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Can Dehydration Cause Reactive Hypoglycemia

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Mnt 2 Midterm Exam Review

Sort Prediabetes Impaired Glucose Homeostasis 2 Forms (may be either or both) 1. Impaired Fasting Glucose (IFG): - fasting plasma glucose (FPG) ABOVE normal 2. Impaired Glucose Tolerance (IGT): - plasma glucose elevated afer 75-g glu load OR Hgb A1c: 5.7 - 6.4% >6.4% = Type 2 Diabetes Type 1 Diabetes 5-10% of ALL diagnosed Diabetes 2 Forms: 1. Immune Mediated - beta cells destroyed by autoimmune process 2. Idiopathic - cause of beta cell function loss unknown Symptoms: -hyperglycemia, polyuria, polydipsia, wt loss, dehydration, electrolyte disturbance, ketoacidosis, fatigue, irritability Complications: -ketoacidosis, macrovascular disease (CHD, peripheral vascular disease, cerebrovascular disease), microvascular disease (retinopathy, nephropathy), neuropathy Medical Management: -insulin injection or infusion pumps Monitoring: -self monitor BG, A1C testing, lipids, BP, ketones, weight/growth in children MNT: -integrate insulin regimen into preferred eating and physical activity schedule; consistency in timing and amount of CHO eaten if on fixed doses -adjust permeal insulin dose based on insulin to CHO ratios -energy intake to prevent wt gain in adults -adequate energy and nutrient Continue reading >>

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  1. picante

    For a long time, I've thought that my adrenal fatigue is really adrenal misregulation.
    Many of my usual symptoms are those of low cortisol and low aldosterone, but I know from a cortisol saliva test that my cortisol can shoot up when I exercise. The problem is that what goes up doesn't come down -- for a day or two. After exercise, my polyuria is worse for a day (at least). And the high cortisol does not give me energy; it feels just like neuro-exhaustion.
    It seems like I've finally discovered the connection between my immune system imbalance and these symptoms, although I'm going to need some cytokine tests to confirm it:
    heapsreal said: ↑
    The PVN is considered to be the final common structure where numerous different inputs initiate a stress response. Cytokines from the immune system, neurotransmitters from the nervous system, input from the limbic system (emotions), and hormones from the endocrine system all converge to elicit a stress response from the HPA axis at the PVN.
    There's a concept in neurology called the central integrative state, which basically states that the net output of a neurological structure is a summation of the excitatory inputs versus the inhibitory inputs. In other words, if a combination of stimulus from neurotransmitters, hormones, and cytokines all result in an excitatory state, the result will be an elevation of cortisol.
    On the other hand, if the combined total input is that of an inhibitory response, the results will be a low output of cortisol. This is how someone can go directly to the adrenal exhaustion phase: If the total summation of inputs is inhibitory to the PVN, there will be a diminished adrenal response and low cortisol.
    Using this model, there's no such thing as "adrenal fatigue." Rather, it's merely a lack of inputs that can generate an adequate adrenal response.
    The following can excite the PVN and therefore contribute to high cortisol: insulin, acetylcholine, elevated epinephrine and norepinepherine, and Th2 cytokines (IL-4, IL-5 and IL-10).
    The following can inhibit the PVN and therefore contribute to low cortisol: GABA, low epinephrine and norepinepherine, endothelial nitric oxide, interferon, tumor necrosis factor, and Th1 cytokines (IFG, IL-12, TNF). On Stop the Thyroid Madness, there is a section called
    How Does Inflammation Inhibit Cortisol?
    A dysfunctional immune system will signal for higher levels of certain cytokines while suppressing others. Certain cytokines such at TGF-B1 (Transforming Growth Factor Beta One) can inhibit not only cortisol secretion but also aldostersone secretion. Then you have cytokines such as TNF-a (Tumor Necrosis Factor Alpha) which can attach itself to ACTH receptors on the adrenal gland and prevent cortisol secretion. Elevated levels of TNF-a are strongly associated with low morning levels of cortisol.
    Certain cytokines such as Interluekin-10 (IL-10) can keep other proinflammatory cytokines at bay while also signaling cortisol secretion. This cytokine in specific can become smothered in people who have adrenal fatigue by other cytokines from the TH1 side of the immune system, which are acting aggressively. One of the functions of IL-10 is to keep the pro-inflammatory cytokines from the TH1 from over reacting. When pro-inflammatory cytokines are not put in check they can run rampant.
    Some researchers are even beginning to theorize that over time our HPA-axis will numb itself to certain cortisol signaling cytokines such as (IL-3) and (IL-6) in order to preserve our body from prolonged exposure to cortisol. Cytokines are also strongly linked to other symptoms that are generally attributed to low cortisol, things like iron dumping, sleep disturbances, low appetite, hair loss and muscle wasting.

  2. picante

    Lately I've been desperate to find some way to keep water in my body; I've been losing weight again, and it's mostly water. But I just learned that my years-long habit of just adding sea salt isn't the ticket:
    Salt should be used sparingly due to very low Aldosterone Levels, which are inhibited even more by higher salt intake. People who are sodium-sensitive generally are well aware of their problem, as they quickly retain extra water when ingesting larger amounts of salt, or their blood pressure goes up, however there are those who respond the opposite, where for instance drinking a medium-sized glass of water saturated with common table salt will have them keep running to the bathroom some time later, with some individuals losing perhaps as much as 10-15 lb of water weight. This is similar to the opposing effect of simple sugar consumption on hypoglycemia versus hyperglycemia.
    In such a case, the strategy is not to increase someone's salt intake (which would otherwise cause more dehydration), but to boost aldosterone levels, which will subsequently increase sodium retention. Nutritional factors such as choline, licorice, iron, Vitamin B1, and - to a small extent - Vitamin E can be used for that purpose, which should be matched to individual requirements. http://www.acu-cell.com/pna.html
    Choline should be used with caution if there is a disposition for water retention (swollen hands or feet), since it boosts aldosterone activity, which in turn results in sodium retention. http://www.acu-cell.com/dis-hyp.html

  3. picante

    If anyone has suggestions on how to rehydrate, please post them.
    Has anyone done immune rebalancing, and has it helped with your adrenal function?
    I've started taking Citicoline, and I already take B1 and E. I don't tolerate licorice well. I'm trying a rehydration drink made of water, salt, maple syrup and fresh lime juice.
    I start out my day weighing about 117 lbs., then by bedtime I'm around 120 lbs. Then I get up to pee 3-4 times and I wind up parched, even though I'm drinking 2 or more 12-oz. glasses of water/rehydration drink at night.
    Nighttime is usually when I have low cortisol symptoms, too, such as hypoglycemia.

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