects Include Diabetes and Joint Pain?
Review The Adverse Effects Of Diabetes On Osteoarthritis: Update On Clinical Evidence And Molecular Mechanisms
Projected increases in the prevalence of both diabetes mellitus (DM) and osteoarthritis (OA) ensure their common co-existence. In an era of increasing attention to personalized medicine, understanding the influence of common comorbidities such as DM should result in improved care of patients with OA. In this narrative review, we summarize the literature addressing the interactions between DM and OA spanning the years from 1962 to 2014. We separated studies depending on whether they investigated clinical populations, animal models, or cells and tissues. The clinical literature addressing the influence of DM on OA and its therapeutic outcomes suggests that DM may augment the development and severity of OA and that DM increases risks associated with joint replacement surgery. The few high quality studies using animal models also support an adverse effect of DM on OA. We review strengths and weaknesses of the common rodent models of DM. The heterogeneous literature derived from studies of articular cells and tissues also supports the existence of biochemical and biomechanical changes in articular tissues in DM, and begins to characterize molecular mechanisms activated in diabetic-like environs which may contribute to OA. Increasing evidence from the clinic and the laboratory supports an adverse effect of DM on the development, severity, and therapeutic outcomes for OA. To understand the mechanisms through which DM contributes to OA, further studies are clearly necessary. Future studies of DM-influenced mechanisms may shed light on general mechanisms of OA pathogenesis and result in more specific and effective therapies for all OA patients. Copyright © 2015 Osteoarthritis Research Society International. Published by Elsevier Ltd. All rights reserved. Continue reading >>
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Association Between Diabetes Mellitus And Osteoarthritis: Systematic Literature Review And Meta-analysis
Objectives To investigate the prevalence of osteoarthritis (OA) in patients with diabetes mellitus (DM) and prevalence of DM in patients with OA and whether OA and DM are associated. Design A systematic literature review and meta-analysis. We included cohort, case–control and cross-sectional studies assessing the number of patients with DM and/or OA. The mean prevalence of OA among patients with DM and DM among patients with OA was calculated. Data from trials assessing an association of diabetes and OA were pooled and results are presented as unadjusted OR and 95% CI. Results From the 299 publications, we included 49 studies in the analysis, including 28 cross-sectional studies, 11 cohort studies and 10 case–control studies. In all, 21, 5 and 23 articles involved patients with OA exclusively, patients with DM and the general population, respectively. For 5788 patients with DM, the mean OA prevalence was 29.5±1.2%. For 645 089 patients with OA, the prevalence of DM was 14.4±0.1%. The risk of OA was greater in the DM than non-DM population (OR=1.46 (1.08 to 1.96), p=0.01), as was DM in the OA than non-OA population (OR=1.41 (1.21 to 1.65), p<0.00 001). Among the 12 studies reporting an OR adjusted on at least the body mass index, 5 showed no association of DM and OA and 7 identified DM as an independent risk factor. Conclusions This meta-analysis highlights a high frequency of OA in patients with DM and an association between both diseases, representing a further step towards the individualisation of DM-related OA within a metabolic OA phenotype. This is an Open Access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, Continue reading >>
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Diabetes And Osteoarthritis: A Vicious Cycle
Type 2 diabetes mellitus (T2DM) and osteoarthritis (OA) often come together. Higher frequencies of other conditions often linked to diabetes, such as obesity, hypertension, and dyslipidemia, also have been associated with OA.1 While the relationship is complex, OA and T2DM may feed into each other in a vicious cycle—with progression of T2DM contributing to worsening OA, and vice versa. Which Comes First? Body mass index (BMI) and age are well-known risk factors for both T2DM and OA, but there may be more here than meets the eye. Injury and mechanical wear and tear on the joints and cartilage over time might not be enough to explain the increased risk of OA in patients with T2DM. Some researchers are beginning to suspect that diabetes itself could be an independent risk factor for OA and have proposed a “diabetes-induced phenotype.”2 Chronic, low-grade inflammation is common in both OA and metabolic disorders. Studies suggest that lipid irregularities, hyperglycemia, and advanced glycosylation end products can be deleterious to cartilage homeostasis. Adipokines, inflammatory mediators released from adipose tissue, could also play a role.3 Moreover, treatment for T2DM could affect bone metabolism, with specific effects varying by type of agent. While thiazolidinediones can promote bone loss, metformin could stimulate bone formation through differentiation of osteoblasts.4 Some patients with T2DM already tend to have higher bone density, possibly linked to hyperinsulinemia and the anabolic action of insulin on bone.4 T2DM and OA: 1 + 1 = 3 While scientists debate which comes first, it is fair to say that concomitant OA and T2DM increases the risk for other comorbidities. People with OA are at increased risk for depression and lower quality of life in general.5 Having Continue reading >>
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