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Can Ketoacidosis Cause Brain Damage

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Cognitive Function In Diabetes

Cognitive deficits, that is clinically relevant problems in cognitive performance, are commonly observed in people with both type 1 (T1DM) as well as type 2 diabetes (T2DM). Both diseases are related specifically to slowing of mental processing speed, psycho-motor speed, executive functions and attention. In T2DM learning and memory problems are often noted but less so in T1DM. Evidence for changes in brain structure and functioning accompanying cognitive dysfunction is accumulating. Chronic hyperglycaemia and associated microvascular disease appear to be the most important determinants of cognitive decrements in diabetes. Hypoglycaemia and hyperglycaemia Hypoglycaemia can lead to unconsciousness, seizure, coma or even death. Mild to moderate levels of hypoglycaemia commonly affect higher-order cognitive functions. Patients may experience mood changes and difficulty with memory, planning, attention and concentration [1]. Mental speed rapidly decreases, while accuracy remains relatively unaffected. With severe hypoglycaemia, all cognitive functions may be affected, seriously decreasing a patient’s mental capabilities. Whereas blood glucose can be quickly restored, cognitive dysfun Continue reading >>

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  1. Justin Mulesa

    Depends on your definition of Damage to the brain. It does, by definition, alter the brain chemistry, whether this alteration is beneficial or harmful depends on the baseline state, and effect of the medication. If all brain chemistry alteration was considered damaging, then all psych meds could be considered to cause brain damage (an interesting consideration). However, if the baseline state is itself abnormal, for example in schizophrenia, then dopamine antagonists, such as anti-psychotics, could be and are considered therapeutic. However, with these, and any drugs that alter brain chemistry (and indeed any drug at all) have undesired effects, known as side effects, due to the crudeness and non-specificity medications have. In the brain, even minor fluctuations can have massive consequences. A consequence of dopamine antagonism, at least for the older, less specific, 1st generation anti-psychotics, such as haloperidol, had a cumulative dose side effect which was like Parkinson's disease, known as tardive dyskinesia. Parkinson’s, and a great deal of all motor skills, are very closely linked to dopamine and their receptors, and too much or too little can lead to a decline in motor function. As a dopamine agnoist, as due to the widespread almost ubiquitous negative feedback loop found in physiology, one could conjecture that long term over stimulation of the dopamine tracts could result in a Parkinson’s like disease or other forms of dementia later on down the road, although long term and geriatric studies on adhd medications can be somewhat difficult to come across, here is a link to a simple google search:

  2. Mike Repik

    However that doesn't mean it will. There are a lot of factors. But the big question is, can you solve the problem by any other means than taking a pill which can possibly cause damage?

  3. Brendan Hardy

    There is some evidence to suggest that releasers like Adderall (a proprietary formulation of amphetamine salts) can cause damage to dopamine receptors over long term high dosage use. It appears that Dopamine reuptake inhibitors like Ritalin or Concerta (methylphenidate) have much less risk of brain damage. Now when I say brain damage, I don't mean it'll put you in a coma. It's more likely to kill off some dopamine receptors via overstimulation. As far as I know, the brain is very plasticine and can heal much of the damage over time.

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